Zobrazeno 1 - 10
of 75
pro vyhledávání: '"Gulzar Wani"'
Autor:
Matteo Puglisi, Chu Lan Lao, Gulzar Wani, Giacomo Masserdotti, Riccardo Bocchi, Magdalena Götz
Publikováno v:
Cells, Vol 13, Iss 17, p 1408 (2024)
Direct neuronal reprogramming is a promising approach to replace neurons lost due to disease via the conversion of endogenous glia reacting to brain injury into neurons. However, it is essential to demonstrate that the newly generated neurons origina
Externí odkaz:
https://doaj.org/article/c35688423d2d42bfb689491aa03e051d
Autor:
Hans‐Georg Sprenger, Gulzar Wani, Annika Hesseling, Tim König, Maria Patron, Thomas MacVicar, Sofia Ahola, Timothy Wai, Esther Barth, Elena I Rugarli, Matteo Bergami, Thomas Langer
Publikováno v:
EMBO Molecular Medicine, Vol 11, Iss 1, Pp n/a-n/a (2019)
Abstract Disturbances in the morphology and function of mitochondria cause neurological diseases, which can affect the central and peripheral nervous system. The i‐AAA protease YME1L ensures mitochondrial proteostasis and regulates mitochondrial dy
Externí odkaz:
https://doaj.org/article/b76862f3c6d1473b9308a814409d2aff
Publikováno v:
PLoS ONE, Vol 11, Iss 7, p e0159344 (2016)
Cell cycle checkpoint is mediated by ATR and ATM kinases, as a prompt early response to a variety of DNA insults, and culminates in a highly orchestrated signal transduction cascade. Previously, we defined the regulatory role of nucleotide excision r
Externí odkaz:
https://doaj.org/article/e4cbd728d2e64377b61988bfc9b759c0
Publikováno v:
PLoS ONE, Vol 5, Iss 6, p e11007 (2010)
Transcription factor II H (TFIIH) is comprised of core TFIIH and Cdk-activating kinase (CAK) complexes. Here, we investigated the molecular and cellular manifestation of the TFIIH compositional changes by XPG truncation mutations. We showed that both
Externí odkaz:
https://doaj.org/article/6766be7b59cd46858beff27a1578b104
Autor:
Altaf A. Wani, Mohamed A. El-Mahdy, Qun Zhao, Gulzar Wani, Bassant M. Barakat, Qianzheng Zhu, El-Shaimaa A. Arafa
Supplementary Methods, Figures 1-3, Table 1 from Tangeretin Sensitizes Cisplatin-Resistant Human Ovarian Cancer Cells through Downregulation of Phosphoinositide 3-Kinase/Akt Signaling Pathway
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::94973edbeb671d326b86065249550ae2
https://doi.org/10.1158/0008-5472.22382879.v1
https://doi.org/10.1158/0008-5472.22382879.v1
Autor:
Altaf A. Wani, Mohamed A. El-Mahdy, Qun Zhao, Gulzar Wani, Bassant M. Barakat, Qianzheng Zhu, El-Shaimaa A. Arafa
Combination of innocuous dietary components with anticancer drugs is an emerging new strategy for cancer chemotherapy to increase antitumor responses. Tangeretin is a citrus flavonoid known to inhibit cancer cell proliferation. Here, we show an enhan
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::193756d1f36061f31ee85b05ebed3893
https://doi.org/10.1158/0008-5472.c.6500351
https://doi.org/10.1158/0008-5472.c.6500351
In this paper, statistical analysis of Acoustic Emission (AE) based b-value parameter has been carried out. In the initial phase, multi-linear regression was carried out on AE parameters released during uniaxial compression of twelve cylindrical samp
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=od______2659::fe4fe4efd27b96352e3d8fa3879599b7
https://zenodo.org/record/7699526
https://zenodo.org/record/7699526
Publikováno v:
Cell Cycle
article-version (VoR) Version of Record
article-version (VoR) Version of Record
Mineralocorticoid and androgen receptor antagonist, spironolactone, was recently identified as an inhibitor of nucleotide excision repair (NER), acting via induction of proteolysis of TFIIH component Xeroderma Pigmentosum B protein (XPB). This activi
Publikováno v:
Nucleic Acids Research. 49:12603-12604
Publikováno v:
Oncotarget. 8:104525-104542
Acetylated histone H3 lysine 56 (H3K56Ac) diminishes in response to DNA damage but is restored following DNA repair. Here, we report that CRL4DDB2 ubiquitin ligase preferentially regulates post-repair chromatin restoration of H3K56Ac through recruitm