Zobrazeno 1 - 10
of 306
pro vyhledávání: '"Hans Oberleithner"'
Autor:
Hermann Schillers, Carmela Rianna, Jens Schäpe, Tomas Luque, Holger Doschke, Mike Wälte, Juan José Uriarte, Noelia Campillo, Georgios P. A. Michanetzis, Justyna Bobrowska, Andra Dumitru, Elena T. Herruzo, Simone Bovio, Pierre Parot, Massimiliano Galluzzi, Alessandro Podestà, Luca Puricelli, Simon Scheuring, Yannis Missirlis, Ricardo Garcia, Michael Odorico, Jean-Marie Teulon, Frank Lafont, Malgorzata Lekka, Felix Rico, Annafrancesca Rigato, Jean-Luc Pellequer, Hans Oberleithner, Daniel Navajas, Manfred Radmacher
Publikováno v:
Scientific Reports, Vol 7, Iss 1, Pp 1-9 (2017)
Abstract We present a procedure that allows a reliable determination of the elastic (Young’s) modulus of soft samples, including living cells, by atomic force microscopy (AFM). The standardized nanomechanical AFM procedure (SNAP) ensures the precis
Externí odkaz:
https://doaj.org/article/b340d2b52d6746c3a98d8f60fb8b97bf
Autor:
Hans Oberleithner, Marianne Wilhelmi
Publikováno v:
Kidney & Blood Pressure Research, Vol 41, Iss 4, Pp 355-364 (2016)
Background/Aims: A significant rise of blood pressure in response to a given salt load is a weak indication of high salt sensitivity, supposed to foster the development of arterial hypertension and related diseases in later life. In search of an alte
Externí odkaz:
https://doaj.org/article/a9efbfe3c8e142ebb8acd5f342a1dc8e
Autor:
Hans Oberleithner
Publikováno v:
Biomedical Applications ISBN: 9783110989380
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::cac757a4843eb2817a76ff16a9bf3bd5
https://doi.org/10.1515/9783110989380-022
https://doi.org/10.1515/9783110989380-022
Publikováno v:
Cellular Physiology and Biochemistry, Vol 35, Iss 3, Pp 1107-1115 (2015)
Background: Similar as in vascular endothelium the negatively charged glycocalyx of erythrocytes selectively buffers sodium. Loss of glycocalyx (i.e. loss of negative charges) leads to increased erythrocyte sodium sensitivity (ESS) quantified by a re
Externí odkaz:
https://doaj.org/article/d0d40e32125c4a2890a6f41ad09efb91
Autor:
Alexander Zarbock, Jan Rossaint, Mike Wälte, Verena Hofschröer, Otto Lindemann, Benedikt Fels, Sandra Schimmelpfennig, Albrecht Schwab, Hans Oberleithner, Karolina Najder
Publikováno v:
Journal of Molecular Medicine (Berlin, Germany)
Abstract Here we report a novel role for TRPC6, a member of the transient receptor potential (TRPC) channel family, in the CXCL1-dependent recruitment of murine neutrophil granulocytes. Representing a central element of the innate immune system, neut
Autor:
Hans Oberleithner
Publikováno v:
Biological chemistryReferences. 402(12)
Inner surfaces of blood vessels and outer surfaces of erythrocytes are coated with a negatively charged protective film of proteoglycans, which serves as an effective buffer system for the positively charged sodium ions. If this protective coating is
Publikováno v:
PLoS ONE, Vol 8, Iss 7 (2013)
Externí odkaz:
https://doaj.org/article/6cbd91bab3a249669e2d7f64fd8cea4d
Autor:
Anne Wiesinger, Wladimir Peters, Daniel Chappell, Dominik Kentrup, Stefan Reuter, Hermann Pavenstädt, Hans Oberleithner, Philipp Kümpers
Publikováno v:
PLoS ONE, Vol 8, Iss 11, p e80905 (2013)
The endothelial glycocalyx (eGC), a carbohydrate-rich layer lining the luminal side of the endothelium, regulates vascular adhesiveness and permeability. Although central to the pathophysiology of vascular barrier dysfunction in sepsis, glycocalyx da
Externí odkaz:
https://doaj.org/article/adcbc038534842fa9a6a4efe36e7b1d5
Publikováno v:
PLoS ONE, Vol 7, Iss 7, p e41520 (2012)
The release of the main vasodilator nitric oxide (NO) by the endothelial NO synthase (eNOS) is a hallmark of endothelial function. We aim at elucidating the underlying mechanism how eNOS activity depends on cortical stiffness (К(cortex)) of living e
Externí odkaz:
https://doaj.org/article/36fc7a35d50943c4bcbf5e66c5ce99bb
Publikováno v:
PLoS ONE, Vol 7, Iss 1, p e29972 (2012)
The endothelial glycocalyx (eGC) plays a pivotal role in the physiology of the vasculature. By binding plasma proteins, the eGC forms the endothelial surface layer (ESL) which acts as an interface between bloodstream and endothelial cell surface. The
Externí odkaz:
https://doaj.org/article/0d09b8ed64a040698c793b705361c0d0