Zobrazeno 1 - 10
of 44
pro vyhledávání: '"Hamid Gholami Pourbadie"'
Autor:
Samira Choopani, Bahereh Kiani, Shayan Aliakbari, Jalal Babaie, Majid Golkar, Hamid Gholami Pourbadie, Mohammad Sayyah
Publikováno v:
Scientific Reports, Vol 13, Iss 1, Pp 1-16 (2023)
Abstract Investigating long-term potentiation (LTP) in disease models provides essential mechanistic insight into synaptic dysfunction and relevant behavioral changes in many neuropsychiatric and neurological diseases. Toxoplasma (T) gondii is an int
Externí odkaz:
https://doaj.org/article/04f3a7e81e92487f87fc7a4146ad3b40
Autor:
Arezou Lari, Hamid Gholami Pourbadie, Ali Sharifi-Zarchi, Maryam Akhtari, Leila Nejatbakhsh Samimi, Ahmadreza Jamshidi, Mahdi Mahmoudi
Publikováno v:
BMC Musculoskeletal Disorders, Vol 22, Iss 1, Pp 1-9 (2021)
Abstract Background Ankylosing spondylitis (AS) is an autoimmune rheumatic disease. Few candidate gene associations have been reported for AS and the current understanding of its pathogenesis remains still poor. Thus, the exact mechanism of AS is nee
Externí odkaz:
https://doaj.org/article/12e430b47a324d288a6827c7447cc272
Autor:
Arezou Lari, Hamid Gholami Pourbadie, Ali Sharifi-Zarchi, Saeed Aslani, Leila Nejatbakhsh Samimi, Ahmadreza Jamshidi, Mahdi Mahmoudi
Publikováno v:
Iranian Journal of Allergy, Asthma and Immunology, Vol 20, Iss 5 (2021)
Ankylosing spondylitis (AS) is a systemic inflammatory disorder of joints and entheses. Recent studies have reported an increased prevalence of dementia in AS patients. However, data for exploring the association between dementia and AS remain uncert
Externí odkaz:
https://doaj.org/article/55c414578bf84deabf02a3c522558c01
Autor:
Sayed Hamid Reza Mozhgani, Sadegh Azimzadeh Jamalkandi, Hamid Gholami Pourbadie, Mehdi Mirzaie, Farsheed Noorbakhsh, Behrouz Vaziri, Alireza Gholami, Naser Ansari-Pour, Mohieddin Jafari
Publikováno v:
Frontiers in Microbiology, Vol 7 (2016)
The prototypical neurotropic virus, rabies, is a member of the Rhabdoviridae family that causes lethal encephalomyelitis. Although there have been a plethora of studies investigating the etiological mechanism of the rabies virus and many precautionar
Externí odkaz:
https://doaj.org/article/d87a3c7f34354d7480e696e05178ccfc
Autor:
Hamid Gholami Pourbadie, Nima Naderi, Nasrin Mehranfard, Mahyar Janahmadi, Fariba Khodagholi, Fereshteh Motamedi
Publikováno v:
PLoS ONE, Vol 10, Iss 2, p e0117555 (2015)
The entorhinal cortex (EC) is one of the earliest affected brain regions in Alzheimer's disease (AD). EC-amyloid pathology induces synaptic failure in the dentate gyrus (DG) with resultant behavioral impairment, but there is little known about its im
Externí odkaz:
https://doaj.org/article/11d567fa59a840f3a33cf34c14c8a158
Autor:
Soudabeh, Naderi, Fariba, Khodagholi, Hamid Gholami, Pourbadie, Nima, Naderi, Shahrbanoo, Rafiei, Mahyar, Janahmadi, Fatemeh, Sayehmiri, Fereshteh, Motamedi
Publikováno v:
NeuroToxicology. 94:71-86
Neuronal cell death as a prominent pathological feature contributes to cognitive decline and memory loss in Alzheimer's disease. We investigated the role of two forms of cell death pathways, ferroptosis and necroptosis, and their interactions followi
Autor:
Soheil, Ghassemi, Hamid Gholami, Pourbadie, Christophe, Prehaud, Monique, Lafon, Mohammad, Sayyah
Publikováno v:
Brain Research Bulletin
Brain Research Bulletin, 2022, 185, pp.107-116. ⟨10.1016/j.brainresbull.2022.05.001⟩
Brain Research Bulletin, 2022, 185, pp.107-116. ⟨10.1016/j.brainresbull.2022.05.001⟩
International audience; Rabies is a lethal infectious disease caused by rabies virus (RABV). Fear and anxiety are the distinguished symptoms in rabies patients. Fusion of RABV envelope glycoprotein (RVG) to host cell membrane initiates rabies pathoge
Autor:
Shokufeh Beglari, Fariba Khodagholi, Hamid Gholami Pourbadie, Alireza Iranbakhsh, Mahdi Rohani
Publikováno v:
Bioremediation Journal. :1-12
Autor:
Soudabeh Naderi, Fereshteh Motamedi, Hamid Gholami Pourbadie, Shahrbanoo Rafiei, Fariba Khodagholi, Nima Naderi, Mahyar Janahmadi
Alzheimer’s disease (AD) is a progressive neurodegenerative disease that is the main form of dementia. Abnormal deposition of amyloid-beta (Aβ) peptides in neurons and synapses cause neuronal loss and cognitive deficits. We have previously reporte
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::9195394bb97904ffd709aac38a9c53da
https://doi.org/10.21203/rs.3.rs-2707199/v1
https://doi.org/10.21203/rs.3.rs-2707199/v1
Autor:
Shahrbanoo Rafiei, Fariba Khodagholi, Leila Dargahi, Fereshteh Motamedi, Hamid Gholami Pourbadie
Publikováno v:
Basic and Clinical Neuroscience Journal. :1-23
Peroxisomes are the essential organelles in lipid metabolism. They contain enzymes for β-oxidation of very-long-chain fatty acids, which cannot break down in mitochondria. A reduced expression in hepatic Acyl-CoA oxidase1 (ACOX1), a peroxisome β-ox