Direct Promoter Repression by BCL11A Controls the Fetal to Adult Hemoglobin Switch.

Autor: Liu N; Cancer and Blood Disorders Center, Dana Farber Cancer Institute and Boston Children's Hospital, Harvard Medical School, Boston, MA, USA., Hargreaves VV; Cancer and Blood Disorders Center, Dana Farber Cancer Institute and Boston Children's Hospital, Harvard Medical School, Boston, MA, USA., Zhu Q; Department of Biostatistics and Computational Biology, Dana Farber Cancer Institute and Harvard T.H. Chan School of Public Health, Boston, MA, USA., Kurland JV; Division of Genetics, Department of Medicine, Brigham and Women's Hospital, Harvard Medical School, Boston, MA, USA., Hong J; Cancer and Blood Disorders Center, Dana Farber Cancer Institute and Boston Children's Hospital, Harvard Medical School, Boston, MA, USA., Kim W; Cancer and Blood Disorders Center, Dana Farber Cancer Institute and Boston Children's Hospital, Harvard Medical School, Boston, MA, USA., Sher F; Cancer and Blood Disorders Center, Dana Farber Cancer Institute and Boston Children's Hospital, Harvard Medical School, Boston, MA, USA., Macias-Trevino C; Cancer and Blood Disorders Center, Dana Farber Cancer Institute and Boston Children's Hospital, Harvard Medical School, Boston, MA, USA., Rogers JM; Division of Genetics, Department of Medicine, Brigham and Women's Hospital, Harvard Medical School, Boston, MA, USA; Committee on Higher Degrees in Biophysics, Harvard University, Cambridge, MA, USA., Kurita R; Cell Engineering Division, RIKEN Bioresource Center, Tsukuba, Japan., Nakamura Y; Cell Engineering Division, RIKEN Bioresource Center, Tsukuba, Japan., Yuan GC; Department of Biostatistics and Computational Biology, Dana Farber Cancer Institute and Harvard T.H. Chan School of Public Health, Boston, MA, USA., Bauer DE; Cancer and Blood Disorders Center, Dana Farber Cancer Institute and Boston Children's Hospital, Harvard Medical School, Boston, MA, USA., Xu J; Children's Medical Center Research Institute, Department of Pediatrics, University of Texas at Southwestern Medical Center, Dallas, TX, USA., Bulyk ML; Division of Genetics, Department of Medicine, Brigham and Women's Hospital, Harvard Medical School, Boston, MA, USA; Committee on Higher Degrees in Biophysics, Harvard University, Cambridge, MA, USA; Department of Pathology, Brigham and Women's Hospital, Harvard Medical School, Boston, MA, USA., Orkin SH; Cancer and Blood Disorders Center, Dana Farber Cancer Institute and Boston Children's Hospital, Harvard Medical School, Boston, MA, USA; Howard Hughes Medical Institute, Boston, MA, USA. Electronic address: stuart_orkin@dfci.harvard.edu.

Publikováno v: Cell [Cell] 2018 Apr 05; Vol. 173 (2), pp. 430-442.e17. Date of Electronic Publication: 2018 Mar 29.

Interaction between the Msh2 and Msh6 nucleotide-binding sites in the Saccharomyces cerevisiae Msh2-Msh6 complex.

Autor: Hargreaves VV; Department of Medicine and Cellular, Cancer Center, Ludwig Institute for Cancer Research, University of California San Diego School of Medicine, La Jolla, California 92093-0669, USA., Shell SS, Mazur DJ, Hess MT, Kolodner RD

Publikováno v: The Journal of biological chemistry [J Biol Chem] 2010 Mar 19; Vol. 285 (12), pp. 9301-10. Date of Electronic Publication: 2010 Jan 20.

The salt dependence of the interferon regulatory factor 1 DNA binding domain binding to DNA reveals ions are localized around protein and DNA.

Autor: Hargreaves VV; Department of Biology, Johns Hopkins University, Baltimore, Maryland 21218, USA., Schleif RF

Publikováno v: Biochemistry [Biochemistry] 2008 Apr 01; Vol. 47 (13), pp. 4119-28. Date of Electronic Publication: 2008 Mar 07.