Výsledky vyhledávání - "Hörsten, S von"

Proteolytic degradation of neuropeptide Y (NPY) from head to toe

Autor: Wagner, L., Wolf, R., Zeitschel, U., Rossner, S., Petersén, A., Leavitt, B.R., Kästner, F., Rothermundt, M., Gärtner, U.-T., Gündel, D., Schlenzig, D., Frerker, N., Schade, J., Manhart, S., Rahfeld, J.-U., Demuth, H.-U., Hörsten, S. von

The bioactivity of neuropeptide Y (NPY) is either N-terminally modulated with respect to receptor selectivity by dipeptidyl peptidase 4 (DP4)-like enzymes or proteolytic degraded by neprilysin or meprins, thereby abrogating signal transduction. Howev

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=od_______610::f9242d9a8112e0d4edea7ee303b615f7
https://publica.fraunhofer.de/handle/publica/243370

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Periodical

In vivo assessment of neuronal dysfunction in rats transgenic for Huntington's disease using small animal FDG-PET and MRI – a 16 months follow-up study

Autor: Lippross, V, Hermann, S, Nagelmann, N, Heselhaus, J, Bohlen, S, Kugel, H, Deppe, M, Sommer, J, Bremer, C, Nguyen, H, Riess, O, Hörsten, S von, Schäfers, M, Reilmann, R

Publikováno v: Aktuelle Neurologie; 20240101, Issue: Preprints

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Airway-specific recruitment of T cells is reduced in a CD26-deficient F344 rat substrain

Autor: Schade, J., Schmiedl, A., Kehlen, A., Veres, T.Z., Stephan, M., Pabst, R., Hörsten, S. von

Asthma is a chronic inflammatory disease affecting the airways. Increased levels of T cells are found in the lungs after the induction of an allergic-like inflammation in rats, and flow cytometry studies have shown that these levels are reduced in CD

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=od_______610::98982c50fd13053bd989d084e4a8af6e
https://publica.fraunhofer.de/handle/publica/219311

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CD26 (dipeptidyl-peptidase IV)-dependent recruitment of T cells in a rat asthma model

Autor: Kruschinski, C., Skripuletz, T., Bedoui, S., Tschernig, T., Pabst, R., Nassenstein, C., Hörsten, S. von

CD26 truncates several chemokines as well as neuropeptides and influences immune responses via modulation of cell adhesion and T cell activation, suggesting an involvement of CD26 in asthmatic and airway inflammation. Therefore, Fischer 344 (F344), B

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=od_______610::04c29995a94140b96f6d8ce7ee4fc621
https://publica.fraunhofer.de/handle/publica/208510

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