Zobrazeno 1 - 10 of 10 pro vyhledávání: '"Gry Fluge Vindedal"'
2
Dynamics of Ionic Shifts in Cortical Spreading Depression
Autor: P. Johannes Helm, Rune Enger, Loren L. Looger, Wannan Tang, Rolf Sprengel, Vidar Jensen, Gry Fluge Vindedal, Erlend A. Nagelhus
Publikováno v: Cerebral Cortex (New York, NY)
Cerebral Cortex
Cortical spreading depression is a slowly propagating wave of near-complete depolarization of brain cells followed by temporary suppression of neuronal activity. Accumulating evidence indicates that cortical spreading depression underlies the migrain
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0fc026a1fcc400b27183bed4df5dd03e
https://doi.org/10.1093/cercor/bhv054
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3
Removal of aquaporin-4 from glial and ependymal membranes causes brain water accumulation
Autor: Ole Petter Ottersen, Vidar Jensen, Gry Fluge Vindedal, Michael J. Holtzman, Arne Klungland, Anna E. Thoren, Erlend A. Nagelhus, Yong Zhang
There is a constitutive production of water in brain. The efflux routes of this excess water remain to be identified. We used basal brain water content as a proxy for the capacity of water exit routes. Basal brain water content was increased in mice
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::de079c309297bc75d26fa47c05af6706
https://europepmc.org/articles/PMC5157926/
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4
IκB Kinase/Nuclear Factor κB-Dependent Insulin-Like Growth Factor 2 (Igf2) Expression Regulates Synapse Formation and Spine Maturation via Igf2 Receptor Signaling
Autor: Ayesha Maqbool, Gry Fluge Vindedal, Bernd Baumann, Jochen Seither, Alexander Magnutzki, Svenja Johannsen, Franz Oswald, Vidar R. Jensen, Thomas Wirth, Øivind Hvalby, Michael J. Schmeisser, Tobias M. Boeckers, Michael Lattke, Rolf Sprengel
Publikováno v: The Journal of Neuroscience : the Official Journal of the Society for Neuroscience
Alterations of learning and memory in mice with deregulated neuron-specific nuclear factor κB (NF-κB) activity support the idea that plastic changes of synaptic contacts may depend at least in part on IκB kinase (IKK)/NF-κB-related synapse-to-nuc
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::79154956cc732c34eec19ec581ddde6c
https://doi.org/10.1523/jneurosci.0111-12.2012
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5
Deletion of aquaporin-4 changes the perivascular glial protein scaffold without disrupting the brain endothelial barrier
Autor: Eystein Hellstrøm Hoddevik, Rune Enger, Marvin E. Adams, Øivind Skare, Georg Andreas Gundersen, Nadia Nabil Haj-Yasein, Erlend A. Nagelhus, Finn Mogens Haug, Anna E. Thoren, Martine Eilert-Olsen, Pétur Henry Petersen, Gry Fluge Vindedal, Stanley C. Froehner, John M. Burkhardt
Publikováno v: Glia. 60:432-440
Expression of the water channel aquaporin-4 (AQP4) at the blood-brain interface is dependent upon the dystrophin associated protein complex. Here we investigated whether deletion of the Aqp4 gene affects the molecular composition of this protein scaf
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::762538288837004d3e85cf3d31dd9605
https://doi.org/10.1002/glia.22277
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6
Glial-conditional deletion of aquaporin-4 ( Aqp4 ) reduces blood–brain water uptake and confers barrier function on perivascular astrocyte endfeet
Autor: Øivind Skare, Anna E. Thoren, Georg Andreas Gundersen, Ole Petter Ottersen, John M. Burkhardt, Erlend A. Nagelhus, Petter Laake, Arne Klungland, Nadia Nabil Haj-Yasein, Gry Fluge Vindedal, Martine Eilert-Olsen
Publikováno v: Proceedings of the National Academy of Sciences. 108:17815-17820
Tissue- and cell-specific deletion of the Aqp4 gene is required to differentiate between the numerous pools of aquaporin-4 (AQP4) water channels. A glial-conditional Aqp4 knockout mouse line was generated to resolve whether astroglial AQP4 controls w
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::602c5fe664cf7caabd8f4e000f5cf181
https://doi.org/10.1073/pnas.1110655108
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8
Loss of perivascular Kir4.1 potassium channels in the sclerotic hippocampus of patients with mesial temporal lobe epilepsy
Autor: Ole Petter Ottersen, Nihal C. de Lanerolle, Dennis D. Spencer, Anna E. Thoren, Erik Taubøll, Kjell Heuser, Fredrik Lauritzen, Erlend A. Nagelhus, Leif Gjerstad, Gry Fluge Vindedal, Tore Eid
Publikováno v: Journal of neuropathology and experimental neurology. 71(9)
Recent experimental data in mice have shown that the inwardly rectifying K+ channel Kir4.1 mediates K+ spatial buffering in the hippocampus. Here we used immunohistochemistry to examine the distribution of Kir4.1 in hippocampi from patients with medi
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::806c97ae85744264075264d140019fce
https://pubmed.ncbi.nlm.nih.gov/22878665
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9
Evidence that compromised K+ spatial buffering contributes to the epileptogenic effect of mutations in the human Kir4.1 gene (KCNJ10)
Autor: Arne Klungland, Gry Fluge Vindedal, Georg Andreas Gundersen, Ole Petter Ottersen, Erlend A. Nagelhus, Vidar Jensen, Nadia Nabil Haj-Yasein, Øivind Hvalby
Publikováno v: Glia. 59(11)
Mutations in the human Kir4.1 potassium channel gene (KCNJ10) are associated with epilepsy. Using a mouse model with glia-specific deletion of Kcnj10, we have explored the mechanistic underpinning of the epilepsy phenotype. The gene deletion was show
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b3ab48869e864e580ab9d4f8fbf38393
https://pubmed.ncbi.nlm.nih.gov/21748805
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10
Evidence that pericytes regulate aquaporin-4 polarization in mouse cortical astrocytes
Autor: Gry Fluge Vindedal, Øivind Skare, Georg Andreas Gundersen, Erlend A. Nagelhus
Publikováno v: Brain Structure & Function
Aquaporin-4 (AQP4) water channels are concentrated in astrocytic endfoot membranes at the brain–blood and brain–cerebrospinal fluid interfaces. The mechanisms underpinning the polarized distribution of AQP4 are poorly understood. Here we tested t
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2006884380723a8524cea5f55b2b106a
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