Výsledky vyhledávání - "H T, Wessels"

Leydig cell hypoplasia: cases with new mutations, new polymorphisms and cases without mutations in the luteinizing hormone receptor gene

Autor: H. G. Brunner, Stenvert L. S. Drop, G. H. G. Sinnecker, Sergio P. A. Toledo, A. Richter-Unruh, W. A. Kors, H. T. Wessels, Axel P. N. Themmen, Miriam Verhoef-Post, John W.M. Martens, Annemie L.M. Boehmer

Publikováno v: Clinical Endocrinology. 56:103-112

Summary background Defective male sex differentiation in patients with hypoplasia of Leydig cells (LCH) is caused by deficient LH receptor signal transduction. To further investigate the variety of LH receptor gene mutations present in LCH patients a

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::ace197765508d46c3cf286e648bdb93e
https://doi.org/10.1046/j.0300-0664.2001.01437.x

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In vitro secretion of FSH by cultured clinically nonfunctioning and gonadotroph pituitary adenomas is directly correlated with locally produced levels of activin A

Autor: L. Van Gastel, P. M. van Koetsveld, W. W. de Herder, Leo J. Hofland, R. Van Der Wal, F. H. De Jong, H. T. Wessels

Publikováno v: Clinical Endocrinology. 54:485-492

OBJECTIVE: Expression of mRNAs encoding activin and its antagonists inhibin and follistatin has been described in human pituitary adenomas, including clinically nonfunctioning adenomas (NFAs) and gonadotroph adenomas (Gn-omas). Since many of the NFAs

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::0bfb11ded197da505869927e444e2254
https://doi.org/10.1046/j.1365-2265.2001.01194.x

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Male LH-independent sexual precocity in a 3.5-year-old boy caused by a somatic activating mutation of the LH receptor in a Leydig cell tumor

Autor: Berthold P. Hauffa, U. Menken, S. Tappeser, Annette Richter-Unruh, H. T. Wessels, J. Schaper, Martin Bergmann, K. Schmittmann-Ohters

Publikováno v: The Journal of clinical endocrinology and metabolism. 87(3)

We describe the clinical features of severe sexual precocity in a 3.5-yr-old boy. Hormonal evaluation showed LH-independent T hypersecretion. Initial examination of the adrenals and testes revealed no evidence of congenital adrenal hyperplasia, hCG-

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a468e27d1f76d5d080a173f8ef20efad
https://pubmed.ncbi.nlm.nih.gov/11889161

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Leydig cell hypoplasia: cases with new mutations, new polymorphisms and cases without mutations in the luteinizing hormone receptor gene

Autor: A, Richter-Unruh, J W M, Martens, M, Verhoef-Post, H T, Wessels, W A, Kors, G H G, Sinnecker, A, Boehmer, S L S, Drop, S P A, Toledo, H G, Brunner, A P N, Themmen

Publikováno v: Clinical endocrinology. 56(1)

Defective male sex differentiation in patients with hypoplasia of Leydig cells (LCH) is caused by deficient LH receptor signal transduction. To further investigate the variety of LH receptor gene mutations present in LCH patients and their influence

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::4401c882922f6c8102c9e7d4ebb93ca1
https://pubmed.ncbi.nlm.nih.gov/11849253

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In vitro secretion of FSH by cultured clinically nonfunctioning and gonadotroph pituitary adenomas is directly correlated with locally produced levels of activin A

Autor: H T, Wessels, L J, Hofland, R, van der Wal, L, van Gastel, P M, van Koetsveld, W W, de Herder, F H, de Jong

Publikováno v: Clinical endocrinology. 54(4)

Expression of mRNAs encoding activin and its antagonists inhibin and follistatin has been described in human pituitary adenomas, including clinically nonfunctioning adenomas (NFAs) and gonadotroph adenomas (Gn-omas). Since many of the NFAs and Gn-oma

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::690dcd03ef4bb8e6e36d82a38e13edff
https://pubmed.ncbi.nlm.nih.gov/11318784

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