Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Graciela López Soop"'
Autor:
Graciela López Soop, Trine Husøy, Marcin Wlodzimierz Wojewodzic, Hege Hjertholm, Anastasia Spyropoulou, Effrosyni S. Katsanou, Petros Batakis, Katerina Kyriakopoulou, Kyriaki Machera, Hubert Dirven, Birgitte Lindeman, Nur Duale
Publikováno v:
Environmental Research
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::02d28e2fda2f1e38a7ec8385530dd8de
https://hdl.handle.net/11250/3065460
https://hdl.handle.net/11250/3065460
Autor:
Laura Rué, Graciela López-Soop, Ellen Gelpi, Marta Martínez-Vicente, Jordi Alberch, Esther Pérez-Navarro
Publikováno v:
Neurobiology of Disease, Vol 52, Iss , Pp 219-228 (2013)
Huntington's disease is characterized by the formation of protein aggregates, which can be degraded by macroautophagy. Here, we studied protein levels and intracellular distribution of p62 and NBR1, two macroautophagy cargo receptors, during disease
Externí odkaz:
https://doaj.org/article/381121954ac843ba849b8ae1af9ada60
Autor:
Nina Richartz, Thomas Küntziger, Heidi Kiil Blomhoff, Bernd Thiede, Agnieszka Rogala, Graciela López-Soop, Philippe Collas, Torunn Rønningen
Publikováno v:
Cell Cycle. 16:947-956
Faithful chromosome segregation during mitosis relies on a proofreading mechanism that monitors proper kinetochore-microtubule attachments. The spindle assembly checkpoint (SAC) is based on the concerted action of numerous components that maintain a
Autor:
Esther Pérez-Navarro, Graciela López-Soop, Laura Rué, Jordi Creus-Muncunill, Rafael Alcalá-Vida, Jordi Alberch
Publikováno v:
NeuroMolecular Medicine. 16:25-37
A balance between cell survival and apoptosis is crucial to avoid neurodegeneration. Here, we analyzed whether the pro-apoptotic protein PKCδ, and the pro-survival PKCα and βII, were dysregulated in the brain of R6/1 mouse model of Huntington's di
Autor:
Ellen Gelpi, Esther Pérez-Navarro, Graciela López-Soop, Jordi Alberch, Laura Rué, Marta Martínez-Vicente
Publikováno v:
Neurobiology of Disease, Vol 52, Iss, Pp 219-228 (2013)
Huntington's disease is characterized by the formation of protein aggregates, which can be degraded by macroautophagy. Here, we studied protein levels and intracellular distribution of p62 and NBR1, two macroautophagy cargo receptors, during disease