Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Heart left ventricle overload"'
Autor:
Perla Pérez-Treviño, Julio Altamirano, Jorge Pérez-Treviño, Cuauhtémoc Borja-Villa, Noemí García
Publikováno v:
Cellular Physiology and Biochemistry, Vol 37, Iss 4, Pp 1329-1344 (2015)
Cellular Physiology and Biochemistry
Cellular Physiology and Biochemistry
Background/Aims: Pressure-overload (PO) causes cardiac hypertrophy (CH), and eventually leads to heart failure (HF). HF ventricular myocytes present transverse-tubules (TT) loss or disarrangement and decreased sarcoplasmic reticulum (SR) density, and
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ef1ae3e5e517164090ff5a4bb3ba2261
https://doi.org/10.1159/000430254
https://doi.org/10.1159/000430254
Autor:
Edgar Parra, Pilar Zambrano, Carolina Hernández, German Toro, Juan David Ramírez, Zulma M. Cucunubá
Publikováno v:
International Journal of Infectious Diseases, Vol 26, Iss C, Pp 146-148 (2014)
(2006), PAHO/WHO (Organización Panamericana de la Salud
Repositorio EdocUR-U. Rosario
Universidad del Rosario
instacron:Universidad del Rosario
(2006), PAHO/WHO (Organización Panamericana de la Salud
Repositorio EdocUR-U. Rosario
Universidad del Rosario
instacron:Universidad del Rosario
Chagas disease is a complex zoonotic pathology caused by the kinetoplastid Trypanosoma cruzi. This parasite presents remarkable genetic variability and has been grouped into six discrete typing units (DTUs). The association between the DTUs and clini
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::47cb11c1f2d9499b07f68841c2aca5af
http://www.sciencedirect.com/science/article/pii/S1201971214014969
http://www.sciencedirect.com/science/article/pii/S1201971214014969
Autor:
Bisinotto, Flora Margarida Barra [UNESP], Fabri, Daniel Capucci, Calçado, Maida Silva, Perfeito, Paula Borela, Tostes, Lucas Vieira, Sousa, Gabriela Denardi
Publikováno v:
Scopus
Repositório Institucional da UNESP
Universidade Estadual Paulista (UNESP)
instacron:UNESP
Repositório Institucional da UNESP
Universidade Estadual Paulista (UNESP)
instacron:UNESP
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Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=od______3056::d428d5285314213f2c6a9595b5927547
Publikováno v:
American journal of physiology. Heart and circulatory physiology, 279(3), H1120-H1127. American Physiological Society
American Journal of Physiology-Heart and Circulatory Physiology, 3 48-3, 279
American Journal of Physiology-Heart and Circulatory Physiology, 3 48-3, 279
It is generally accepted that the left ventricle (LV) hypertrophies (LVH) to normalize systolic wall stress (ςs) in chronic pressure overload. However, LV filling pressure (Pv) may be elevated as well, supporting the alternative hypothesis of end-di
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::70ec1ea267dd766a72a11ab13b282a89
https://pure.amc.nl/en/publications/left-ventricular-wall-stress-normalization-in-chronic-pressureoverloaded-heart-a-mathematical-model-study(56517281-92dc-4bc0-9c19-d2334555d259).html
https://pure.amc.nl/en/publications/left-ventricular-wall-stress-normalization-in-chronic-pressureoverloaded-heart-a-mathematical-model-study(56517281-92dc-4bc0-9c19-d2334555d259).html
It is generally accepted that the left ventricle (LV) hypertrophies (LVH) to normalize systolic wall stress (σ(s)) in chronic pressure overload. However, LV filling pressure (P(v)) may be elevated as well, supporting the alternative hypothesis of en
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=dris___00893::1a891eb999e420cdb970d713a4405a38
http://resolver.tudelft.nl/uuid:27fd9b6b-b492-4626-975a-4b0e1e23952e
http://resolver.tudelft.nl/uuid:27fd9b6b-b492-4626-975a-4b0e1e23952e