Zobrazeno 1 - 10
of 202
pro vyhledávání: '"H. Valtin"'
Publikováno v:
American Journal of Physiology-Renal Physiology. 261:F345-F353
To test the hypothesis that rapid adenosine 3',5'-cyclic monophosphate (cAMP) catabolism via cyclic 3',5'-nucleotide phosphodiesterase (PDE) is a cause of the unresponsiveness to vasopressin (VP) in mice with hereditary nephrogenic diabetes insipidus
Autor:
H Valtin, H A Schroeder
Publikováno v:
Journal of the American Society of Nephrology : JASN. 8(8)
Autor:
H, Valtin
Publikováno v:
Nihon Jinzo Gakkai shi. 37(11)
Differential diagnosis of diabetes insipidus has been discussed in this paper in terms of pathophysiological aspects. Three points are emphasized: (1) we should include dipsogenic D.I. as one form of diabetes insipidus; (2) the water deprivation test
Publikováno v:
Physiologia Bohemoslovaca. 39(1)
In a strain of mice called DI +/+ Severe, nephrogenic (or vasopressin-resistant) diabetes insipidus is caused by an inability of the antidiuretic hormone (ADH, or vasopressin) to increase the water permeability of the renal collecting system. That in
Publikováno v:
Journal of Applied Physiology. 14:844-848
Co2 diuresis is probably mediated via a nonosmotic influence on the supraopticohypophyseal system. Since the left atrial stretch receptor mechanism is one such nonosmotic system which Co2 might influence, experiments were designed to elucidate its ro
Autor:
H Valtin
Publikováno v:
Journal of Clinical Investigation. 45:337-345
Autor:
H. Valtin, J. C. Mithoefer, F. Kreuzer, R. E. Nye, S. M. Tenney, A. Naitove, D. C. Andresen, B. F. Schreiner
Publikováno v:
Journal of Applied Physiology. 15:796-800
The effect of a sojourn of 5 days at an altitude of 14,300 feet (White Mountain, Cal.) on the alveolar-arterial oxygen gradient (A-aDo2) in the anesthetized dog was studied at three levels of oxygenation. Two dogs breathed 11% o2, air and 100% o2 at
Autor:
S. M. Tenney, H. Valtin
Publikováno v:
Journal of Applied Physiology. 15:1107-1112
In order to test the general postulate that increases in whole body metabolism are accompanied by net respiratory stimulation, respiratory adaptation to thyrotoxicosis was studied in clinical hyperthyroidism and in experimental hyperthyroidism in man