Zobrazeno 1 - 3
of 3
pro vyhledávání: '"Guilherme T. Ribas"'
Autor:
Thiago J. Borges, Yoshikazu Ganchiku, Jeffrey O. Aceves, Ronald van Gaal, Sebastien G.M. Uzel, Ivy A. Rosales, Jonathan E. Rubins, Kenichi Kobayashi, Ken Hiratsuka, Murat Tekguc, Guilherme T. Ribas, Karina Lima, Rodrigo B. Gassen, Ryuji Morizane, Jennifer A. Lewis, Leonardo V. Riella
Publikováno v:
iScience, Vol 27, Iss 10, Pp 110957- (2024)
Summary: The increasing scarcity of organs and the significant morbidity linked to dialysis require the development of engineered kidney tissues from human-induced pluripotent stem cells. Integrative approaches that synergize scalable kidney organoid
Externí odkaz:
https://doaj.org/article/a643a4f97bf94f35ad5928910be26236
Autor:
Karim Yatim, MD, Ayman Al Jurdi, MD, Christopher El Mouhayyar, MD, Leela Morena, MD, Frank E. Hullekes, MD, Ruchama Verhoeff, MD, Guilherme T. Ribas, MSc, Daniel S. Pearson, MD, PhD, Leonardo V. Riella, MD, PhD, FASN
Publikováno v:
Transplantation Direct, Vol 10, Iss 9, p e1697 (2024)
Background. There are no high-quality data to guide long-term mycophenolate mofetil (MMF) dosing in kidney transplant recipients (KTRs) to balance the long-term risks of allograft rejection with that of infections and malignancy. At our center, KTRs
Externí odkaz:
https://doaj.org/article/9d65cc3769a64372a1ab600032708d61
Autor:
Cristian V, Riella, Michelle, McNulty, Guilherme T, Ribas, Calum F, Tattersfield, Chandra, Perez-Gill, Felix, Eichinger, Jessica, Kelly, Justin, Chun, Balajikarthick, Subramanian, Dieval, Guizelini, Seth L, Alper, Martin R, Pollak, Matthew G, Sampson, David J, Friedman
Publikováno v:
Proceedings of the National Academy of Sciences. 119
APOL1 risk variants are associated with increased risk of kidney disease in patients of African ancestry, but not all individuals with the APOL1 high-risk genotype develop kidney disease. As APOL1 gene expression correlates closely with the degree of
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7a0ffb44d962b84e6b405a0b6920030e
https://doi.org/10.1073/pnas.2210150119
https://doi.org/10.1073/pnas.2210150119