Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Hellen Buijze"'
Autor:
Lukas Page, Kevin Dennehy, Katharina Mueller, Philipp Girl, Eva Loell, Hellen Buijze, Johanna-Maria Classen, Helmut Messmann, Christoph Roemmele, Reinhard Hoffmann, Sebastian Wurster, Andre Fuchs
Publikováno v:
Frontiers in Immunology, Vol 15 (2024)
IntroductionPre-existent pools of coronavirus-specific or cross-reactive T cells were shown to shape the development of cellular and humoral immune responses after primary mRNA vaccination against SARS-CoV-2. However, the cellular determinants of res
Externí odkaz:
https://doaj.org/article/6aa8e4d9843148aaa0700c3b2b7c8d10
Autor:
Pei, Hellen Buijze, Volker Brinkmann, Robert Hurwitz, Anca Dorhoi, Stefan H. E. Kaufmann, Gang
Publikováno v:
International Journal of Molecular Sciences; Volume 24; Issue 11; Pages: 9701
Mouse guanylate-binding proteins (mGBPs) are recruited to various invasive pathogens, thereby conferring cell-autonomous immunity against these pathogens. However, whether and how human GBPs (hGBPs) target M. tuberculosis (Mtb) and L. monocytogenes (
Autor:
Anca Dorhoi, Gang Pei, Stefan H. E. Kaufmann, Hellen Buijze, Haipeng Liu, Volker Brinkmann, Pedro Moura-Alves, Hiroshi Kawabe, Christian Goosmann
Publikováno v:
Autophagy
The E3 ubiquitin ligase NEDD4 has been intensively studied in processes involved in viral infections, such as virus budding. However, little is known about its functions in bacterial infections. Our investigations into the role of NEDD4 in intracellu
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4398b34676463aaef9aea66d51e6e9f8
https://hdl.handle.net/21.11116/0000-0000-3152-C21.11116/0000-0000-3154-A
https://hdl.handle.net/21.11116/0000-0000-3152-C21.11116/0000-0000-3154-A
Autor:
Nisha Verwey, Yotam Raz, Vered Raz, Seyed Yahya Anvar, Silvère M. van der Maarel, Annemieke Aartsma-Rus, Hellen Buijze
Publikováno v:
American Journal of Pathology, 184(4), 1119-1131
Alanine expansion mutations in poly(A)-binding protein nuclear 1 (PABPN1) cause muscle weakness in the late-onset disorder oculopharyngeal muscular dystrophy. In affected muscles, expanded PABPN1 forms nuclear aggregates, depleting levels of soluble
Autor:
Hellen Buijze, Niki Fens, Tom van der Poll, Marc P. van der Schee, Peter J. Sterk, Rutger Top
Publikováno v:
D96. WHAT'S NEW IN TUBERCULOSIS DIAGNOSTICS.
Autor:
Erik van der Wal, Andrea Venema, Vered Raz, Hellen Buijze, Rinse Klooster, Kirsten R. Straasheijm, Michael Antoniou, Samantha Routledge, Seyed Yahya Anvar, Silvère M. van der Maarel
Publikováno v:
American Journal of Pathology, 179(4), 1988-2000
Oculopharyngeal muscular dystrophy (OPMD) is an autosomal dominant disease caused by an alanine tract expansion mutation in poly(A) binding protein nuclear 1 (expPABPN1). To model OPMD in a myogenic and physiological context, we generated mouse myobl
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7844bafa05d2f7aa7d12811534b91d6c
https://doi.org/10.1016/j.ajpath.2011.06.044
https://doi.org/10.1016/j.ajpath.2011.06.044