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of 6
pro vyhledávání: '"Hailey Harris"'
Autor:
Hailey Harris, Natasha Brison
Publikováno v:
Journal of Athlete Development and Experience, Vol 6, Iss 1 (2024)
Externí odkaz:
https://doaj.org/article/51de7c8c05b24cc4ae2ea9d916c3d072
Autor:
Hailey Harris, Sanjay Khandelwal, Lubica Rauova, John Lambris, Brandon Garcia, Douglas B. Cines, Gowthami M. Arepally
Publikováno v:
Blood. 140:8372-8373
Autor:
Sarah E. M. Herman, Hailey Harris, Priti Patel, Raquel Izumi, Maryalice Stetler-Stevenson, Constance M. Yuan, Michael Gulrajani, Todd Covey, Irina Maric, Christopher Pleyer, Jean Cheung, Adrian Wiestner, Stefania Pittaluga, Min Hui Wang, Clare Sun, Pia Nierman, Mohammed Farooqui, Ellen K. Kendall, Erika M Gaglione, Inhye E. Ahn, Ahmed Hamdy
Publikováno v:
Blood. 136:93-105
Inhibition of the B-cell receptor pathway, and specifically of Bruton tyrosine kinase (BTK), is a leading therapeutic strategy in B-cell malignancies, including chronic lymphocytic leukemia (CLL). Target occupancy is a measure of covalent binding to
Autor:
Jacquelyn L. Dillon, Hailey Harris, Gregory Zemtsov, Amanda M. Craig, Matthew R. Grace, Sarah K. Dotters-Katz
Publikováno v:
American Journal of Obstetrics and Gynecology. 226:S696
Autor:
Lauren T Vaughn, Hailey Harris, Liqiang Xi, Adrian Wiestner, Chingiz Underbayev, Mehdi Pirooznia, Mark Raffeld, Inhye E. Ahn, Sarah E. M. Herman
Publikováno v:
Blood. 134:1287-1287
Disease progression in chronic lymphocytic leukemia (CLL) patients receiving single-agent ibrutinib has been associated with either histological transformation or acquired mutations in BTK and PLCG2 genes which are found in up to 85% of patients with
Autor:
Arthur T. Sands, Hailey Harris, Sarah E. M. Herman, Neil Bence, Adrian Wiestner, Aileen Kelly, Sivasubramanian Baskar, May Tan, Timothy Ingallinera, Daniel W. Robbins, Deyi Zhang
Publikováno v:
Blood. 134:174-174
B-cell receptor (BCR) signaling plays an essential role in normal B-cell development and is a key driver of proliferation and survival of chronic lymphocytic leukemia (CLL) B cells. BCR signals through BTK to NF-ĸB. Targeted inhibition of BTK with i