Zobrazeno 1 - 10
of 17
pro vyhledávání: '"H J, James"'
Autor:
Ning Tong, Sandra A. Kinnear, H. J. James, Thomas J. Schall, Andrew I. Brooks, Steven M. Fine, Harris A. Gelbard, Leon G. Epstein, Stephen Dewhurst, Daniel J. Dairaghi, Howard E. Gendelman, Seth W. Perry, Leroy R. Sharer, Huang Guo, Harshawardhan P. Bal, Qing Zhang
Publikováno v:
The Journal of Immunology. 164:1333-1339
HIV-1 infection of the brain results in chronic inflammation, contributing to the neuropathogenesis of HIV-1 associated neurologic disease. HIV-1-infected mononuclear phagocytes (MP) present in inflammatory infiltrates produce neurotoxins that mediat
Autor:
John C. Reed, H. J. James, Harris A. Gelbard, Q. Zhang, Hong Gang Wang, Leroy R. Sharer, Leon G. Epstein
Publikováno v:
ResearcherID
Apoptosis of neurones, macrophages, and microglia occurs in the brains of paediatric patients with human immunodeficiency virus (HIV) type 1 encephalitis, which is often associated with pre-mortem neurological disease (progressive encephalopathy). We
Autor:
J. C. Reed, Leroy R. Sharer, Benjamin M. Blumberg, Suryaram Gummuluru, Stephen Dewhurst, Harris A. Gelbard, S. Krajewski, J. Ross, H. J. James, Howard E. Gendelman, Leon G. Epstein
Publikováno v:
Neuropathology and Applied Neurobiology. 23:242-253
We have previously demonstrated the presence of DNA fragmentation in neurons, macrophages and microglia consistent with apoptosis, but not in reactive astrocytes in brain tissue from paediatric patients with HIV-1 encephalitis (HIVE). To further unde
Autor:
S. Krajewski, H. J. James, J. Ross, B. M. Blumberg, L. G. Epstein, H. E. Gendelman, S. Gummuluru, S. Dewhurst, L. R. Sharer, J. C. Reed, H. A. Gelbard
Publikováno v:
Neuropathology and Applied Neurobiology. 23:242-253
Autor:
Harris A. Gelbard, H. J. James, Sheila C. Dollard, Stephen Dewhurst, Leon G. Epstein, Leroy R. Sharer
Publikováno v:
Neuropathology and Applied Neurobiology. 21:518-528
Human immunodeficiency virus type 1 (HIV-1) infection of the central nervous system is associated with decreased neuronal density in discrete areas of the brain. Neuronal loss may occur via apoptosis, initiated by soluble neurotoxic factors secreted
Autor:
H. J. James, S. M. Blumberg, A. M. Kazee, Seth W. Perry, Yoshihiro Saito, Leon G. Epstein, Leroy R. Sharer, Harris A. Gelbard
Publikováno v:
Neuropathology and Applied Neurobiology. 21:208-217
The pathogenesis of human immunodeficiency virus type 1 (HIV-1) associated dementia in adults involves neuronal loss from discrete areas of the neocortex and subcortical regions, but the mechanism for neuronal death is poorly understood. Gene-directe
Autor:
S, Krajewski, H J, James, J, Ross, B M, Blumberg, L G, Epstein, H E, Gendelman, S, Gummuluru, S, Dewhurst, L R, Sharer, J C, Reed, H A, Gelbard
Publikováno v:
Neuropathology and applied neurobiology. 23(3)
We have previously demonstrated the presence of DNA fragmentation in neurons, macrophages and microglia consistent with apoptosis, but not in reactive astrocytes in brain tissue from paediatric patients with HIV-1 encephalitis (HIVE). To further unde
Publikováno v:
Neuropathology and applied neurobiology. 21(6)
Human immunodeficiency virus type 1 (HIV-1) infection of the central nervous system is associated with decreased neuronal density in discrete areas of the brain. Neuronal loss may occur via apoptosis, initiated by soluble neurotoxic factors secreted
Autor:
H A, Gelbard, H J, James, L R, Sharer, S W, Perry, Y, Saito, A M, Kazee, B M, Blumberg, L G, Epstein
Publikováno v:
Neuropathology and applied neurobiology. 21(3)
The pathogenesis of human immunodeficiency virus type 1 (HIV-1) associated dementia in adults involves neuronal loss from discrete areas of the neocortex and subcortical regions, but the mechanism for neuronal death is poorly understood. Gene-directe
Publikováno v:
Journal of the American College of Surgeons. 180(1)
Negative nitrogen balance after burn injury mainly indicates muscle catabolism, but the exact influence of burn on protein synthesis and breakdown in different types of skeletal muscle and the role of glucocorticoids in this metabolic response are un