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Publikováno v:
In Biomedicine & Pharmacotherapy September 2017 93:1238-1245
Autor:
Chandraprabha Vineetha, Radhakrishnan, Anitha Geetha Raj, John, Devipriya, Padmakumar, Sreelatha Mahitha, Mohanan, Hariharan, Sreedharan
Publikováno v:
International Journal of Laboratory Hematology; Feb2024, Vol. 46 Issue 1, p33-41, 9p
Autor:
Ramachandran Krishna Chandran, Narayanan Geetha, Kunnathur Murugesan Sakthivel, Raveendran Suresh Kumar, Kumarapillai Mohanan Nair Jagathnath Krishna, Hariharan Sreedharan
Publikováno v:
Frontiers in Oncology, Vol 9 (2019)
The emergence of additional chromosomal abnormalities (ACAs) in Philadelphia chromosome/BCR-ABL1 positive chronic myeloid leukemia (CML), is considered to be a feature of disease evolution. However, their frequency of incidence, impact on prognosis a
Externí odkaz:
https://doaj.org/article/6d051bd32026448ca60a16ea3d437225
Autor:
Remya Remani Sathyan, Gopakumar Chandrasekhara Menon, Hari Prasad, Hariharan Sreedharan, Duraisamy Jude Hemanth
Publikováno v:
International Journal of Imaging Systems and Technology. 32:2017-2033
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::ef535eb0042b22526db5f633df98f264
https://doi.org/10.1002/ima.22741
https://doi.org/10.1002/ima.22741
Autor:
Santhi Sarojam, Sangeetha Vijay, Sureshkumar Raveendran, Jayadevan Sreedharan, Geetha Narayanan, Hariharan Sreedharan
Publikováno v:
Middle East Journal of Cancer, Vol 5, Iss 4, Pp 185-196 (2014)
Background: Fms-like tyrosine kinase 3 is a tyrosine kinase receptor that plays an important role in proliferation and differentiation of hematopoietic stem cells. Internal tandem duplication and tyrosine kinase domain mutation are the two most com
Externí odkaz:
https://doaj.org/article/3e6c0bcc7f254950a182360fa52e0ac7
Autor:
Narayanan Geetha, Kumarapillai Mohanan Nair Jagathnath Krishna, Kunnathur Murugesan Sakthivel, Hariharan Sreedharan, Thampirajan Vimaladevi Akhila Raj, Ramachandran Krishna Chandran
Publikováno v:
Journal of Environmental Pathology, Toxicology and Oncology. 40:51-64
The clinical implications of cohesin gene complex mutation in acute myeloid leukemia (AML) are not well characterized. In the present study, a cohort of 152 de novo unselected adult AML patients underwent conventional and molecular cytogenetic analys
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e1a8efb78e260e02285b0138943abb05
https://doi.org/10.1615/jenvironpatholtoxicoloncol.2020035890
https://doi.org/10.1615/jenvironpatholtoxicoloncol.2020035890
Publikováno v:
Annals of Saudi Medicine, Vol 33, Iss 6, Pp 619-622 (2013)
Chromosomal abnormalities/genetic mutations associated with hematological malignancies alter the structure and function of genes controlling cell proliferation and differentiation through multiple and complex pathways, resulting different clinical ou
Externí odkaz:
https://doaj.org/article/678aa1b4cdde4a399d3ac73ea9d7836e
Autor:
Narayanan Geetha, Ramachandran Krishna Chandran, Raveendran Suresh Kumar, Kumarapillai Mohanan Nair Jagathnath Krishna, Kunnathur Murugesan Sakthivel, Hariharan Sreedharan
Publikováno v:
Blood Cells, Molecules, and Diseases. 77:51-60
The molecular mechanisms responsible for disease progression of CML are not conclusive. The main functional changes associated with disease evolution in CML was high proliferation rate, decreased apoptosis, blockade of differentiation, and strong res
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1eb6192db84fddcbef740ac5dffbf852
https://doi.org/10.1016/j.bcmd.2019.03.004
https://doi.org/10.1016/j.bcmd.2019.03.004
Autor:
Preethi Gopinath, Thampirajan Vimaladevi Akhila Raj, Kunnathur Murugesan Sakthivel, Jagathnath Krishna Kumarapillai Mohanan Nair, Ramachandran Krishna Chandran, Hariharan Sreedharan, Geetha Priya, Narayanan Geetha, Chandran Geetha Aswathy
Publikováno v:
Gene. 686:85-91
Identification of BCR-ABL1 fusion gene amplification status is critically important in the effective management of chronic myelogenous leukemia (CML) patients. Earlier reports suggested that overexpression of BCR-ABL1 either through amplification of
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7ed809a81d1f5061f133879eafffa522
https://doi.org/10.1016/j.gene.2018.11.005
https://doi.org/10.1016/j.gene.2018.11.005