Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Harriet C. Rea"'
Autor:
GuoJing Xu, Harriet C. Rea, J. Regino Perez-Polo, Geda Unabia, Claire E. Hulsebosch, Douglas S. DeWitt, Kathia M. Johnson, Smitha K. Infante, Margaret A. Parsley
Publikováno v:
Journal of Neurotrauma. 30:727-740
Mild traumatic brain injury (mTBI), particularly mild “blast type” injuries resulting from improvised exploding devices and many sport-caused injuries to the brain, result in long-term impairment of cognition and behavior. Our central hypothesis
Autor:
Tor C. Savidge, Kizhake V. Soman, Alexander Kurosky, J. Regino Perez-Polo, Susan Stafford, Harriet C. Rea, Petri Urvil, John E. Wiktorowicz
Publikováno v:
Biochemistry. 50(25)
Cysteinyl-S-nitrosylation has emerged as an important post-translational modification affecting protein function in health and disease. Great emphasis has been placed on global, unbiased quantification of S-nitrosylated proteins due to physiologic an
Publikováno v:
International journal of developmental neuroscience : the official journal of the International Society for Developmental Neuroscience. 29(6)
Exposure to hypoxia and hyperoxia in a rodent model of perinatal ischemia results in delayed cell death and and inflammation. Hyperoxia increases oxidative stress that can trigger inflammatory cascades, neutrophil activation, and brain microvascular
Autor:
Ziming Ye, J. Regino Perez-Polo, David J. McAdoo, Jingxin Qiu, Clarie E. Hulsebosch, Guo Ying Xu, Karin N. Westlund, Olivera Nesic, Harriet C. Rea
Publikováno v:
Journal of neurotrauma. 18(11)
After contusion-derived spinal cord injury, (SCI) there is localized tissue disruption and energy failure that results in early necrosis and delayed apoptosis, events that contribute to chronic central pain in a majority of patients. We assessed the
Autor:
J. Regino Perez-Polo, Tadahide Izumi, Thomas A. Kent, Mike Quast, Harriet C. Rea, Jinqua Wei, Sankar Mitra, Michael Edwards
Publikováno v:
Scopus-Elsevier
Cerebral ischemia and the aftermath of reperfusion form a hypoxic/hyperoxic sequence of events that can trigger oxidative stress response cascades in neurons of the central nervous system. After transient ischemia there is an increase in intracellula
Externí odkaz:
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