Zobrazeno 1 - 10
of 37
pro vyhledávání: '"György, Veress"'
Publikováno v:
Virology Journal, Vol 20, Iss 1, Pp 1-10 (2023)
Abstract Background High-risk human papillomaviruses (HPVs) are responsible for the development of cervical and other anogenital cancers. Intratype sequence variants of certain high-risk HPV types (e.g. 16, 18 and 31) are thought to have different on
Externí odkaz:
https://doaj.org/article/5231750f82774ab9b2d16c7cd45a6562
Autor:
Brigitta László, László Antal, Eszter Gyöngyösi, Anita Szalmás, Szilárd Póliska, György Veress, József Kónya
Publikováno v:
BMC Cancer, Vol 21, Iss 1, Pp 1-19 (2021)
Abstract Background miRNAs and lncRNAs can regulate cellular biological processes both under physiological and pathological conditions including tumour initiation and progression. Interactions between differentially expressed diverse RNA species, as
Externí odkaz:
https://doaj.org/article/4318c6af93da4eabb9512649ca6d3374
Publikováno v:
Virus Genes
The functional analysis of human papillomavirus (HPV) sequence variation requires the molecular cloning of different genomic regions of virus variants. In this study, we report an unexpected difficulty experienced when trying to clone HPV33 long cont
Autor:
Levente Szakács, Brigitta László, László Madar, József Kónya, Eszter Gyöngyösi, Anita Szalmás, Annamária Ferenczi, György Veress
Publikováno v:
Virus Genes. 52:552-555
The mechanisms that regulate papillomavirus gene expression include DNA methylation. The transcription of papillomavirus oncogenes E6 and E7 is controlled by certain regulatory elements in the LCR, which include binding sites for the E2 protein, a vi
Autor:
József Kónya, Annamária Ferenczi, Anita Szalmás, Eszter Gyöngyösi, György Veress, Brigitta László
Publikováno v:
Infection, genetics and evolution : journal of molecular epidemiology and evolutionary genetics in infectious diseases. 43
High-risk human papillomaviruses (HPV) are the causative agents of cervical and other anogenital cancers as well as a subset of head and neck cancers. The E6 and E7 oncoproteins of HPV contribute to oncogenesis by associating with the tumour suppress
Autor:
Balázs Ádám, Katinka Hernádi, Ildikó-Márton, György Veress, Anita Szalmás, Eszter Gyöngyösi, Eszter Csoma, József Kónya
Publikováno v:
Oral Surgery, Oral Medicine, Oral Pathology, Oral Radiology, and Endodontology. 112:401-406
The occurrence of human herpesvirus (HHV) 6 subtypes A and B in apical periodontitis was determined. The relationship of HHV-6 subtypes to other disease associated herpesviruses, i.e., Epstein-Barr virus (EBV) and human cytomegalovirus, was also inve
Autor:
Györgyike Soós, Tamás Sápy, Zoltán Hernádi, György Veress, Zoárd Tibor Krasznai, Lajos Gergely, József Kónya, Krisztina Szoke
Publikováno v:
European Journal of Obstetrics & Gynecology and Reproductive Biology. 118:229-234
Objective: To evaluate the role of human papillomavirus (HPV) testing in post-treatment follow-up of patients after therapeutic excision of the cervix due to positive screening tests. Study design: A hospital-based retrospective analysis was performe
Publikováno v:
Archives of Virology. 149:2379-2392
The effect of the human papillomavirus type 16 (HPV 16) E6 and E7 proteins was studied on the transcriptional activity of the human transforming growth factor beta2 (TGF-beta) promoter in different cell lines. Luciferase tests were performed after co
Autor:
Lajos Gergely, Zoárd Tibor Krasznai, József Kónya, Joakim Dillner, György Veress, Krisztina Szoke, Tamás Sápy, Zoltán Hernádi, Györgyi Szládek
Publikováno v:
Journal of Medical Virology. 71:585-592
The oncogenic potential of human papillomavirus (HPV) infection was assessed by following the disease course in 455 patients who had had a routine diagnostic Hybrid Capture HPV test due to squamous cell abnormalities of the uterine cervix as detected
Autor:
Krisztina Szarka, Róbert Póka, Tamás Sápy, Zoltán Hernádi, György Veress, Zoárd Tibor Krasznai
Publikováno v:
BJOG: An International Journal of Obstetrics and Gynaecology. 110:205-209
Objective Prognostic evaluation of HPV-16 genome status of the pelvic lymph nodes, the integration status of HPV-16 and p53 codon 72 polymorphism in cervical cancer. Design Prospective cohort study. Setting Department of Gynaecological Oncology, Univ