Zobrazeno 1 - 10
of 398
pro vyhledávání: '"Gorno Tempini ML"'
Publikováno v:
Clinical Interventions in Aging, Vol Volume 14, Pp 453-471 (2019)
Heather R Dial,1,* Holly A Hinshelwood,2,* Stephanie M Grasso,1 H Isabel Hubbard,3 Maria-Luisa Gorno-Tempini,4 Maya L Henry1 1Communication Sciences and Disorders, University of Texas at Austin, Austin, TX, USA; 2Speech Pathology, Wake Forest Baptist
Externí odkaz:
https://doaj.org/article/2fc1653fbaf64c62a6e85153d846f749
Autor:
Borghesani, V, Wang, C, Miller, C, Mandelli, ML, Shapiro, K, Miller, Z, Fox, C, Dronkers, NF, Gorno-Tempini, ML, Watson, C
Publikováno v:
Neurocase, vol 27, iss 4
Decades of neuroscientific findings have elucidated the highly specialized brain areas involved in reading, especially along the ventral occipitotemporal stream where the critical step of recognizing words occurs. We report on a14-year-oldfemale with
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=od_______325::c451db13624b9a439f3aa9dfb799202c
https://escholarship.org/uc/item/12j3g0x1
https://escholarship.org/uc/item/12j3g0x1
Autor:
Lopez, A, Lee, SE, Wojta, K, Ramos, EM, Klein, E, Chen, J, Boxer, AL, Gorno-Tempini, ML, Geschwind, DH, Schlotawa, L, Ogryzko, NV, Bigio, EH, Rogalski, E, Weintraub, S, Mesulam, MM, Fleming, A, Coppola, G, Miller, BL, Rubinsztein, DC
Publikováno v:
Lopez, A; Lee, SE; Wojta, K; Ramos, EM; Klein, E; Chen, J; et al.(2017). A152T tau allele causes neurodegeneration that can be ameliorated in a zebrafish model by autophagy induction. BRAIN, 140, 1128-1146. doi: 10.1093/brain/awx005. UCLA: Retrieved from: http://www.escholarship.org/uc/item/3p51f6zm
Mutations in the gene encoding tau ( MAPT) cause frontotemporal dementia spectrum disorders. A rare tau variant p.A152T was reported as a risk factor for frontotemporal dementia spectrum and Alzheimer's disease in an initial case-control study. Such
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=dedup_wf_001::2321cb9d8e3b2e7357c90a6623948b0a
https://avesis.deu.edu.tr/publication/details/49a1570a-73a9-4bf6-82b5-452429a1d5a3/oai
https://avesis.deu.edu.tr/publication/details/49a1570a-73a9-4bf6-82b5-452429a1d5a3/oai
Autor:
Miller, Bruce, Brambati, SM, Amici, S, Racine, CA, Neuhaus, J, Miller, Z, Ogar, J, Dronkers, N, Miller, BL, Rosen, H, Gorno-Tempini, ML
Publikováno v:
Miller, Bruce; Brambati, SM; Amici, S; Racine, CA; Neuhaus, J; Miller, Z; et al.(2015). Longitudinal gray matter contraction in three variants of primary progressive aphasia: A tenser-based morphometry study. UC San Francisco: Retrieved from: http://www.escholarship.org/uc/item/19w2x8jd
© 2015 Published by Elsevier Inc.The present study investigated the pattern of longitudinal changes in cognition and anatomy in three variants of primary progressive aphasia (PPA). Eight patients with the non-fluent variant of PPA (nfvPPA), 13 patie
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=od_______325::ec7120baf640175a563f34cb2e653efe
http://www.escholarship.org/uc/item/19w2x8jd
http://www.escholarship.org/uc/item/19w2x8jd
Autor:
Caso F, Gesierich B, Henry M, Sidhu M, LaMarre A, Babiak M, Miller BL, Rabinovici GD, Huang EJ, Magnani G, FILIPPI , MASSIMO, COMI , GIANCARLO, Seeley WW, Gorno Tempini ML
Publikováno v:
Behavioural neurology, vol 26, iss 1-2
