Zobrazeno 1 - 10
of 59
pro vyhledávání: '"HCAECs"'
Autor:
Xiaoling Li, Mengnan Wang, Jan-Ole Kalina, Benedikt Preckel, Markus W. Hollmann, Martin Albrecht, Coert J. Zuurbier, Nina C. Weber
Publikováno v:
Redox Biology, Vol 69, Iss , Pp 102979- (2024)
Background: Empagliflozin (EMPA) ameliorates reactive oxygen species (ROS) generation in human endothelial cells (ECs) exposed to 10 % stretch, but the underlying mechanisms are still unclear. Pathological stretch is supposed to stimulate protein kin
Externí odkaz:
https://doaj.org/article/2f33de2efafb4ef0b4548402cf79295b
Publikováno v:
Vascular Biology, Vol 4, Iss 1, Pp 11-18 (2022)
Atherosclerosis is characterised by abnormal lipid and cell accumulation within arterial layers that leads to disturbed blood flow. Modified cholesterol forms such as oxidised low-density lipoprotein (oxLDL) enter cells altering their phenotype, trig
Externí odkaz:
https://doaj.org/article/a213f6cd3c37453ca6b35addbd85a764
Akademický článek
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Publikováno v:
BMC Cardiovascular Disorders, Vol 19, Iss 1, Pp 1-9 (2019)
Abstract Background Kawasaki disease (KD) is an acute febrile childhood systemic vasculitis that disturbs coronary arteries. The pathogenesis remains unknown. The study of phosphorylated proteins helps to elucidate the relevant pathophysiological mec
Externí odkaz:
https://doaj.org/article/2f255403527940e8918b7eb1e27d5ec9
Akademický článek
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Publikováno v:
Iranian Journal of Basic Medical Sciences, Vol 18, Iss 6, Pp 610-615 (2015)
Objective(s): This study was designed to investigate the effect of receptor for advanced glycation end products (RAGE), S100A12 and C-reactive protein (CRP) on the release of circulating endothelial cells (CECs) from human coronary artery endothelial
Externí odkaz:
https://doaj.org/article/9b313713703a4de098d51b5b92baf309
Akademický článek
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Publikováno v:
Journal of Advanced Research, Vol 26, Iss, Pp 123-135 (2020)
Journal of Advanced Research
Journal of Advanced Research
Graphical abstract The map illustrating mechanisms associated with lncRNA HIF1A-AS2-mediated inflammation in the atherosclerosis. LncRNA HIF1A-AS2 forms a complex with USF1, which is recruited into the ATF2 promoter to elevate ATF2 expression, thus p
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::47972e7d0a5a67e00cdb998231298698
http://www.sciencedirect.com/science/article/pii/S2090123220301740
http://www.sciencedirect.com/science/article/pii/S2090123220301740
Publikováno v:
International Immunopharmacology. 38:26-30
Introduction The inflammatory receptor Toll-like receptors (TLRs) activation could induce endothelial inflammatory responses, which plays an important role in the development of many diseases including atherosclerosis. We already found that TLR2 acti
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::68f008b6b417d716a713b78bb1e50150
Publikováno v:
BMC Cardiovascular Disorders
BMC Cardiovascular Disorders, Vol 19, Iss 1, Pp 1-9 (2019)
BMC Cardiovascular Disorders, Vol 19, Iss 1, Pp 1-9 (2019)
Background Kawasaki disease (KD) is an acute febrile childhood systemic vasculitis that disturbs coronary arteries. The pathogenesis remains unknown. The study of phosphorylated proteins helps to elucidate the relevant pathophysiological mechanisms o
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c4c9b668fe28359889c4f9b6d7331609
https://pubmed.ncbi.nlm.nih.gov/30654760
https://pubmed.ncbi.nlm.nih.gov/30654760