Zobrazeno 1 - 10
of 27
pro vyhledávání: '"Han-I Lin"'
Autor:
Yu-Che Cheng, Ching-Ying Huang, Ming-Ching Ho, Yu-Hung Hsu, Shih-Han Syu, Huai-En Lu, Han-I Lin, Chin-Hsien Lin, Patrick C.H. Hsieh
Publikováno v:
Stem Cell Research, Vol 28, Iss , Pp 1-5 (2018)
Leucine rich repeat kinase (LRRK2) is the most prevalent genetic cause for Parkinson's disease. LRRK2 p.G2385R is an Asian specific genetic risk factor for sporadic Parkinson's disease. We generated two induced pluripotent stem cells (iPSCs), IBMS-iP
Externí odkaz:
https://doaj.org/article/f1d5cc0ddc224aa8b86c5b0dbe21abff
Autor:
Chin-Hsien Lin, Yu-Che Cheng, Han-I Lin, Ming-Ching Ho, Yu-Hung Hsu, Cheng-Hao Wen, Hui-Wen Ko, Huai-En Lu, Ching-Ying Huang, Patrick C.H. Hsieh
Publikováno v:
Stem Cell Research, Vol 25, Iss C, Pp 123-127 (2017)
Parkinson's disease (PD) is a progressive neurodegenerative disorder caused by interactions between genetic and environmental factors. Leucine rich repeat kinase (LRRK2) is the most prevalent mutation in autosomal-dominant inheritance of PD. Here, we
Externí odkaz:
https://doaj.org/article/b66f18682c7a433d8fb30e601c600330
Autor:
Han-I Lin, Yu-Che Cheng, Hui-Wen Ko, Cheng-Hao Wen, Huai-En Lu, Ching-Ying Huang, Patrick C.H. Hsieh, Chin-Hsien Lin
Publikováno v:
Stem Cell Research, Vol 37, Iss , Pp - (2019)
Leucine rich repeat kinase 2 (LRRK2) is the causative gene for autosomal-dominant familial forms of Parkinson's disease (PD). Here, we generated induced pluripotent stem cells (iPSCs) from the peripheral blood mononuclear cells of a female patient wi
Externí odkaz:
https://doaj.org/article/5ad19269e17a49cc91d5d418cb84b93e
Autor:
Yu-Che Cheng, Han-I Lin, Shih-Han Syu, Huai-En Lu, Ching-Ying Huang, Chin-Hsien Lin, Patrick C.H. Hsieh
Publikováno v:
Stem Cell Research, Vol 37, Iss , Pp - (2019)
A recessive mutation in PLA2G6, which is known to cause a heterogeneous neurodegenerative clinical spectrum, has recently been shown to be responsible for autosomal-recessive familial forms of Parkinson's disease (PD). Here, we generated induced plur
Externí odkaz:
https://doaj.org/article/4e1b56e631194aaaa0735ffb65900a4d
95
With the rapid development of wireless networking technology and the great growth of service demand, accurate wireless location estimation has gained considerable attention. Most wireless location system may suffer from non-line-of-sight (NLO
With the rapid development of wireless networking technology and the great growth of service demand, accurate wireless location estimation has gained considerable attention. Most wireless location system may suffer from non-line-of-sight (NLO
Externí odkaz:
http://ndltd.ncl.edu.tw/handle/b8hcv8
Autor:
Pin-Shiuan Chen, Meng-Chen Wu, Chun-Hwei Tai, Yung-Yee Chang, Min-Yu Lan, Ying-Fa Chen, Han-I Lin, Ni-Chung Lee, Chin-Hsien Lin
Publikováno v:
Parkinsonism & Related Disorders. 107:105294
Publikováno v:
Stem Cell Research. 63:102856
Aceruloplasminemia is a rare autosomal recessive disorder caused by mutations in the CP gene, encoding the copper-binding protein ceruloplasmin. A mutation in the CP gene results in brain and systemic iron overload, which is classified as a rare subt
Autor:
Patrick C.H. Hsieh, Chin-Hsien Lin, Ching-Ying Huang, Ming-Ching Ho, Yu-Che Cheng, Shih-Han Syu, Han-I Lin, Huai-En Lu, Yu-Hung Hsu
Publikováno v:
Stem Cell Research, Vol 28, Iss, Pp 1-5 (2018)
Leucine rich repeat kinase (LRRK2) is the most prevalent genetic cause for Parkinson's disease. LRRK2 p.G2385R is an Asian specific genetic risk factor for sporadic Parkinson's disease. We generated two induced pluripotent stem cells (iPSCs), IBMS-iP
Publikováno v:
Parkinsonism & Related Disorders. 62:236-238
Autor:
Patrick C.H. Hsieh, Ming Ching Ho, Huai En Lu, Cheng Hao Wen, Han I. Lin, Yu Hung Hsu, Chin-Hsien Lin, Yu Che Cheng, Ching-Ying Huang, Hui Wen Ko
Publikováno v:
Stem Cell Research, Vol 25, Iss C, Pp 123-127 (2017)
Parkinson's disease (PD) is a progressive neurodegenerative disorder caused by interactions between genetic and environmental factors. Leucine rich repeat kinase (LRRK2) is the most prevalent mutation in autosomal-dominant inheritance of PD. Here, we