Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Henk A. Vink"'
Publikováno v:
Frontiers in Molecular Neuroscience, Vol 15 (2022)
Severe hearing loss or deafness is often caused by cochlear hair cell loss and can be mitigated by a cochlear implant (CI). CIs target the auditory nerve, consisting of spiral ganglion cells (SGCs), which degenerate gradually, following hair cell los
Externí odkaz:
https://doaj.org/article/67256d60cdbe4d5ea8b57740a18e4e6c
Publikováno v:
Brain Sciences, Vol 12, Iss 1, p 2 (2021)
We investigated whether treatment with brain-derived neurotrophic factor (BDNF), which is known to protect spiral ganglion cells (SGCs), could also protect hair cells (HCs) and supporting cells (SCs) in the organ of Corti of a guinea pig model of sen
Externí odkaz:
https://doaj.org/article/ef2898fe3b2240029ccf439e03c4c8f8
Publikováno v:
Brain Sciences, Vol 10, Iss 11, p 787 (2020)
In deaf subjects using a cochlear implant (CI) for hearing restoration, the auditory nerve is subject to degeneration, which may negatively impact CI effectiveness. This nerve degeneration can be reduced by neurotrophic treatment. Here, we compare th
Externí odkaz:
https://doaj.org/article/f0f8e6fa838f40f08dcb3b3b4ffaf9f7
Publikováno v:
Brain Sciences, Vol 12, Iss 2, p 2 (2022)
Brain Sciences
Brain Sciences; Volume 12; Issue 1; Pages: 2
Brain Sciences
Brain Sciences; Volume 12; Issue 1; Pages: 2
We investigated whether treatment with brain-derived neurotrophic factor (BDNF), which is known to protect spiral ganglion cells (SGCs), could also protect hair cells (HCs) and supporting cells (SCs) in the organ of Corti of a guinea pig model of sen
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6bbcf8b187efd6c1d04708b4872e112d
http://hdl.handle.net/11697/174532
http://hdl.handle.net/11697/174532
Publikováno v:
Hearing research. 400
Treatment with neurotrophins prevents degeneration of spiral ganglion cells (SGCs) after severe hair cell loss. In a previous study we demonstrated a long-lasting effect with brain-derived neurotrophic factor (BDNF) after cessation of treatment. In t