Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Helena Paszeková"'
Autor:
Tomas Zikmund, Helena Paszekova, Juraj Kokavec, Paul Kerbs, Shefali Thakur, Tereza Turkova, Petra Tauchmanova, Philipp A. Greif, Tomas Stopka
Publikováno v:
International Journal of Molecular Sciences, Vol 21, Iss 6, p 2073 (2020)
ISWI chromatin remodeling ATPase SMARCA5 (SNF2H) is a well-known factor for its role in regulation of DNA access via nucleosome sliding and assembly. SMARCA5 transcriptionally inhibits the myeloid master regulator PU.1. Upregulation of SMARCA5 was pr
Externí odkaz:
https://doaj.org/article/b166c7a9913442da974931b8d07ed751
Autor:
Sabina Sevcikova, Helena Paszekova, Lenka Besse, Lenka Sedlarikova, Veronika Kubaczkova, Martina Almasi, Ludek Pour, Roman Hajek
Publikováno v:
Biomedical Papers, Vol 159, Iss 2, Pp 288-293 (2015)
Background: Multiple myeloma (MM) is characterized by malignant proliferation of plasma cells (PC) which accumulate in the bone marrow (BM). The advent of new drugs has changed the course of the disease from incurable to treatable, but most patients
Externí odkaz:
https://doaj.org/article/bdd4b12404aa4c73a5c883b3899fd0dc
Autor:
Petra Bašová, Helena Paszeková, Lubomír Minařík, Martina Dluhošová, Pavel Burda, Tomáš Stopka
Publikováno v:
International Journal of Molecular Sciences, Vol 23, Iss 12, p 6729 (2022)
The transcription factor PU.1 (Purine-rich DNA binding, SPI1) is a key regulator of hematopoiesis, whose level is influenced by transcription through its enhancers and its post-transcriptional degradation via microRNA-155 (miR-155). The degree of tra
Externí odkaz:
https://doaj.org/article/693d1e284c9e4a7c911512e344ee4176
Autor:
Petra Bašová, Helena Paszeková, Lubomír Minařík, Martina Dluhošová, Pavel Burda, Tomáš Stopka
Publikováno v:
International Journal of Molecular Sciences; Volume 23; Issue 12; Pages: 6729
The transcription factor PU.1 (Purine-rich DNA binding, SPI1) is a key regulator of hematopoiesis, whose level is influenced by transcription through its enhancers and its post-transcriptional degradation via microRNA-155 (miR-155). The degree of tra