Zobrazeno 1 - 10
of 15
pro vyhledávání: '"Havard Attramadal"'
Autor:
Havard Attramadal, Sushanta K. Banerjee, Brahim Chaqour, Gary Fisher, Lester Lau, Bernard Perbal, Ulf Smith, Herman Yeger
Publikováno v:
Journal of Cell Communication and Signaling. 17:7-11
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::288b276e4e8d299f3273f80823609aab
https://doi.org/10.1007/s12079-023-00731-4
https://doi.org/10.1007/s12079-023-00731-4
Autor:
Maria Belland Olsen, Xiang Yi Kong, Mieke C. Louwe, Knut H. Lauritzen, Ylva Schanke, Ole Jørgen Kaasbøll, Håvard Attramadal, Jonas Øgaard, Sverre Holm, Pål Aukrust, Liv Ryan, Terje Espevik, Maria Yurchenko, Bente Halvorsen
Publikováno v:
Frontiers in Immunology, Vol 15 (2024)
Acute myocardial infarction (MI) results in tissue damage to affected areas of the myocardium. The initial inflammatory response is the most damaging for residual cardiac function, while at later stages inflammation is a prerequisite for proper heali
Externí odkaz:
https://doaj.org/article/ce016b2ea7294e4086757b857055d202
Autor:
Robert J. Lefkowitz, T. M. Dawson, Havard Attramadal, Chiye Aoki, J. L. Arriza, J. Codina, S. H. Snyder, Madan M. Kwatra, Marc G. Caron
Publikováno v:
Journal of Biological Chemistry. 267:17882-17890
Homologous or agonist-specific desensitization of beta 2-adrenergic receptors (beta 2AR) is mediated by the beta-adrenergic receptor kinase (beta ARK) which specifically phosphorylates the agonist-occupied form of the receptor. However, the capacity
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::84180824243d10c02b064c91d750795e
https://doi.org/10.1016/s0021-9258(19)37125-x
https://doi.org/10.1016/s0021-9258(19)37125-x
Autor:
Theis Christian Tønnessen, Arne Olav Melleby, Ida Marie Hauge-Iversen, Emil Knut Stenersen Espe, Mohammed Shakil Ahmed, Thor Ueland, Espen Andre Haavardsholm, Sara Marie Atkinson, Espen Melum, Håvard Attramadal, Ivar Sjaastad, Leif Erik Vinge
Publikováno v:
PLoS ONE, Vol 17, Iss 1, p e0262821 (2022)
AimsPatients with rheumatoid arthritis (RA) have increased risk of heart failure (HF). The mechanisms and cardiac prerequisites explaining this association remain unresolved. In this study, we sought to determine the potential cardiac impact of an ex
Externí odkaz:
https://doaj.org/article/96fa321cee4146c688a05b660af2d1f4
Autor:
Havard Attramadal, Berg K
Publikováno v:
Europe PubMed Central
Cardiovascular disease is the most frequent cause of death in the western hemisphere. Although significant advances in pharmacotherapy have been achieved, the morbidity and mortality of cardiovascular disease will probably remain a scourge of affluen
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid_dedup__::3f12062ff14fc414dd2f04559dcae4db
https://pubmed.ncbi.nlm.nih.gov/11255869
https://pubmed.ncbi.nlm.nih.gov/11255869
Autor:
Havard Attramadal
Publikováno v:
Europe PubMed Central
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid_dedup__::c14c4be9e1864c7e95168d7eccb8b041
https://pubmed.ncbi.nlm.nih.gov/10815483
https://pubmed.ncbi.nlm.nih.gov/10815483
Autor:
Knut H Lauritzen, Maria Belland Olsen, Mohammed Shakil Ahmed, Kuan Yang, Johanne Egge Rinholm, Linda H Bergersen, Qin Ying Esbensen, Lars Jansen Sverkeli, Mathias Ziegler, Håvard Attramadal, Bente Halvorsen, Pål Aukrust, Arne Yndestad
Publikováno v:
eLife, Vol 10 (2021)
Poly(ADP-ribose) polymerase (PARP) enzymes initiate (mt)DNA repair mechanisms and use nicotinamide adenine dinucleotide (NAD+) as energy source. Prolonged PARP activity can drain cellular NAD+ reserves, leading to de-regulation of important molecular
Externí odkaz:
https://doaj.org/article/156b773902e04f898047b04a65716854
Akademický článek
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Autor:
Yangchen Dhondup, Ivar Sjaastad, Øystein Sandanger, Jan Magnus Aronsen, Muhammad Shakil Ahmed, Håvard Attramadal, Alexandra Vanessa Finsen, Lili Zhang, Trine Ranheim, Katrine Alfsnes, Pål Aukrust, Geir Christensen, Arne Yndestad, Leif Erik Vinge
Publikováno v:
Mediators of Inflammation, Vol 2017 (2017)
Aim. Inflammation is important in heart failure (HF). The role of the immune receptor toll-like receptor 9 (TLR9) in HF is not understood and not investigated in diastolic HF. We investigated the role of TLR9 in a murine diastolic HF model caused by
Externí odkaz:
https://doaj.org/article/574432b69998439ab5a5f81e21132c63
CTGF/CCN2 Postconditioning Increases Tolerance of Murine Hearts towards Ischemia-Reperfusion Injury.
Autor:
Ole Jørgen Kaasbøll, Ingvild Tronstad Moe, Mohammad Shakil Ahmed, Espen Stang, Else Marie Valbjørn Hagelin, Håvard Attramadal
Publikováno v:
PLoS ONE, Vol 11, Iss 2, p e0149000 (2016)
BACKGROUND AND PURPOSE:Previous studies of ischemia-reperfusion injury (IRI) in hearts from mice with cardiac-restricted overexpression of CCN2 have shown that CCN2 increases tolerance towards IRI. The objectives of this study were to investigate to
Externí odkaz:
https://doaj.org/article/477267d77ca8411d812f4adf71f2331b