Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Hajna Péterfy"'
Autor:
József Prechl, Krisztián Papp, Zoltán Hérincs, Hajna Péterfy, Veronika Lóránd, Zoltán Szittner, Andone Estonba, Paolo Rovero, Ilaria Paolini, Jokin Del Amo, Maria Uribarri, Maria Claudia Alcaro, Otsanda Ruiz-Larrañaga, Paola Migliorini, László Czirják
Publikováno v:
PLoS ONE, Vol 11, Iss 3, p e0150685 (2016)
Systemic lupus erythematosus is a chronic autoimmune disease with multifactorial ethiopathogenesis. The complement system is involved in both the early and late stages of disease development and organ damage. To better understand autoantibody mediate
Externí odkaz:
https://doaj.org/article/a890a6da11714206b50bfc1b5d70ef13
Autor:
E. Kovács, Eszter Tóth, Éva Katona, István Édes, László Balogh, Róbert Gábor Kiss, László Muszbek, Nóra Homoródi, Zsuzsanna Bereczky, Hajna Péterfy
Publikováno v:
Thrombosis Research. 133(5):811-816
Background Aspirin, a commonly used antiplatelet agent, blocks platelet thromboxane A 2 (TXA 2 ) formation from arachidonic acid (AA) by acetylating platelet cyclooxygenase-1 (COX-1). Laboratory methods currently used to detect this antiplatelet effe
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::780849102ec51ed77d2725c68dd16845
Autor:
Zsuzsanna Bereczky, István Édes, László Muszbek, E. Kovács, Nóra Homoródi, Eszter Tóth, Éva Katona, Róbert Gábor Kiss, Hajna Péterfy, László Balogh
Publikováno v:
Thrombosis Research. 131:320-324
Background Aspirin is widely used in the prevention of acute atherothrombotic complications. It acetylates Ser529 residue in cyclooxygenase-1 (COX-1) and prevents thromboxane A2 (TXA2) formation from arachidonic acid (AA) in platelets. Laboratory met
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::dda02eb15f17a6d06ead84c06cf1b4b4
https://doi.org/10.1016/j.thromres.2013.01.033
https://doi.org/10.1016/j.thromres.2013.01.033
Publikováno v:
International immunology. 20(10)
A peptide with the natural sequence derived from the complement component C3a, designated C3a7, and C3a9, having a modified sequence of that, was previously shown to inhibit the high-affinity IgER (FcepsilonRI)-induced secretory response of both muco
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5376773a96cd5d7176a63a0fd2625410
https://pubmed.ncbi.nlm.nih.gov/18653698
https://pubmed.ncbi.nlm.nih.gov/18653698
Publikováno v:
Immunology letters. 92(1-2)
It is known for more than 25 years that the complement-derived anaphylatoxic peptides, C3a, C4a and C5a are potent activators of basophils and certain types of mast cells. Although tissue distribution of receptors for C3a and C5a well exceeds myeloid
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8fa40cdd5eaf2293db13115262d9cdee
https://pubmed.ncbi.nlm.nih.gov/15081525
https://pubmed.ncbi.nlm.nih.gov/15081525
Autor:
Anna Erdei, György Vámosi, Krisztina Kerekes, János Matkó, Hajna Péterfy, Márton Andrásfalvy, Jakub Abramson, Israel Pecht, Gábor Tóth
Publikováno v:
Scopus-Elsevier
Peptides originally derived from complement component C3a were earlier shown to inhibit the type I FcεR (FcεRI)-mediated degranulation of mucosal type mast cells. In the present study, we show that C3a7, a peptide with a natural sequence, and its m
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::805e36826734697475cb6e356d0c7cc8
http://www.scopus.com/inward/record.url?eid=2-s2.0-23844511108&partnerID=MN8TOARS
http://www.scopus.com/inward/record.url?eid=2-s2.0-23844511108&partnerID=MN8TOARS
Autor:
László Balogh, László Muszbek, Gizella Haramura, Gabriella Szőke, Nóra Homoródi, Éva Katona, Róbert Gábor Kiss, István Édes, Amir-Houshang Shemirani, Zsuzsanna Bereczky, Sarolta Leé, Hajna Péterfy, E. Kovács
Publikováno v:
Journal of Translational Medicine
Background Aspirin resistance established by different laboratory methods is still a debated problem. Using COX1 specific methods no aspirin resistance was detected among healthy volunteers. Here we tested the effect of chronic aspirin treatment on p
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::caa51c39dc365946708e880955b39bc7