Zobrazeno 1 - 10
of 441
pro vyhledávání: '"H B, Hechtman"'
Autor:
J. M. Klausner, S. Abu-Abid, J. S. Alexander, R. Hanshke-Mineau, G. Goldman, N. Morel, C. R. Valeri, D. Shepro, H. B. Hechtman
Publikováno v:
Mediators of Inflammation, Vol 3, Iss 2, Pp 149-153 (1994)
The study tests the role of thromboxane in modulating microvascular permeability in vitro. Cultured monolayers of bovine aortic endothelial cells were challenged with the thromboxane (Tx) mimic U46619. This led to disassembly of actin microfilaments,
Externí odkaz:
https://doaj.org/article/55e98f69a4634e508b7d2591ae691b2d
Publikováno v:
The Journal of Immunology. 149:1723-1728
We examined the role of C activation in ischemia reperfusion injury by inhibiting C activation in a rat model of mesenteric arterial occlusion. In anesthetized rats, 60 min of mesenteric arterial occlusion was followed by 3 h of reperfusion. PBS alon
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::9338926b045945baa862d59a1214e200
https://doi.org/10.4049/jimmunol.149.5.1723
https://doi.org/10.4049/jimmunol.149.5.1723
Publikováno v:
The Journal of Immunology. 145:1906-1911
Neutrophils and eicosanoid chemoattractants are centrally involved with ischemia-reperfusion (I/R) injury. The CD 18 complex of adhesive glycoproteins, readily up-regulated by chemoattractants in vitro, is required for polymorphonuclear leukocyte (PM
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::9ff0d67a46248d77afc51806f2fb7663
https://doi.org/10.4049/jimmunol.145.6.1906
https://doi.org/10.4049/jimmunol.145.6.1906
Publikováno v:
The Annals of thoracic surgery. 72(5)
Shed nonwashed blood and shed washed red blood cells (RBC) are being used as alternatives to allogeneic liquid-preserved RBC for patients during thoracic and cardiovascular surgical procedures.Mongrel dogs were bled a volume of blood into the abdomin
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::24eafc5401ca6674fd3e79b7eb63b478
https://pubmed.ncbi.nlm.nih.gov/11722051
https://pubmed.ncbi.nlm.nih.gov/11722051
Publikováno v:
Journal of cellular biochemistry. 75(1)
Vascular pathologies induced by ischemia/reperfusion involve the production of reactive oxygen species (ROS) that in part cause tissue injury. The production of ROS that occurs upon reperfusion activates specific second messenger pathways. In diabeti
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::532af9acffded7243e83cb28cc9ff29c
https://pubmed.ncbi.nlm.nih.gov/10462710
https://pubmed.ncbi.nlm.nih.gov/10462710
Publikováno v:
Surgery. 126(2)
The dependence of intestinal ischemia-reperfusion injury on the classical complement pathway has been shown with the complement antagonist (sCR1) and complement-specific knockout mice. Using C5 deficient mice, we show that the membrane attack complex
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::4a7e6ae83cc3fdd1143ba7f454133f76
https://pubmed.ncbi.nlm.nih.gov/10455904
https://pubmed.ncbi.nlm.nih.gov/10455904
Publikováno v:
Journal of cellular biochemistry. 68(4)
Hypoxia/reoxygenation injury to cultured endothelial cells results in cytoskeletal rearrangement and second messenger activation related to increased monolayer junctional permeability. Cytoskeletal rearrangement by reactive oxygen species may be rela
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::b534635cd7a0462dd0cda54ed1fe80f6
https://pubmed.ncbi.nlm.nih.gov/9493913
https://pubmed.ncbi.nlm.nih.gov/9493913
Autor:
Booker, Gary C.1
Publikováno v:
Journal of Aviation / Aerospace Education & Research. 2024, Vol. 33 Issue 2, p101-134. 34p.
Publikováno v:
Journal of cellular physiology. 172(3)
Hypoxia/reoxygenation injury in vitro causes endothelial cell cytoskeletal rearrangement that is related to increased monolayer permeability. Nonmuscle filamin (ABP-280) promotes orthogonal branching of F-actin and links microfilaments to membrane gl
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::95dbad96ea105abc87bfcc5aae26e061
https://pubmed.ncbi.nlm.nih.gov/9284957
https://pubmed.ncbi.nlm.nih.gov/9284957