The Hippo Kinase LATS2 Controls Helicobacter pylori-Induced Epithelial-Mesenchymal Transition and Intestinal Metaplasia in Gastric Mucosa

Autor: Silvia Molina-Castro, Alban Giese, Christine Varon, Philippe Lehours, Pierre Dubus, Julie Giraud, Camille Tiffon, Francis Mégraud, Emilie Bessède, Hélène Boeuf, Elodie Sifré, Geneviève Belleannée, Cathy Staedel

Publikováno v: Cellular and Molecular Gastroenterology and Hepatology
Cellular and Molecular Gastroenterology and Hepatology, 2020, 9 (2), pp.257-276. ⟨10.1016/j.jcmgh.2019.10.007⟩
Cellular and Molecular Gastroenterology and Hepatology 9(2)
Kérwá
Universidad de Costa Rica
instacron:UCR
Cellular and Molecular Gastroenterology and Hepatology, Philadelphia, PA : American Gastroenterological Association, [2015]-, 2020, 9 (2), pp.257-276. ⟨10.1016/j.jcmgh.2019.10.007⟩
Cellular and Molecular Gastroenterology and Hepatology, Vol 9, Iss 2, Pp 257-276 (2020)

Background & Aims Gastric carcinoma is related mostly to CagA+-Helicobacter pylori infection, which disrupts the gastric mucosa turnover and elicits an epithelial-mesenchymal transition (EMT) and preneoplastic transdifferentiation. The tumor suppress

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::be896e418fc10c8566166d6137999770
https://doi.org/10.1016/j.jcmgh.2019.10.007