Zobrazeno 1 - 10
of 12
pro vyhledávání: '"Hani'ah Abdullah"'
Publikováno v:
Frontiers in Microbiology, Vol 12 (2021)
Respiratory syncytial virus (RSV) or measles virus (MeV) infection modifies host responses through small non-coding RNA (sncRNA) expression. We show that RSV or MeV infection of neuronal cells induces sncRNAs including various microRNAs and transfer
Externí odkaz:
https://doaj.org/article/8af30236fc6848d4b07f7f92b7fb77f4
Autor:
Judith M. Kennedy, J.A. Philip Earle, Shadia Omar, Hani’ah Abdullah, Ole Nielsen, Melody E. Roelke-Parker, S. Louise Cosby
Publikováno v:
Viruses, Vol 11, Iss 10, p 944 (2019)
Canine distemper virus (CDV) and phocine distemper (PDV) are closely-related members of the Paramyxoviridae family, genus morbillivirus, in the order Mononegavirales. CDV has a broad host range among carnivores. PDV is thought to be derived from CDV
Externí odkaz:
https://doaj.org/article/f3f72f08812c4d72ad75e0a8f7c60800
Autor:
Hani'ah Abdullah, S. Louise Cosby, Melody E. Roelke-Parker, Shadia Omar, Judith M. Kennedy, Ole Nielsen, J.A. Philip Earle
Publikováno v:
Viruses
Viruses, Vol 11, Iss 10, p 944 (2019)
Viruses, Vol 11, Iss 10, p 944 (2019)
Canine distemper virus (CDV) and phocine distemper (PDV) are closely-related members of the Paramyxoviridae family, genus morbillivirus, in the order Mononegavirales. CDV has a broad host range among carnivores. PDV is thought to be derived from CDV
Publikováno v:
Abdullah, H, Heaney, L G, Cosby, S L & McGarvey, L P A 2014, ' Rhinovirus upregulates transient receptor potential channels in a human neuronal cell line: Implications for respiratory virus-induced cough reflex sensitivity ' Thorax, vol 69, no. 1, pp. 46-54 . DOI: 10.1136/thoraxjnl-2013-203894
Background: The mechanism underlying respiratory virus-induced cough hypersensitivity is unknown. Upregulation of airway neuronal receptors responsible for sensing physical and chemical stimuli is one possibility, and the transient receptor potential
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5031e503ab1ab216cb60bbe9d1c4748f
https://pure.qub.ac.uk/ws/files/5046440/Abdullah_et_al._Thorax_paper_revised_July_2013.pdf
https://pure.qub.ac.uk/ws/files/5046440/Abdullah_et_al._Thorax_paper_revised_July_2013.pdf
Publikováno v:
Articles
Small numbers of brain endothelial cells (BECs) are infected in children with neurologic complications of measles virus (MV) infection. This may provide a mechanism for virus entry into the central nervous system, but the mechanisms are unclear. Both
Autor:
Michael D. Shields, Liam G Heaney, JC Parker, Sara Louise Cosby, Lorcan McGarvey, Hani'ah Abdullah
Publikováno v:
B69. AIRWAY STRUCTURE AND FUNCTION.
Publikováno v:
D38. TRANS-MEMBRANE COMMUNICATION: RECEPTORS AND CHANNELS.
Publikováno v:
Neuropathology and Applied Neurobiology
Neuropathology and Applied Neurobiology, Wiley, 2009, 35 (5), pp.473-86. ⟨10.1111/j.1365-2990.2009.01023.x⟩
Neuropathology and Applied Neurobiology, 2009, 35 (5), pp.473-86. ⟨10.1111/j.1365-2990.2009.01023.x⟩
Neuropathology and Applied Neurobiology, Wiley, 2009, 35 (5), pp.473-86. ⟨10.1111/j.1365-2990.2009.01023.x⟩
Neuropathology and Applied Neurobiology, 2009, 35 (5), pp.473-86. ⟨10.1111/j.1365-2990.2009.01023.x⟩
International audience; AIMS: Infection of the mouse central nervous system with wild type (WT) and vaccine strains of measles virus (MV) results in lack of clinical signs and limited antigen detection. It is considered that cell entry receptors for
Autor:
Susan Stokesberry, Liam Polley, Alexander Zholos, Sadaf Ashraf, Hani'ah Abdullah, Lorcan McGarvey, David F. Choy, Mary K. McGahon, Liam G Heaney, Stephen McQuaid, Joe Arron, Tim J. Warke, Richard W. Costello, Claire A. Butler, Tim M. Curtis, Peter Bradding, Madeleine Ennis
Publikováno v:
Journal of Allergy and Clinical Immunology. 133:704-712.e4
Background The airway epithelium is exposed to a range of physical and chemical irritants in the environment that are known to trigger asthma. Transient receptor potential (TRP) cation channels play a central role in sensory responses to noxious phys