Caso, F; Gesierich, B; Henry, M; Sidhu, M; Lamarre, A; Babiak, M; et al.(2013). Nonfluent/agrammatic PPA with in-vivo cortical amyloidosis and Pick's disease pathology. Behavioural Neurology, 26(1-2), 95-106. doi: 10.3233/BEN-2012-120255. UCSF: Retrieved from: http://www.escholarship.org/uc/item/8wt9b744
Caso, F; Gesierich, B; Henry, M; Sidhu, M; Lamarre, A; Babiak, M; et al.(2013). Nonfluent/agrammatic PPA with in-vivo cortical amyloidosis and Pick's disease pathology. Behavioural Neurology, 26(1-2), 95-106. doi: 10.3233/BEN-2012-120255. UCSF: Retrieved from: http://www.escholarship.org/uc/item/8wt9b744
The role of biomarkers in predicting pathological findings in the frontotemporal dementia (FTD) clinical spectrum disorders is still being explored. We present comprehensive, prospective longitudinal data for a 66 year old, right-handed female who me
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=dedup_wf_001::2c6c5c985a4cb3a48f05b0d21d53fd87
https://escholarship.org/uc/item/8wt9b744
https://escholarship.org/uc/item/8wt9b744
Autor:
Ossenkoppele, R, Schonhaut, DR, Baker, SL, O'Neil, JP, Janabi, M, Ghosh, PM, Santos, M, Miller, ZA, Bettcher, BM, Gorno-Tempini, ML, Miller, BL, Jagust, WJ, Rabinovici, GD
Publikováno v:
Annals of Neurology, 77(2), 338-342. John Wiley and Sons Inc.
Annals of neurology, vol 77, iss 2
Ossenkoppele, R; Schonhaut, DR; Baker, SL; O'Neil, JP; Janabi, M; Ghosh, PM; et al.(2015). Tau, amyloid, and hypometabolism in a patient with posterior cortical atrophy. Annals of Neurology, 77(2), 338-342. doi: 10.1002/ana.24321. UC Berkeley: Retrieved from: http://www.escholarship.org/uc/item/54v288ks
Ossenkoppele, R, Schonhaut, D R, Baker, S L, O'Neil, J P, Janabi, M, Ghosh, P M, Santos, M, Miller, Z A, Bettcher, B M, Gorno-Tempini, M L, Miller, B L, Jagust, W J & Rabinovici, G D 2015, ' Tau, Amyloid, and Hypometabolism in a Patient with Posterior Cortical Atrophy ', Annals of Neurology, vol. 77, no. 2, pp. 338-342 . https://doi.org/10.1002/ana.24321
Annals of Neurology, vol 77, iss 2
Annals of neurology, vol 77, iss 2
Ossenkoppele, R; Schonhaut, DR; Baker, SL; O'Neil, JP; Janabi, M; Ghosh, PM; et al.(2015). Tau, amyloid, and hypometabolism in a patient with posterior cortical atrophy. Annals of Neurology, 77(2), 338-342. doi: 10.1002/ana.24321. UC Berkeley: Retrieved from: http://www.escholarship.org/uc/item/54v288ks
Ossenkoppele, R, Schonhaut, D R, Baker, S L, O'Neil, J P, Janabi, M, Ghosh, P M, Santos, M, Miller, Z A, Bettcher, B M, Gorno-Tempini, M L, Miller, B L, Jagust, W J & Rabinovici, G D 2015, ' Tau, Amyloid, and Hypometabolism in a Patient with Posterior Cortical Atrophy ', Annals of Neurology, vol. 77, no. 2, pp. 338-342 . https://doi.org/10.1002/ana.24321
Annals of Neurology, vol 77, iss 2
© 2014 American Neurological Association. Determining the relative contribution of amyloid plaques and neurofibrillary tangles to brain dysfunction in Alzheimer disease is critical for therapeutic approaches, but until recently could only be assesse
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid_dedup__::0396b3ddaf612f5637f7f09b64af9780
https://research.vumc.nl/en/publications/77ab87ae-32d6-4c73-89dd-5aa868989b1b
https://research.vumc.nl/en/publications/77ab87ae-32d6-4c73-89dd-5aa868989b1b