Zobrazeno 1 - 10
of 52
pro vyhledávání: '"Guillaume Calmettes"'
Publikováno v:
PLoS ONE, Vol 18, Iss 11 (2023)
Externí odkaz:
https://doaj.org/article/2547c224892842e893066ac1aa8bffdd
Autor:
Guillaume Calmettes, James N. Weiss
Publikováno v:
Heliyon, Vol 3, Iss 11, Pp e00451- (2017)
How did egalitarianism emerge in early human societies? In contrast to dominance hierarchies in non-human primates, human simple forager bands are typically egalitarian, with male hunters often serving as the collective alpha. Here we present a therm
Externí odkaz:
https://doaj.org/article/60a4b8d691554ef5a85b9b8dbaceabad
Autor:
Guillaume Calmettes, James N Weiss
Publikováno v:
PLoS ONE, Vol 8, Iss 12, p e81988 (2013)
The genetic expression of cloned fluorescent proteins coupled to time-lapse fluorescence microscopy has opened the door to the direct visualization of a wide range of molecular interactions in living cells. In particular, the dynamic translocation of
Externí odkaz:
https://doaj.org/article/1670e56617584552815a5420c4b1083f
Autor:
Gilles Gouspillou, Richard Rouland, Guillaume Calmettes, Véronique Deschodt-Arsac, Jean-Michel Franconi, Isabelle Bourdel-Marchasson, Philippe Diolez
Publikováno v:
PLoS ONE, Vol 6, Iss 6, p e20709 (2011)
BackgroundMitochondrial dysfunctions appear strongly implicated in a wide range of pathologies. Therefore, there is a growing need in the determination of the normal and pathological integrated response of oxidative phosphorylation to cellular ATP de
Externí odkaz:
https://doaj.org/article/3f4a36c8711f4b90b3b248039ef4a77a
Autor:
Guillaume Calmettes, Véronique Deschodt-Arsac, Gilles Gouspillou, Sylvain Miraux, Bernard Muller, Jean-Michel Franconi, Eric Thiaudiere, Philippe Diolez
Publikováno v:
PLoS ONE, Vol 5, Iss 2, p e9306 (2010)
Hypoxic states of the cardiovacular system are undoubtedly associated with the most frequent diseases of modern time. Therefore, understanding hypoxic resistance encountered after physiological adaptation such as chronic hypoxia, is crucial to better
Externí odkaz:
https://doaj.org/article/d66182112a9f40faa3643a8b34278c3f
Autor:
Riccardo Olcese, Marina Angelini, Guillaume Calmettes, Antonios Pantazis, James N. Weiss, Hrayr S. Karagueuzian, Kyle Scranton, Nicoletta Savalli, Federica Steccanella, Michela Ottolia, Marvin G. Chang, Arash Pezhouman
Publikováno v:
Journal of General Physiology. 153
Ventricular arrhythmias, a leading cause of sudden cardiac death, can be triggered by cardiomyocyte early afterdepolarizations (EADs). EADs can result from an abnormal late activation of L-type Ca2+ channels (LTCCs). Current LTCC blockers (class IV a
Autor:
Philippe Diolez, Véronique Deschodt-Arsac, Guillaume Calmettes, Gilles Gouspillou, Laurent Arsac, Pierre Jais, Michel Haissaguerre, Pierre dos Santos
Publikováno v:
Methods in Molecular Biology ISBN: 9781071612699
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::5b2cec0d19ca7ae493e13e53dc8593ae
https://doi.org/10.1007/978-1-0716-1270-5_25
https://doi.org/10.1007/978-1-0716-1270-5_25
Publikováno v:
Korge, P; John, SA; Calmettes, G; & Weiss, JN. (2017). Reactive oxygen species production induced by pore opening in cardiac mitochondria: The role of complex II. JOURNAL OF BIOLOGICAL CHEMISTRY, 292(24), 9896-9905. doi: 10.1074/jbc.M116.768325. UCLA: Retrieved from: http://www.escholarship.org/uc/item/8r6319c4
Succinate-driven reverse electron transport (RET) through complex I is hypothesized to be a major source of reactive oxygen species (ROS) that induces permeability transition pore (PTP) opening and damages the heart during ischemia/reperfusion. Becau
Publikováno v:
Korge, P; Calmettes, G; John, SA; & Weiss, JN. (2017). Reactive oxygen species production induced by pore opening in cardiac mitochondria: The role of complex III. JOURNAL OF BIOLOGICAL CHEMISTRY, 292(24), 9882-9895. doi: 10.1074/jbc.M116.768317. UCLA: Retrieved from: http://www.escholarship.org/uc/item/6b08s006
Recent evidence has implicated succinate-driven reverse electron transport (RET) through complex I as a major source of damaging reactive oxygen species (ROS) underlying reperfusion injury after prolonged cardiac ischemia. However, this explanation m
Autor:
Scott A. John, Riccardo Olcese, Guillaume Calmettes, Kyle Scranton, Joshua I. Goldhaber, Mansoureh Eghbali, Soban Umar, Ariel L. Escobar, Michela Ottolia
Publikováno v:
Biophysical Journal. 118:100a-101a