The significance of glutaredoxins for diabetes mellitus and its complications.

Autor: Zhou M; Clinical Research Unit, Medical Clinic and Polyclinic III, Center of Internal Medicine, Justus Liebig University, Giessen, Germany., Hanschmann EM; Experimental and Translational Research, Department of Otorhinolaryngology, University Hospital Essen, Essen, Germany., Römer A; Clinical Research Unit, Medical Clinic and Polyclinic III, Center of Internal Medicine, Justus Liebig University, Giessen, Germany., Linn T; Clinical Research Unit, Medical Clinic and Polyclinic III, Center of Internal Medicine, Justus Liebig University, Giessen, Germany., Petry SF; Clinical Research Unit, Medical Clinic and Polyclinic III, Center of Internal Medicine, Justus Liebig University, Giessen, Germany. Electronic address: sebastian.petry@innere.med.uni-giessen.de.

Publikováno v: Redox biology [Redox Biol] 2024 May; Vol. 71, pp. 103043. Date of Electronic Publication: 2024 Jan 20.

Redox signaling and metabolism in Alzheimer's disease.

Autor: Holubiec MI; IBioBA-MPSP Instituto de Investigación en Biomedicina de Buenos Aires, Partner Institute of the Max Planck Society, Buenos Aires, Argentina., Gellert M; Institute for Medical Biochemistry and Molecular Biology, University Medicine Greifwald, University Greifswald, Greifswald, Germany., Hanschmann EM; Independent Researcher Essen, Germany.

Publikováno v: Frontiers in aging neuroscience [Front Aging Neurosci] 2022 Nov 03; Vol. 14, pp. 1003721. Date of Electronic Publication: 2022 Nov 03 (Print Publication: 2022).

Thiol Modifications in the Extracellular Space-Key Proteins in Inflammation and Viral Infection.

Autor: Brücksken KA; Department of Neurology, Medical Faculty, Heinrich-Heine University, Düsseldorf, Germany., Loreto Palacio P; Department of Neurology, Medical Faculty, Heinrich-Heine University, Düsseldorf, Germany., Hanschmann EM; Department of Neurology, Medical Faculty, Heinrich-Heine University, Düsseldorf, Germany.

Publikováno v: Frontiers in immunology [Front Immunol] 2022 Jun 27; Vol. 13, pp. 932525. Date of Electronic Publication: 2022 Jun 27 (Print Publication: 2022).

GDAP1 loss of function inhibits the mitochondrial pyruvate dehydrogenase complex by altering the actin cytoskeleton.

Autor: Wolf C; Institute of Molecular Medicine, University Medical Center Mainz, Mainz, Germany., Pouya A; Institute of Molecular Medicine, University Medical Center Mainz, Mainz, Germany., Bitar S; Institute of Molecular Medicine, University Medical Center Mainz, Mainz, Germany., Pfeiffer A; Institute of Molecular Medicine, University Medical Center Mainz, Mainz, Germany., Bueno D; Institute of Molecular Medicine, University Medical Center Mainz, Mainz, Germany., Rojas-Charry L; Institute of Molecular Medicine, University Medical Center Mainz, Mainz, Germany., Arndt S; Institute for Immunology, University Medical Center Mainz, Mainz, Germany., Gomez-Zepeda D; Institute for Immunology, University Medical Center Mainz, Mainz, Germany., Tenzer S; Institute for Immunology, University Medical Center Mainz, Mainz, Germany., Bello FD; Institute of Molecular Medicine, University Medical Center Mainz, Mainz, Germany.; Department of Biology, University of Padova, Padova, Italy.; Department of Biomedical Sciences, University of Padova, Padova, Italy., Vianello C; Institute of Molecular Medicine, University Medical Center Mainz, Mainz, Germany.; Department of Biology, University of Padova, Padova, Italy.; Department of Biomedical Sciences, University of Padova, Padova, Italy., Ritz S; Institute of Molecular Biology (IMB) gGmbH, Mainz, Germany., Schwirz J; Institute of Molecular Biology (IMB) gGmbH, Mainz, Germany., Dobrindt K; Institute of Reconstructive Neurobiology, University of Bonn Medical Faculty and University Hospital Bonn, Bonn, Germany., Peitz M; Institute of Reconstructive Neurobiology, University of Bonn Medical Faculty and University Hospital Bonn, Bonn, Germany.; Cell Programming Core Facility, University of Bonn School of Medicine, Bonn, Germany., Hanschmann EM; Department of Neurology, Medical Faculty, Heinrich-Heine University, Düsseldorf, Germany., Mencke P; Translational Neuroscience, Luxembourg Centre for Systems Biomedicine (LCSB), University of Luxembourg, Belvaux, Luxembourg., Boussaad I; Translational Neuroscience, Luxembourg Centre for Systems Biomedicine (LCSB), University of Luxembourg, Belvaux, Luxembourg.; Disease Modeling and Screening Platform (DMSP), Luxembourg Centre of Systems Biomedicine (Biomedicine), University of Luxembourg and Luxembourg Institute of Health (LIH), Belvaux, Luxembourg., Silies M; Institute of Development Biology and Neurobiology, Johannes Gutenberg-University Mainz, Mainz, Germany., Brüstle O; Institute of Reconstructive Neurobiology, University of Bonn Medical Faculty and University Hospital Bonn, Bonn, Germany., Giacomello M; Department of Biology, University of Padova, Padova, Italy.; Department of Biomedical Sciences, University of Padova, Padova, Italy., Krüger R; Translational Neuroscience, Luxembourg Centre for Systems Biomedicine (LCSB), University of Luxembourg, Belvaux, Luxembourg.; Transversal Translational Medicine, Luxembourg Institute of Health (LIH), Strassen, Luxembourg., Methner A; Institute of Molecular Medicine, University Medical Center Mainz, Mainz, Germany. axel.methner@gmail.com.

