Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Gu-Yu Niu"'
Autor:
Chen-Meng Qiao, Wen-Yan Huang, Yu Zhou, Wei Quan, Gu-Yu Niu, Ting Li, Mei-Xuan Zhang, Jian Wu, Li-Ping Zhao, Wei-Jiang Zhao, Chun Cui, Yan-Qin Shen
Publikováno v:
Brain Sciences, Vol 14, Iss 3, p 238 (2024)
Increasing evidence suggests that the gut microbiota may represent potential strategies for Parkinson’s disease (PD) treatment. Our previous research revealed a decreased abundance of Akkermansia muciniphila (Akk) in PD mice; however, whether Akk i
Externí odkaz:
https://doaj.org/article/770f2fcdb0064653b87c47e0372a4742
Autor:
Li-Ping Zhao, Jian Wu, Wei Quan, Yu Zhou, Hui Hong, Gu-Yu Niu, Ting Li, Shu-Bing Huang, Chen-Meng Qiao, Wei-Jiang Zhao, Chun Cui, Yan-Qin Shen
Publikováno v:
Frontiers in Immunology, Vol 13 (2023)
Accumulative studies suggest that inflammatory bowel disease (IBD) may cause multiple central nervous system (CNS) pathologies. Studies have found that indoleamine-2,3-dioxygenase (IDO, rate-limiting enzyme of the kynurenine (Kyn) pathway) deficient
Externí odkaz:
https://doaj.org/article/5f29231e6cb143bebf7ecd53f2cd091b
Autor:
Wei Quan, Chen-Meng Qiao, Gu-Yu Niu, Jian Wu, Li-Ping Zhao, Chun Cui, Wei-Jiang Zhao, Yan-Qin Shen
Publikováno v:
Brain Sciences, Vol 13, Iss 5, p 790 (2023)
Observational studies have shown abnormal changes in trimethylamine N-oxide (TMAO) levels in the peripheral circulatory system of Parkinson’s disease (PD) patients. TMAO is a gut microbiota metabolite that can cross the blood–brain barrier and is
Externí odkaz:
https://doaj.org/article/960e3ffb27584aa4960d43ae4274541c
Autor:
Li-Ping Zhao, Jian Wu, Wei Quan, Yu Zhou, Hui Hong, Gu-Yu Niu, null Ting-Li, Shu-Bing Huang, Chen-Meng Qiao, Wei-Jiang Zhao, Chun Cui, Yan-Qin Shen
Publikováno v:
The Journal of Nutritional Biochemistry. 115:109282
Autor:
Yan-Qin Shen, Yu Zhou, Li-Ping Zhao, Yun Shi, Chen-Meng Qiao, Chun Cui, Wei-Jiang Zhao, Gu-Yu Niu, Hui Hong, Wei Quan
Publikováno v:
Brain research bulletin. 176
Aryl Hydrocarbon Receptor (AHR) is a ligand-activated transcription factor expressed in various brain regions. However, little is known about the role of AHR during neuroinflammation in the 1-methyl-4-phenyl-1,2,3,6-tetrathydropyridine (MPTP)-induced