Zobrazeno 1 - 10
of 53
pro vyhledávání: '"Gregory S, Retzinger"'
Publikováno v:
Frontiers in Allergy, Vol 5 (2024)
The Acari Hypothesis posits that acarians, i.e., mites and ticks, are causative agents of IgE-mediated conditions. This report further develops The Hypothesis, providing rationale for the childhood predilection of allergy. In short, Malassezia, a fun
Externí odkaz:
https://doaj.org/article/5b8e4a2c9d6c453fa2471498e6e9fb64
Publikováno v:
Frontiers in Allergy, Vol 5 (2024)
The Acari Hypothesis posits that acarians, i.e., mites and ticks, are operative agents of allergy. It derived from observations that allergens are molecular elements of acarians or acarian foodstuffs. A corollary of The Hypothesis provides how acaria
Externí odkaz:
https://doaj.org/article/e6e21af271ab4f64985b15c5623f6688
Publikováno v:
Frontiers in Allergy, Vol 5 (2024)
Allergy and its manifestations were first appreciated in the 1870 s. Today, the mechanism by which specific substances elicit allergic reactions remains poorly understood. This is problematic from a healthcare perspective because the prevalence of al
Externí odkaz:
https://doaj.org/article/b24aa1d1e6714bdfac42fbb16a9df9da
Publikováno v:
Pathogens, Vol 11, Iss 10, p 1083 (2022)
Atopic dermatitis is a chronic relapsing dermatopathology involving IgE against allergenic materials present on mammalian epithelial surfaces. Allergens are as diverse as pet danders, and polypeptides expressed by microbes of the mammalian microbiome
Externí odkaz:
https://doaj.org/article/b2f4fff5cf99447ea00308f0e573079f
Publikováno v:
Pathogens, Vol 10, Iss 9, p 1220 (2021)
Hypersensitivity to galactose-α-1,3-galactose (α-gal) is an informative example of a pathologic IgE-mediated process. By way of their saliva, ticks are able to sensitize humans to tick dietary elements that express α-gal. Mites, which along with t
Externí odkaz:
https://doaj.org/article/0edf0c90b114480fae085d689dcf1f5a
Autor:
Richa B. Shah, Rachna D. Shah, Damien G. Retzinger, Andrew C. Retzinger, Deborah A. Retzinger, Gregory S. Retzinger
Publikováno v:
Pathogens, Vol 10, Iss 9, p 1204 (2021)
Data from Chicago confirm the end of flu season coincides with the beginning of pollen season. More importantly, the end of flu season also coincides with onset of seasonal aerosolization of mold spores. Overall, the data suggest bioaerosols, especia
Externí odkaz:
https://doaj.org/article/f26472278e0547ceb6c2da5a2564f89a
Publikováno v:
COVID. 1:465-471
Recently, an inverse relationship between incidence of COVID-19 and seasonal aerosolization of mold spores was demonstrated. Analyses of that relationship suggested mold spores compete with SARS-CoV-2 virions for a receptor on the pulmonary epithelia
Autor:
Gregory S. Retzinger, Andrew C. Retzinger, Deborah A. Retzinger, Richa B. Shah, Rachna D. Shah, Damien G. Retzinger
Publikováno v:
Pathogens
Volume 10
Issue 9
Pathogens, Vol 10, Iss 1204, p 1204 (2021)
Volume 10
Issue 9
Pathogens, Vol 10, Iss 1204, p 1204 (2021)
Data from Chicago confirm the end of flu season coincides with the beginning of pollen season. More importantly, the end of flu season also coincides with onset of seasonal aerosolization of mold spores. Overall, the data suggest bioaerosols, especia
Publikováno v:
Medical Hypotheses
Aspergillus superinfection often accompanies severe COVID-19, even in immunocompetent individuals [1]. An inverse relationship between the incidence of COVID-19 and seasonal pollens [2] and mold spores [3] has recently been demonstrated. The relation
Autor:
Damien G. Retzinger, Gregory S. Retzinger, Richa B. Shah, Rachna D. Shah, Deborah A. Retzinger, Andrew C. Retzinger
Data from Chicago confirm the end of flu season coincides with the beginning of pollen season. The end of flu season also coincides with onset of seasonal aerosolization of mold spores. Overall, the data suggest bioaerosols, especially mold spores, c
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::b906b8d804e888ac8d8d071e07a2fdf5
https://doi.org/10.1101/2021.02.07.21251322
https://doi.org/10.1101/2021.02.07.21251322