Zobrazeno 1 - 10
of 47
pro vyhledávání: '"Guang-Hui Jin"'
Autor:
An-Hao Liu, Xiao-Lu Sun, Di-Zhi Liu, Fan Xu, Shi-Jie Feng, Shi-Yu Zhang, Lu-Zhou Li, Jia-Li Zhou, Yi-Ting Wang, Li Zhang, Xiao Lin, Shu-Bin Gao, Xin Yue, Xiao-Ming Liu, Guang-Hui Jin, Bin Xu
Publikováno v:
Heliyon, Vol 9, Iss 4, Pp e15345- (2023)
Background: Hypertrophic scar (HS) and keloid (KD) are common dermal fibroproliferative growth caused by pathological wound healing. HS's prevalence is currently undetermined in China. Though it primarily occurs in dark-skinned individuals, KD can de
Externí odkaz:
https://doaj.org/article/020a965f9b7149a29c557299b9e485ce
Autor:
Huan Qiu, Bang-Ming Jin, Zhan-Feng Wang, Bin Xu, Qi-Fan Zheng, Li Zhang, Ling-Yu Zhu, Shuang Shi, Jun-Bo Yuan, Xiao Lin, Shu-Bin Gao, Guang-Hui Jin
Publikováno v:
Nature Communications, Vol 11, Iss 1, Pp 1-12 (2020)
Loss ofMEN1 results in an inherited multiple endocrine neoplastic type 1 syndrome. Here, the authors generate an alveolar type II cell MEN1 knockout mouse model and show that on a Kras mutant background the mice develop lung tumors with features of n
Externí odkaz:
https://doaj.org/article/6fb64604831e428da09697a05d822f59
Autor:
Guang-Hui Jin, Li-Xiang Xue, Xiao Lin, Qi-Lin Zheng, Yue Zhao, Kang-Li Li, Chang-Bao Pan, Bin Xu, Qi-Fan Zheng, Shu-Bin Gao
PDF file - 484K, S1. The efficiency of EZH2 and SUZ12 silencing by shRNA. S2. EZH2 repress TP53 expression in HCC cells. S3. PcG regulated P53 expression through H3K27me3 in an independent manner. Supplemental Table 1. The list of antibodies used in
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::eaf66fea0ee5c9d713ddc10b241a3b38
https://doi.org/10.1158/1541-7786.22513884
https://doi.org/10.1158/1541-7786.22513884
Supplementary Data from Loss of MLL Induces Epigenetic Dysregulation of Rasgrf1 to Attenuate Kras-Driven Lung Tumorigenesis
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::240976cc58a2b7827920e4bb14d64833
https://doi.org/10.1158/0008-5472.22432688
https://doi.org/10.1158/0008-5472.22432688
Supplementary Figure from Loss of MLL Induces Epigenetic Dysregulation of Rasgrf1 to Attenuate Kras-Driven Lung Tumorigenesis
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::09b78cb529433696f9d466ca8a995485
https://doi.org/10.1158/0008-5472.22432673
https://doi.org/10.1158/0008-5472.22432673
Menin is necessary for the formation of the menin/mixed lineage leukemia (MLL) complex and is recruited directly to chromatin. Menin is an important tumor suppressor in several cancer types, including lung cancer. Here, we investigated the role of ML
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::26455b5573c6b16d690c286411bbdab9
https://doi.org/10.1158/0008-5472.c.6514208
https://doi.org/10.1158/0008-5472.c.6514208
Publikováno v:
Cancer research. 82(22)
Menin is necessary for the formation of the menin/mixed lineage leukemia (MLL) complex and is recruited directly to chromatin. Menin is an important tumor suppressor in several cancer types, including lung cancer. Here, we investigated the role of ML
Autor:
Lue Xiang, Ji-Neng Lv, Jia Qu, Zi-Bing Jin, Kun-Chao Wu, Xiao Zhang, Jie Chen, Xiu-Feng Huang, Deng Pan, Guang-Hui Jin, Xue-Bi Cai, Fan Lu, Bin Lu
Publikováno v:
Clinical Genetics. 96:61-71
Adenosine diphosphate (ADP)-ribosylation factor-like 2 (ARL2) protein participates in a broad range of cellular processes and acts as a mediator for mutant ARL2BP in cilium-associated retinitis pigmentosa and for mutant HRG4 in mitochondria-related p
Autor:
Fei Chen, Hongqin Zhuang, Feng Wang, Weiwei Jiang, Fangfang Cai, Wei Dong, Nini Cao, Xiao Chen, Guang-Hui Jin, Jia Liu, Zi-Chun Hua, Xiangyu Zhang
Publikováno v:
Experimental and Molecular Medicine, Vol 51, Iss 2, Pp 1-20 (2019)
Experimental & Molecular Medicine
Experimental & Molecular Medicine
Radiotherapy is a frequent mode of cancer treatment, although the development of radioresistance limits its effectiveness. Extensive investigations indicate the diversity of the mechanisms underlying radioresistance. Here, we aimed to explore the eff
Autor:
Xue‐Bi Cai, Kun‐Chao Wu, Xiao Zhang, Ji‐Neng Lv, Guang‐Hui Jin, Lue Xiang, Jie Chen, Xiu‐Feng Huang, Deng Pan, Bin Lu, Fan Lu, Jia Qu, Zi‐Bing Jin
Publikováno v:
Clinical Genetics. 96