Zobrazeno 1 - 8
of 8
pro vyhledávání: '"Hans‐Willi Mittruecker"'
Autor:
Annika Volmari, Katharina Foelsch, Elisabeth Zierz, Karsten Yan, Minyue Qi, Karlotta Bartels, Stephanie Kondratowicz, Marius Boettcher, Daniel Reimers, Masahiro Nishibori, Keyue Liu, Robert F. Schwabe, Ansgar W. Lohse, Samuel Huber, Hans‐Willi Mittruecker, Peter Huebener
Publikováno v:
Hepatology Communications, Vol 5, Iss 12, Pp 2104-2120 (2021)
High‐mobility group box 1 (HMGB1) is a nucleoprotein with proinflammatory functions following cellular release during tissue damage. Moreover, antibody‐mediated HMGB1 neutralization alleviates lipopolysaccharide (LPS)‐induced shock, suggesting
Externí odkaz:
https://doaj.org/article/e8ddfffaf1494235a97e00fc6451d930
Autor:
Peter Huebener, Robert F. Schwabe, K. Luecke, Annika Volmari, Hans Willi Mittruecker, Ansgar W. Lohse, B Arnold
Publikováno v:
Zeitschrift für Gastroenterologie. 56:E2-E89
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::561728d917543a6aaabacd21b7c54f0d
https://doi.org/10.1055/s-0037-1612876
https://doi.org/10.1055/s-0037-1612876
Autor:
Keyue Liu, Peter Huebener, Minyue Qi, Katharina Foelsch, Ansgar W. Lohse, Annika Volmari, Samuel Huber, Hans Willi Mittruecker, Karlotta Bartels, Masahiro Nishibori, Karsten Yan, Robert F. Schwabe, Stephanie Kondratowicz, Marius Boettcher
High-mobility group box 1 (HMGB1) is a damage-associated molecular pattern with key proinflammatory functions following tissue injury. Moreover, HMGB1 neutralization was shown to alleviate LPS-induced shock, suggesting a role for the protein as a mas
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a72e5e98cc34f6ae9b116efdf9658d47
Publikováno v:
Journal of Hepatology. 66:S641-S642
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::11f0dee9c62215817a11ebcf6b5fe99d
https://doi.org/10.1016/s0168-8278(17)31737-3
https://doi.org/10.1016/s0168-8278(17)31737-3
Publikováno v:
The Journal of Immunology. 160:545-549
Spontaneous resorption in the CBA × DBA/2 model is attributed to NK cells, macrophages, and Th1-type cytokines. In vivo depletion of NK cells by anti-asialoGM1 Ab or macrophage depletion by silicon dioxide treatment reduced abortion rates, which cou
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::c09467697fda74b8f95f1c0b31b2b1eb
https://doi.org/10.4049/jimmunol.160.2.545
https://doi.org/10.4049/jimmunol.160.2.545
Autor:
Tak W. Mak, Henrik Griesser, Ryuichi Amakawa, Thomas M. Kündig, Hans-Willi Mittruecker, Rudolf Schmits, John J.L. Simard, Anne Hakem, Arda Shahinian, Andrew Wakeham, Josef M. Penninger, Toshifumi Matsuyama, Emma Timms, Hiroaki Takimoto, Pamela S. Ohashi
Publikováno v:
Cell. 84:551-562
CD30 is found on Reed-Sternberg cells of Hodgkin's disease and on a variety of non-Hodgkin's lymphoma cells and is up-regulated on cells after Epstein-Barr virus, human T cell leukemia virus, and HIV infections. We report here that the thymus in CD30
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3da485efc6b4aa9bbf6fcba28a5cbc69
https://doi.org/10.1016/s0092-8674(00)81031-4
https://doi.org/10.1016/s0092-8674(00)81031-4
Autor:
Mi Ryu, Hans-Joachim Anders, Heni Eka Susanti, Maciej Lech, Marc Weidenbusch, Murthy N. Darisipudi, Tak W. Mak, Onkar P. Kulkarni, Hans-Willi Mittruecker
Publikováno v:
Journal of the American Society of Nephrology : JASN. 22(8)
The IFN-regulatory factors IRF1, IRF3, IRF5, and IRF7 modulate processes involved in the pathogenesis of systemic lupus and lupus nephritis, but the contribution of IRF4, which has multiple roles in innate and adaptive immunity, is unknown. To determ
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5a8aab48d4861b683382d2370be8b322
https://pubmed.ncbi.nlm.nih.gov/21742731
https://pubmed.ncbi.nlm.nih.gov/21742731
Autor:
Maciej Lech, Christoph Römmele, Tak W. Mak, Hans-Willi Mittruecker, Hans-Joachim Anders, Saraswati Lassen
Publikováno v:
Journal of immunology (Baltimore, Md. : 1950). 185(3)
Ischemia reperfusion (IR) activates TLRs causing subsequent sterile inflammation, for example in postischemic acute renal failure. Unexpectedly, TLR signaling predominates in intrinsic renal cells and not in intrarenal APCs in the postischemic kidney
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c7913e33a2efb1f333625241651ed8b8
https://pubmed.ncbi.nlm.nih.gov/20601597
https://pubmed.ncbi.nlm.nih.gov/20601597