Zobrazeno 1 - 10
of 38
pro vyhledávání: '"H. Helen Lin"'
The HMGA2 architectural protein functions in a variety of cellular processes, such as cell growth, transcription regulation, neoplastic transformation, and progression. Up-regulation of HMGA2 protein is observed in many tumors and is associated with
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::269cf221d795724c8a08192f9ea7b8e0
https://doi.org/10.1158/1535-7163.c.6530915.v1
https://doi.org/10.1158/1535-7163.c.6530915.v1
Supplementary Figure 1 from SUMOylation of HMGA2: selective destabilization of promyelocytic leukemia protein via proteasome
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::49b6d9925deb088cf385b855724b9983
https://doi.org/10.1158/1535-7163.22482398.v1
https://doi.org/10.1158/1535-7163.22482398.v1
Autor:
David K. Ann, David J. Chen, Benjamin P.C. Chen, Yun Yen, Clay C.C. Wang, H. Helen Lin, Shih-Ya Wang, Lee Ming Boo, Angela Y.J. Li
Supplementary Figure 1 from Suppression of Nonhomologous End Joining Repair by Overexpression of HMGA2
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0019f2470fe7fa116a2bfbf1f6cc6a44
https://doi.org/10.1158/0008-5472.22378407.v1
https://doi.org/10.1158/0008-5472.22378407.v1
Autor:
David K. Ann, David J. Chen, Benjamin P.C. Chen, Yun Yen, Clay C.C. Wang, H. Helen Lin, Shih-Ya Wang, Lee Ming Boo, Angela Y.J. Li
Supplementary Figure 2 from Suppression of Nonhomologous End Joining Repair by Overexpression of HMGA2
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4aa23d295900fc51fba9aa7b4f00ff18
https://doi.org/10.1158/0008-5472.22378404
https://doi.org/10.1158/0008-5472.22378404
Autor:
David K. Ann, David J. Chen, Benjamin P.C. Chen, Yun Yen, Clay C.C. Wang, H. Helen Lin, Shih-Ya Wang, Lee Ming Boo, Angela Y.J. Li
Supplementary Figure 4 from Suppression of Nonhomologous End Joining Repair by Overexpression of HMGA2
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::145a54845625280917d8d893e789bb5b
https://doi.org/10.1158/0008-5472.22378398.v1
https://doi.org/10.1158/0008-5472.22378398.v1
Autor:
David K. Ann, David J. Chen, Benjamin P.C. Chen, Yun Yen, Clay C.C. Wang, H. Helen Lin, Shih-Ya Wang, Lee Ming Boo, Angela Y.J. Li
Supplementary Figure 5 from Suppression of Nonhomologous End Joining Repair by Overexpression of HMGA2
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4e88143c15e648fad64a7756118c7f6c
https://doi.org/10.1158/0008-5472.22378395.v1
https://doi.org/10.1158/0008-5472.22378395.v1
Autor:
David K. Ann, David J. Chen, Benjamin P.C. Chen, Yun Yen, Clay C.C. Wang, H. Helen Lin, Shih-Ya Wang, Lee Ming Boo, Angela Y.J. Li
Supplementary Figure 3 from Suppression of Nonhomologous End Joining Repair by Overexpression of HMGA2
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::564ceb97d98b5181417f4aaa59efbc18
https://doi.org/10.1158/0008-5472.22378401.v1
https://doi.org/10.1158/0008-5472.22378401.v1
Autor:
Shuting Cao, Yu-Wen Hung, Yi-Chang Wang, Yiyin Chung, Yue Qi, Ching Ouyang, Xiancai Zhong, Weidong Hu, Alaysia Coblentz, Ellie Maghami, Zuoming Sun, H. Helen Lin, David K. Ann
RationaleImmunosuppression in the tumor microenvironment (TME) is key to the pathogenesis of solid tumors. Tumor cell-intrinsic autophagy is critical for sustaining both tumor cell metabolism and survival. However, the role of autophagy in the host i
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9dc7765b755c537b2a978b4fe9780e3d
https://doi.org/10.1101/2022.04.29.490103
https://doi.org/10.1101/2022.04.29.490103
Autor:
Jenny C.Y. Chu, Hung Jung Wang, Christina M. Vidal, Myung Eun Oh, Dustin E. Schones, Yiyin Chung, Yu Han Chen, David K. Ann, Peiguo Chu, Hsing Jien Kung, Yue Qi, Ching Ouyang, Chun Ting Cheng, Yulong Tang, Yun Ru Liu, Lei Jiang, H. Helen Lin, Yi Chang Wang, Yun-Ru Chen, Kyle M. Miller, Ching Ying Kuo, Xiangpeng Sheng, Yun Yen, Kevin K. Chi
Publikováno v:
Communications Biology, Vol 1, Iss 1, Pp 1-15 (2018)
Communications Biology
Communications Biology
Defective arginine synthesis, due to the silencing of argininosuccinate synthase 1 (ASS1), is a common metabolic vulnerability in cancer, known as arginine auxotrophy. Understanding how arginine depletion kills arginine-auxotrophic cancer cells will
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4d84519922e774356ed489cf4b6da881
http://link.springer.com/article/10.1038/s42003-018-0178-4
http://link.springer.com/article/10.1038/s42003-018-0178-4
Autor:
Chun Ting Cheng, Kirsten H. Limesand, Ching Ying Kuo, Ching Ouyang, Hsing Jien Kung, David K. Ann, Yong Fu, H. Helen Lin, Kevin K. Chi, Maryam Sadeghi, Chien-Feng Li, Yiyin Chung, Peiguo Chu
Publikováno v:
Autophagy. 14:1481-1498
Defects in basal autophagy limit the nutrient supply from recycling of intracellular constituents. Despite our understanding of the pro-survival role of macroautophagy/autophagy, how nutrient deprivation, caused by compromised autophagy, affects onco
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::071682633ebcb3d940cbf940c3a43243
https://doi.org/10.1080/15548627.2018.1450708
https://doi.org/10.1080/15548627.2018.1450708