Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Gregory Sonnenberg"'
Publikováno v:
The Journal of Immunology. 192:133.30-133.30
IgA is a component of the mucosal barrier surface and plays an important part in maintaining separation between the host and the commensal microbiota. Mucosal IgA responses are considered short-lived and restricted to mucosal sites with no systemic d
Autor:
Brian Kim, Mark Siracusa, Steven Saenz, Mario Noti, Laurel Monticelli, Gregory Sonnenberg, Matthew Hepworth, Abby Van Voorhees, Michael Comeau, David Artis
Publikováno v:
The Journal of Immunology. 190:114.21-114.21
Innate lymphoid cells (ILCs) are a recently identified family of immune cells that regulate inflammation and tissue repair at barrier surfaces such as the intestine and lung. Previous studies have demonstrated that group 2 ILCs found in the intestine
Autor:
Vesselin Tomov, Lisa Osborne, Douglas Dolfi, Gregory Sonnenberg, Laurel Monticelli, Kathleen Mansfield, Herbert Virgin, David Artis, E. Wherry
Publikováno v:
The Journal of Immunology. 190:171.6-171.6
Norovirus (NV) gastroenteritis is a major contributor to global morbidity and mortality, yet little is known about immune mechanisms leading to NV control. Previous studies using the murine norovirus (MNV) model have established a key role for T cell
Innate lymphoid cells promote airway epithelial repair through the amphiregulin-EGFR pathway (P3250)
Autor:
Laurel Monticelli, Gregory Sonnenberg, Michael Abt, Lisa Osborne, Elia Tait Wojno, Theresa Alenghat, Carly Ziegler, E. Wherry, David Artis
Publikováno v:
The Journal of Immunology. 190:136.7-136.7
Innate lymphoid cells (ILCs) are a recently described group of immune cells that regulate immunity and inflammation at barrier surfaces. While recent studies have implicated Group 2 ILCs as initiators of pathologic Type 2 airway inflammation, the tis
Publikováno v:
The Journal of Immunology. 190:61.1-61.1
Mammals are colonized with trillions of commensal bacteria that are essential for normal intestinal physiology and immune cell development. However, dysregulated host-microbiota interactions have been causally linked with the pathogenesis and progres