Zobrazeno 1 - 10
of 24
pro vyhledávání: '"Hela G. Zouari"'
Publikováno v:
Pain Research and Management, Vol 2020 (2020)
Introduction. The management of neuropathic pain remains complex, generally because of the psychiatric comorbidity that is often underdiagnosed. The objectives of our work were to determine the link between depression and the characteristics of NP on
Externí odkaz:
https://doaj.org/article/250feca923c14970a8d3374886673e62
Autor:
Farida Gorram, Tarik Nordine, V. Plante-Bordeneuve, Jean-Pascal Lefaucheur, Thibaud Damy, Hela G. Zouari
Publikováno v:
Clinical Neurophysiology. 129:1565-1569
Objective To reappraise the value of electrochemical skin conductance (ESC) measurement by Sudoscan® to assess the distal involvement of small autonomic fibers in familial amyloid polyneuropathy (FAP) due to various transthyretin (TTR) mutations. Me
Autor:
Firas Kammoun, Jean-Pascal Lefaucheur, Kaouthar Masmoudi, Rim Kammoun, Hela G. Zouari, Leila Triki, Ines Kammoun
Publikováno v:
Neurophysiologie Clinique. 47:427-436
Summary Objectives To reappraise the respective involvement of small- and large-fiber damage in carpal tunnel syndrome (CTS) and to determine the diagnostic sensitivity of autonomic tests compared to conventional nerve conduction study (NCS). Methods
Publikováno v:
Pain medicine (Malden, Mass.). 20(5)
Objective To determine whether clinical features of neuropathic pain differ with respect to the presence of small-fiber neuropathy (SFN) in patients with primary Sjogren's syndrome (pSS). Methods We compared the clinical presentation of neuropathic p
Autor:
Jean-Pascal Lefaucheur, Hela G. Zouari, Samar S. Ayache, Jora Xhaxho, Abir Wahab, Sophie Ng Wing Tin, Alain Créange, Christiane Ajzenberg, Anne-Isabelle Tropeano
Publikováno v:
Neurophysiologie Clinique/Clinical Neurophysiology
Neurophysiologie Clinique/Clinical Neurophysiology, Elsevier Masson, 2019, 49, pp.59-67. ⟨10.1016/j.neucli.2018.12.004⟩
Neurophysiologie Clinique = Clinical Neurophysiology
Neurophysiologie Clinique = Clinical Neurophysiology, Elsevier Masson, 2019, 49, pp.59-67. ⟨10.1016/j.neucli.2018.12.004⟩
Neurophysiologie Clinique/Clinical Neurophysiology, Elsevier Masson, 2019, 49, pp.59-67. ⟨10.1016/j.neucli.2018.12.004⟩
Neurophysiologie Clinique = Clinical Neurophysiology
Neurophysiologie Clinique = Clinical Neurophysiology, Elsevier Masson, 2019, 49, pp.59-67. ⟨10.1016/j.neucli.2018.12.004⟩
Summary Objectives Metabolic abnormalities, such as, glycemic disorders and metabolic syndrome (GDMS) are one of the main causes of peripheral neuropathies. The objective of this study was to evaluate the impact of adding specific coaching care (CC)
Publikováno v:
Pain practice : the official journal of World Institute of Pain. 19(4)
Objective We attempted to determine whether clinical features could differentiate painful small-fiber neuropathy related to primary Sj€ogren's syndrome (pSS-SFN) from idiopathic SFN (idio-SFN). Methods Validated clinical questionnaires and neurophy
Publikováno v:
European journal of neurology. 26(1)
BACKGROUND AND PURPOSE Distal involvement of autonomic nerve fibers is critical in familial amyloid polyneuropathy (FAP) due to transthyretin (TTR) mutation. This study compares different methods for assessing autonomic foot innervation in TTR-FAP pa
Publikováno v:
Neurophysiologie Clinique. 49:189-190
Contexte Le SGB est une polyradiculoneuropahie auto-immune aigue. La classification electrophysiologique de ses variantes est tres interessante pour evaluer le pronostic et adapter le rythme de surveillance. Objectifs Evaluer l’apport de la classif
Autor:
Tarik Al-Ani, Hela G. Zouari, Alain Créange, Samar S. Ayache, Moussa A. Chalah, Jean-Pascal Lefaucheur, Wassim H. Farhat
Publikováno v:
Journal of the Neurological Sciences. 358:351-356
Tremor is frequently encountered in multiple sclerosis (MS) patients. However, its underlying pathophysiological mechanisms remain poorly understood. Our aim was to assess the potential role of the cerebellum and brain stem structures in the generati
Publikováno v:
Neurophysiologie Clinique. 49:190
Contexte La paralysie flasque aigue (PFA) est un tableau clinique aigu de paralysie flasque caracterise par l’hypotonie et l’abolition des reflexes osteotendineux. Vu que le diagnostic etiologique n’est pas toujours aise, le clinicien recourt a