Zobrazeno 1 - 10
of 13
pro vyhledávání: '"Hanna Pelli"'
Publikováno v:
Scandinavian Journal of Surgery. 98:221-224
Background and Aims: The objective of this study was to measure the association of Helicobacter pylori infection with alcoholic acute pancreatitis. Material and Methods: This study comprised of 50 patients with their first alcoholic pancreatitis and
Autor:
Hanna Pelli, Riitta Lappalainen-Lehto, Isto Nordback, Juhani Sand, Anneli Piironen, Satu Järvinen
Publikováno v:
Pancreatology. 9:245-251
Acute alcoholic pancreatitis (AAP) recurs in up to half of the patients, continuous alcohol consumption being an important risk factor. Changes in pancreatic function and morphology after acute pancreatitis have been characterized previously, but the
Autor:
E. Richard Stanley, James DeGregori, Jeffery S. Tessem, Xiao-Hua Zong, Hanna Pelli, Jan Jensen, Jan Nygaard Jensen, Xuming Dai
Publikováno v:
Diabetes. 57:1605-1617
OBJECTIVE— Chronic pancreatitis, characterized by pancreatic exocrine tissue destruction with initial maintenance of islets, eventually leads to insulin-dependent diabetes in most patients. Mice deficient for the transcription factors E2F1 and E2F2
Publikováno v:
Scandinavian Journal of Gastroenterology. 43:614-621
In an earlier retrospective study we showed that 46% of patients with acute alcoholic pancreatitis had recurrent attacks within 10-20 years, about 30% having a recurrence during the first 3 years. The aim of this prospective follow-up study was to de
Autor:
Sari Räty, Sanna T.H. Uotila, Anneli Piironen, Karl-Heinz Herzig, Juhani Sand, Isto Nordback, Hanna Pelli, Mohan Babu
Publikováno v:
Scandinavian Journal of Gastroenterology. 42:1000-1005
Mutations in the cationic trypsinogen gene (PRSS1) have been linked with hereditary pancreatitis (HP). A change in R122H in the third exon is one of the mutations most frequently associated with HP. A mutation N34S in the serine protease inhibitor Ka
Duodenal diazepam‐binding protein expression and plasma cholecystokinin after alcoholic pancreatitis
Publikováno v:
Scandinavian Journal of Clinical and Laboratory Investigation. 66:677-684
After the first acute alcohol-induced pancreatitis (AAIP) episode 46 % of patients will have a recurrent attack, but the pathophysiology is unclear. The hyperstimulation of the pancreas with cholecystokinin (CCK) induces acute pancreatitis. Alcohol i
Publikováno v:
Scandinavian Journal of Gastroenterology. 40:1235-1239
Unlike patients with alcoholic hepatitis, patients with acute alcoholic pancreatitis seldom come into the hospital in an intoxicated state. Long-term history of heavy drinking induces increases in the serum pancreatic enzymes and pancreatitis-associa
Publikováno v:
Duodecim; laaketieteellinen aikakauskirja. 125(11)
70% of pancreatitis cases are considered to be induced by alcohol. Half of those fallen ill with alcohol-induced acute pancreatitis will have relapses. A prospective follow-up study showed that the level of dependence on alcohol constitutes the most
Publikováno v:
Gastroenterology. 136(3)
Background & Aims In the long term, half of patients with their first alcohol-associated acute pancreatitis (AP) develop acute recurrence, alcohol consumption being the main risk factor. None of the recent national or international guidelines for tre
Autor:
Marko Lempinen, Gediminas Kiudelis, Esko Kemppainen, Johanna Laukkarinen, Sari Räty, Hanna Pelli, Juhani Sand, Isto Nordback, Caj Haglund
Publikováno v:
The Journal of clinical endocrinology and metabolism. 92(11)
Context: Earlier, we have shown an increased prevalence of previously diagnosed hypothyroidism in common bile duct (CBD) stone patients and a delayed emptying of the biliary tract in hypothyroidism, explained partly by the missing prorelaxing effect