Zobrazeno 1 - 3
of 3
pro vyhledávání: '"Hee J. Cha"'
Autor:
Jae-A Han, Jong S. Lee, Byoung S. Kwon, Hong R. Cho, Hye Jin Kim, Kyung Min Kim, Hee J. Cha, Robert S. Mittler, Ahra Kim, Yoonkyung Do, Sumi Koo, Byungsuk Kwon
Publikováno v:
The Journal of Immunology. 191:2657-2664
Damage-associated molecular patterns released from damaged kidney cells initiate postischemic inflammation, an essential step in the progression of kidney ischemia–reperfusion injury (IRI). However, the mechanism that coordinates this highly specif
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7349f33a5acf30926500cdac4cf64543
https://doi.org/10.4049/jimmunol.1300358
https://doi.org/10.4049/jimmunol.1300358
Publikováno v:
Journal of immunological methods. 407
Kidney ischemia-reperfusion injury (IRI) occurs as a result of complex interactions of kidney parenchymal cells and immune cells that are initiated by hypoxic damage of parenchymal cells. In particular, tubular epithelial cells (TECs) not only are su
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c6f22ccfdb0fe0ee2c6dd639885147c7
https://pubmed.ncbi.nlm.nih.gov/24681240
https://pubmed.ncbi.nlm.nih.gov/24681240
Autor:
Sun K. Lee, Hee J. Cha, Sang C. Lee, Ahra Kim, Jung Dae Kim, Byoung S. Kwon, Hye Jin Kim, Hong R. Cho, Byungsuk Kwon, Robert S. Mittler, Jong S. Lee
Publikováno v:
Proceedings of the National Academy of Sciences. 109
Renal ischemia-reperfusion injury (IRI) after kidney transplantation is a major cause of delayed graft function. Even though IRI is recognized as a highly coordinated and specific process, the pathways and mechanisms through which the innate response
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9f83c7a8ade301fe231ff41dee60c7df
https://doi.org/10.1073/pnas.1112256109
https://doi.org/10.1073/pnas.1112256109