Zobrazeno 1 - 10
of 266
pro vyhledávání: '"HITOO NISHINO"'
Autor:
Hitoo Nishino, Kazuma Sakakibara, Hideki Hida, Cha-Gyun Jung, Motoyuki Shiraki, Hisao Nishijo, Masateru Yamauchi, Susumu Urakawa
Publikováno v:
Journal of Neuroscience and Neuroengineering. 4:13-20
Publikováno v:
Journal of Neuroscience Research. 89:936-944
Metabolic activity in the suprachiasmatic nucleus (SCN), a center of biological rhythm, is higher during the daytime than at night. The rhythmic oscillation in the SCN is feedback controlled by the CLOCK/BMAL1 heterodimer binding to the E-box in targ
Publikováno v:
Neuroscience Letters. 436:321-325
Systemic administration of 3-nitropropionic acid (3-NPA) leads to a shortage of cellular ATP and induces striatum-specific lesions that resemble Huntington's disease. Gender differences, in terms of vulnerability of striatum to 3-NPA, have been shown
Publikováno v:
Neuroscience. 144:920-933
Rats raised in an enriched environment (enriched rats) have been reported to show less motor dysfunction following brain lesions, but the neuronal correlates of this improvement have not been well clarified. The present study aimed to elucidate the e
Publikováno v:
Brain Research. 1028:219-224
During the analysis of wheel running activity, we found that corticosterone (1 mg/100 g BW) injection decreased wheel activity, while dexamethasone (0.1 mg/100 g) increased the activity. To clarify the functional differences between corticosterone an
Autor:
Hitoo Nishino, Yoshiaki Isobe
Publikováno v:
Brain Research. 1013:204-211
Arg-vasopressin (AVP) containing neurons are one of the output paths from the suprachiasmatic nucleus (SCN), the center of the biological clock. AVP mRNA transcription is controlled by a negative feedback loop of clock genes. Circadian rhythm of mela
Publikováno v:
Journal of Pineal Research. 36:18-24
Melatonin, a pineal secretory product synthesized from tryptophan, has been found to be effective against neurotoxicity. The present study was aimed at demonstrating the effectiveness of melatonin in vivo in reducing ischemia-induced cerebral edema u