Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Heidi Oehlandt"'
Publikováno v:
Journal of International Advanced Otology, Vol 18, Iss 6, Pp 495-500 (2022)
Externí odkaz:
https://doaj.org/article/17a9fc0e6d9f4ee99655a96dcc1ca243
Autor:
Heidi, Oehlandt, Juha, Laakso, Oskari, Lindfors, Joonas, Toivonen, Dennis, Poe, Saku T, Sinkkonen
Publikováno v:
Otology & Neurotology. 43:611-618
Baro-challenge-induced Eustachian tube dysfunction (ETD) manifests due to inadequate Eustachian tube (ET) function during rapid ambient pressure changes, although ET function may be normal in normobaric situations. This systematic review and retrospe
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5a84dbd4f90c344d2f1ccfa33d9502d8
https://doi.org/10.1097/mao.0000000000003558
https://doi.org/10.1097/mao.0000000000003558
Objectives Currently, there is no consensus regarding the best protocol for diagnosing Eustachian tube dysfunction (ETD). We aimed to evaluate how patient characteristics affect tubomanometry (TMM) results. If an association between patient character
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7af6155f0abf10672510f2654bdfc061
http://hdl.handle.net/10138/349780
http://hdl.handle.net/10138/349780
Publikováno v:
Otology & Neurotology.
OBJECTIVES Tubomanometry (TMM) is an objective method for investigating Eustachian tube (ET) function, but difficulties in technique and interpretation may reduce its usability. The objective of our study was to investigate common difficulties with T
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::676244b1f75c20647b0336ef2668b78b
https://doi.org/10.1097/mao.0000000000003018
https://doi.org/10.1097/mao.0000000000003018
Autor:
Jorma Toppari, Kirsi Vaaralahti, Johanna Tommiska, Helena E. Virtanen, Heidi Oehlandt, Rosanna Koivu, Taneli Raivio, Eeva-Maria Laitinen
Publikováno v:
Molecular and Cellular Endocrinology
Molecular and Cellular Endocrinology, Elsevier, 2011, 341 (1-2), pp.35. ⟨10.1016/j.mce.2011.05.015⟩
Molecular and Cellular Endocrinology, Elsevier, 2011, 341 (1-2), pp.35. ⟨10.1016/j.mce.2011.05.015⟩
Mutations in FGFR1 , GNRHR , PROK2 , PROKR2 , TAC3 , or TACR3 underlie isolated hypogonadotropic hypogonadism (IHH) with clinically variable phenotypes, and, by causing incomplete intrauterine activation of the hypothalamic–pituitary–gonadal axis
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::eb55faac50bfc21d563c745856215f26
https://hal.archives-ouvertes.fr/hal-00719875/document
https://hal.archives-ouvertes.fr/hal-00719875/document