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The TLR4 adaptor TRAM controls the phagocytosis of Gram-negative bacteria by interacting with the Rab11-family interacting protein 2.

Autor: Skjesol A; Centre of Molecular Inflammation Research, Department of Clinical and Molecular Medicine, Norwegian University of Science and Technology, Trondheim, Norway., Yurchenko M; Centre of Molecular Inflammation Research, Department of Clinical and Molecular Medicine, Norwegian University of Science and Technology, Trondheim, Norway., Bösl K; Centre of Molecular Inflammation Research, Department of Clinical and Molecular Medicine, Norwegian University of Science and Technology, Trondheim, Norway., Gravastrand C; Centre of Molecular Inflammation Research, Department of Clinical and Molecular Medicine, Norwegian University of Science and Technology, Trondheim, Norway., Nilsen KE; Centre of Molecular Inflammation Research, Department of Clinical and Molecular Medicine, Norwegian University of Science and Technology, Trondheim, Norway., Grøvdal LM; Centre of Molecular Inflammation Research, Department of Clinical and Molecular Medicine, Norwegian University of Science and Technology, Trondheim, Norway., Agliano F; Centre of Molecular Inflammation Research, Department of Clinical and Molecular Medicine, Norwegian University of Science and Technology, Trondheim, Norway.; Department of Clinical and Experimental Medicine, University of Messina, Messina, Italy., Patane F; Centre of Molecular Inflammation Research, Department of Clinical and Molecular Medicine, Norwegian University of Science and Technology, Trondheim, Norway.; Department of Clinical and Experimental Medicine, University of Messina, Messina, Italy., Lentini G; Centre of Molecular Inflammation Research, Department of Clinical and Molecular Medicine, Norwegian University of Science and Technology, Trondheim, Norway.; Department of Clinical and Experimental Medicine, University of Messina, Messina, Italy., Kim H; Centre of Molecular Inflammation Research, Department of Clinical and Molecular Medicine, Norwegian University of Science and Technology, Trondheim, Norway., Teti G; Department of Clinical and Experimental Medicine, University of Messina, Messina, Italy., Kumar Sharma A; Centre of Molecular Inflammation Research, Department of Clinical and Molecular Medicine, Norwegian University of Science and Technology, Trondheim, Norway., Kandasamy RK; Centre of Molecular Inflammation Research, Department of Clinical and Molecular Medicine, Norwegian University of Science and Technology, Trondheim, Norway., Sporsheim B; Centre of Molecular Inflammation Research, Department of Clinical and Molecular Medicine, Norwegian University of Science and Technology, Trondheim, Norway., Starheim KK; Centre of Molecular Inflammation Research, Department of Clinical and Molecular Medicine, Norwegian University of Science and Technology, Trondheim, Norway., Golenbock DT; Program in Innate Immunity, Division of Infectious Diseases and Immunology, Department of Medicine, University of Massachusetts Medical School, Worcester, MA, United States of America., Stenmark H; Centre of Molecular Inflammation Research, Department of Clinical and Molecular Medicine, Norwegian University of Science and Technology, Trondheim, Norway.; Centre for Cancer Cell Reprogramming, Faculty of Medicine, University of Oslo, Oslo, Norway.; Department for Molecular Cell Biology, Institute for Cancer Research, Oslo University Hospital, Oslo Norway., McCaffrey M; Molecular Cell Biology Laboratory, Biochemistry Department, Biosciences Institute, University College Cork, Cork, Ireland., Espevik T; Centre of Molecular Inflammation Research, Department of Clinical and Molecular Medicine, Norwegian University of Science and Technology, Trondheim, Norway.; The Central Norway Regional Health Authority, St. Olavs Hospital HF, Trondheim, Norway., Husebye H; Centre of Molecular Inflammation Research, Department of Clinical and Molecular Medicine, Norwegian University of Science and Technology, Trondheim, Norway.; The Central Norway Regional Health Authority, St. Olavs Hospital HF, Trondheim, Norway.

Publikováno v: PLoS pathogens [PLoS Pathog] 2019 Mar 18; Vol. 15 (3), pp. e1007684. Date of Electronic Publication: 2019 Mar 18 (Print Publication: 2019).

Molecular constituents of the extracellular matrix in rat liver mounting a hepatic progenitor cell response for tissue repair.

Autor: Vestentoft PS, Jelnes P, Andersen JB, Tran TA, Jørgensen T, Rasmussen M, Bornholdt J, Grøvdal LM, Jensen CH, Vogel LK, Thorgeirsson SS, Bisgaard HC; Department of Cellular and Molecular Medicine, Faculty of Health and Medical Sciences, University of Copenhagen, Blegdamsvej 3, DK-2200 Copenhagen, Denmark. hcb@sund.ku.dk.

Publikováno v: Fibrogenesis & tissue repair [Fibrogenesis Tissue Repair] 2013 Dec 20; Vol. 6 (1), pp. 21. Date of Electronic Publication: 2013 Dec 20.

Differential roles of Grb2 and AP-2 in p38 MAPK- and EGF-induced EGFR internalization.

Autor: Grandal MV; Department of Cellular and Molecular Medicine, University of Copenhagen, Copenhagen, Denmark., Grøvdal LM, Henriksen L, Andersen MH, Holst MR, Madshus IH, van Deurs B

Publikováno v: Traffic (Copenhagen, Denmark) [Traffic] 2012 Apr; Vol. 13 (4), pp. 576-85. Date of Electronic Publication: 2012 Jan 17.

Dysregulation of Ack1 inhibits down-regulation of the EGF receptor.

Autor: Grøvdal LM; University of Oslo, Institute of Pathology, Rikshospitalet HF, Oslo, Norway., Johannessen LE, Rødland MS, Madshus IH, Stang E

Publikováno v: Experimental cell research [Exp Cell Res] 2008 Apr 01; Vol. 314 (6), pp. 1292-300. Date of Electronic Publication: 2008 Jan 05.