Publikováno v: Communications biology [Commun Biol] 2022 Jun 03; Vol. 5 (1), pp. 541. Date of Electronic Publication: 2022 Jun 03.

Cofilin1 oxidation links oxidative distress to mitochondrial demise and neuronal cell death.

Autor: Hoffmann L; Institute for Pharmacology and Clinical Pharmacy, Biochemical-Pharmacological Center Marburg, University of Marburg, Karl-von Frisch Straße 2, 35043, Marburg, Germany.; Center for Mind, Brain and Behavior-CMBB, Hans-Meerwein-Straße 6, 35032, Marburg, Germany., Waclawczyk MS; Department of Neurology, Heinrich-Heine University Düsseldorf, Moorenstraße 5, 40225, Düsseldorf, Germany., Tang S; Institute for Pharmacology and Clinical Pharmacy, Biochemical-Pharmacological Center Marburg, University of Marburg, Karl-von Frisch Straße 2, 35043, Marburg, Germany.; Center for Mind, Brain and Behavior-CMBB, Hans-Meerwein-Straße 6, 35032, Marburg, Germany., Hanschmann EM; Department of Neurology, Heinrich-Heine University Düsseldorf, Moorenstraße 5, 40225, Düsseldorf, Germany., Gellert M; Institute for Medical Biochemistry and Molecular Biology, University Medicine Greifswald, Ferdinand-Sauerbruch-Straße, 17475, Greifswald, Germany., Rust MB; Center for Mind, Brain and Behavior-CMBB, Hans-Meerwein-Straße 6, 35032, Marburg, Germany.; Molecular Neurobiology Group, Institute of Physiological Chemistry, Biochemical-Pharmacological Center Marburg, University of Marburg, Karl-von Frisch Straße 2, 35043, Marburg, Germany.; DFG Research Training Group 'Membrane Plasticity in Tissue Development and Remodeling', GRK 2213, University of Marburg, 35032, Marburg, Germany., Culmsee C; Institute for Pharmacology and Clinical Pharmacy, Biochemical-Pharmacological Center Marburg, University of Marburg, Karl-von Frisch Straße 2, 35043, Marburg, Germany. culmsee@staff.uni-marburg.de.; Center for Mind, Brain and Behavior-CMBB, Hans-Meerwein-Straße 6, 35032, Marburg, Germany. culmsee@staff.uni-marburg.de.; Third Affiliated Hospital, Zhengzhou University, Zhengzhou, China. culmsee@staff.uni-marburg.de.

Publikováno v: Cell death & disease [Cell Death Dis] 2021 Oct 16; Vol. 12 (11), pp. 953. Date of Electronic Publication: 2021 Oct 16.