Contribution of Cyclooxygenase End Products and Oxidative Stress to Intrahepatic Endothelial Dysfunction in Early Non-Alcoholic Fatty Liver Disease.

Autor: Gonzalez-Paredes FJ; Institute of Biomedical Technologies and Center of Biomedical Research of the Canary Islands (CIBICAN), University of La Laguna, La Laguna, Tenerife, Spain., Hernández Mesa G; Gastroenterology Department, University Hospital of the Canary Islands, La Laguna, Tenerife, Spain., Morales Arraez D; Gastroenterology Department, University Hospital of the Canary Islands, La Laguna, Tenerife, Spain., Marcelino Reyes R; Gastroenterology Department, University Hospital of the Canary Islands, La Laguna, Tenerife, Spain., Abrante B; Institute of Biomedical Technologies and Center of Biomedical Research of the Canary Islands (CIBICAN), University of La Laguna, La Laguna, Tenerife, Spain., Diaz-Flores F; Central Laboratory, University Hospital of the Canary Islands, La Laguna, Tenerife, Spain., Salido E; Institute of Biomedical Technologies and Center of Biomedical Research of the Canary Islands (CIBICAN), University of La Laguna, La Laguna, Tenerife, Spain., Quintero E; Institute of Biomedical Technologies and Center of Biomedical Research of the Canary Islands (CIBICAN), University of La Laguna, La Laguna, Tenerife, Spain.; Gastroenterology Department, University Hospital of the Canary Islands, La Laguna, Tenerife, Spain.; Department of Medicine and Psychiatry, University of La Laguna, La Laguna, Tenerife, Spain., Hernández-Guerra M; Institute of Biomedical Technologies and Center of Biomedical Research of the Canary Islands (CIBICAN), University of La Laguna, La Laguna, Tenerife, Spain.; Gastroenterology Department, University Hospital of the Canary Islands, La Laguna, Tenerife, Spain.; Department of Medicine and Psychiatry, University of La Laguna, La Laguna, Tenerife, Spain.

Publikováno v: PloS one [PLoS One] 2016 May 26; Vol. 11 (5), pp. e0156650. Date of Electronic Publication: 2016 May 26 (Print Publication: 2016).

Splicing defects caused by exonic mutations in PKD1 as a new mechanism of pathogenesis in autosomal dominant polycystic kidney disease.

Autor: Claverie-Martin F; a Unidad de Investigacion; Hospital Nuestra Señora de Candelaria ; Santa Cruz de Tenerife , Spain., Gonzalez-Paredes FJ, Ramos-Trujillo E

Publikováno v: RNA biology [RNA Biol] 2015; Vol. 12 (4), pp. 369-74.