Zobrazeno 1 - 10 of 10 pro vyhledávání: '"Guillermo Garcia-Eguren"'
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Akademický článek
Circulatory Immune Cells in Cushing Syndrome: Bystanders or Active Contributors to Atherometabolic Injury? A Study of Adhesion and Activation of Cell Surface Markers
Autor: Gloria Aranda, Cristina Lopez, Rebeca Fernandez-Ruiz, Yaiza Esteban, Guillermo Garcia-Eguren, Mireia Mora, Irene Halperin, Gregori Casals, Joaquim Enseñat, Felicia A. Hanzu
Publikováno v: International Journal of Endocrinology, Vol 2017 (2017)
Glucocorticoids (GC) induce cardiometabolic risk while atherosclerosis is a chronic inflammation involving immunity. GC are immune suppressors, and the adrenocorticotrophic hormone (ACTH) has immune modulator activities. Both may act in atherothrombo
Externí odkaz: https://doaj.org/article/d4213fd57ac5495fa0fcc5ec7017e3be
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2
Endogenous cortisol excess confers a unique lipid signature and metabolic network
Autor: Vanesa Flores, Mireia Mora, Gemma Rojo, Felicia A. Hanzu, Ting Hu, Francesc Carmona, Oriol Giró, Guillermo Garcia-Eguren, Gloria Aranda, Marta Iruarrizaga, Ramon Gomis, Arturo Vega-Beyhart, Gregori Casals, Adriana Pané, Laura Boswell, Cristina Alonso, Joaquim Enseñat, Irene Halperin, Oscar Vidal
Publikováno v: Journal of Molecular Medicine. 99:1085-1099
Chronic cortisol excess induces several alterations on protein, lipid and carbohydrate metabolism resembling those found in the metabolic syndrome. However, patients exposed to prolonged high levels of cortisol in Cushing syndrome (CS) present exceed
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0ca6fc536f5a278fe47c3983adb506e3
https://doi.org/10.1007/s00109-021-02076-0
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3
Long-term hypercortisolism induces lipogenesis promoting palmitic acid accumulation and inflammation in visceral adipose tissue compared with HFD-induced obesity
Autor: Arturo Vega-Beyhart, Oriol Giró, Guillermo Garcia-Eguren, Felicia A. Hanzu, Aleix Sala-Vila
Publikováno v: American Journal of Physiology-Endocrinology and Metabolism. 318:E995-E1003
Glucocorticoids (GCs) play critical roles in adipose tissue metabolism. Here, we compare in a mouse model the effects of chronic glucocorticoid excess and diet-induced obesity on white adipose tissue mass and distribution, by focusing on visceral adi
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::bf14b19802ca9b2021d31d3f4ad9aa5c
https://doi.org/10.1152/ajpendo.00516.2019
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4
Chronic hypercortisolism causes more persistent visceral adiposity than HFD-induced obesity
Autor: Oriol Giró, Felicia A. Hanzu, María del Mar Romero, Grasa Mm, Guillermo Garcia-Eguren
Publikováno v: Journal of Endocrinology. 242:65-77
Excessive and prolonged glucocorticoid (GC) exposure, resulting from either prescribed or endogenous hypercortisolism, is associated with a high cardiovascular and metabolic burden (Cushing’s syndrome). Although previous studies in humans and mice
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8077bfc30f18ebf707ffa7bac7d69182
https://doi.org/10.1530/joe-19-0168
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5
Glucocorticoid-induced Fingerprints on Visceral Adipose Tissue Transcriptome and Epigenome
Autor: Pedro Vizán, Mar González-Ramírez, Mireia Mora, Laura Boswell, Guillermo Garcia-Eguren, Oscar Vidal, Oriol Giró, Irene Halperin, Mattia Squarcia, Meritxell Gracia, Arturo Vega-Beyhart, Gregori Casals, Joaquim Enseñat, Luciano Di Croce, Francisco Carmona, Felicia A. Hanzu
Publikováno v: The Journal of clinical endocrinology and metabolism. 107(1)
Context Chronic glucocorticoid (GC) overexposure, resulting from endogenous Cushing’s syndrome (CS) or exogenous GC therapy, causes several adverse outcomes, including persistent central fat accumulation associated with a low-grade inflammation. Ho
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1526c702884325b8e1614c0ff4474b22
https://pubmed.ncbi.nlm.nih.gov/34487152
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7
OR03-05 Novel Lipidome Signature in Active Cushing Syndrome Revealed by UHPLC-MS Metabolomics
Autor: Irene Halperin, Gloria Aranda, Joaquim Enseñat, Ting Hu, Guillermo Garcia-Eguren, Adriana Pané, Gemma Rojo, Oscar Vidal, Marta Iruarrizaga, Mireia Mora Porta, Felicia A. Hanzu, Oriol Giró, Arturo Vega-Beyhart, Gregori Casals, Francesc Carmona
Publikováno v: Journal of the Endocrine Society
Background: Alterations in circulating amino acids, polyamines and acylcarnitines have been reported in patients with endogenous chronic hypercortisolism. However, lipid metabolites profiling and its interplay with the serum metabolome and degree of
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9443f11df500d468c87331b916b2674d
https://doi.org/10.1210/jendso/bvaa046.1295
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10
Circulatory immune cells in Cushing syndrome: bystanders or active contributors to atherometabolic injury? A study of adhesion and activation of cell surface markers
Autor: Mireia Mora, Joaquim Enseñat, Guillermo Garcia-Eguren, Rebeca Fernandez-Ruiz, Gregori Casals, Yaiza Esteban, Felicia A. Hanzu, Gloria Aranda, Irene Halperin, Cristina Lopez
Publikováno v: Recercat. Dipósit de la Recerca de Catalunya
instname
International Journal of Endocrinology, Vol 2017 (2017)
International Journal of Endocrinology
Dipòsit Digital de la UB
Universidad de Barcelona
Glucocorticoids (GC) induce cardiometabolic risk while atherosclerosis is a chronic inflammation involving immunity. GC are immune suppressors, and the adrenocorticotrophic hormone (ACTH) has immune modulator activities. Both may act in atherothrombo
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::fb2c11e7b120d47a106745236fd865b2
http://hdl.handle.net/2445/127138
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