Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Gourav R Choudhury"'
Publikováno v:
Brain Circulation, Vol 2, Iss 3, Pp 141-145 (2016)
Nonhuman primates (NHPs) are alike humans in size, behavior, physiology, biochemistry, and immunology. Given close similarities to humans, the NHP model offers exceptional opportunities to understand the biological mechanisms and translational applic
Externí odkaz:
https://doaj.org/article/ee7d330a02254079b85fb6eee20e7eae
Autor:
Gourav R Choudhury, Marcel M Daadi
Publikováno v:
PLoS ONE, Vol 13, Iss 8, p e0202770 (2018)
Parkinson's disease is a progressive neurodegenerative disease increasingly affecting our aging population. Remarkable advances have been made in developing novel therapies to control symptoms, halt or cure the disease, ranging from physiotherapy and
Externí odkaz:
https://doaj.org/article/84574da5d96f4e17858aeb63b464ef29
Autor:
Teerapat Rojsajjakul, Juliette J. Hordeaux, Gourav R. Choudhury, Christian J. Hinderer, Clementina Mesaros, James M. Wilson, Ian A. Blair
Publikováno v:
Communications Biology, Vol 6, Iss 1, Pp 1-13 (2023)
Abstract Deficiency in human mature frataxin (hFXN-M) protein is responsible for the devastating neurodegenerative and cardiodegenerative disease of Friedreich’s ataxia (FRDA). It results primarily through epigenetic silencing of the FXN gene by GA
Externí odkaz:
https://doaj.org/article/47677f60c0e74b1081e6b149da70b2a3
Autor:
Juliette Hordeaux, Brianne A. Jeffrey, Jinlong Jian, Gourav R. Choudhury, Kristofer Michalson, Thomas W. Mitchell, Elizabeth L. Buza, Jessica Chichester, Cecilia Dyer, Jessica Bagel, Charles H. Vite, Allison M. Bradbury, James M. Wilson
Publikováno v:
Human gene therapy. 33(9-10)
Krabbe disease is a lysosomal storage disease caused by mutations in the gene that encodes galactosylceramidase, in which galactosylsphingosine (psychosine) accumulation drives demyelination in the central and peripheral nervous systems, ultimately p
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::450f4a43e12fd74bd092d6865f466f03
https://pubmed.ncbi.nlm.nih.gov/35333110
https://pubmed.ncbi.nlm.nih.gov/35333110
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