Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Glucosephosphate Dehydrogenase/genetics"'
Autor:
Satoru Watanabe, Hitoshi Nakamoto, Takahiro Maruyama, Emi Suenaga, Kensaku Fujita, Tomoko Misono, Hirofumi Yoshikawa, Pierre Goloubinoff, Shoichi Narumi, Aguru Ohtaki, Penmetcha K. R. Kumar
Publikováno v:
The Journal of biological chemistry
Journal of Biological Chemistry, vol. 289, no. 9, pp. 6110-6119
Journal of Biological Chemistry, vol. 289, no. 9, pp. 6110-6119
In eukaryotes, heat shock protein 90 (Hsp90) is an essential ATP-dependent molecular chaperone that associates with numerous client proteins. HtpG, a prokaryotic homolog of Hsp90, is essential for thermotolerance in cyanobacteria, and in vitro it sup
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::cc267e0d60c616546cae5ddadd849f93
Autor:
Alphaxard Manjurano, Nuno Sepulveda, Behzad Nadjm, George Mtove, Hannah Wangai, Caroline Maxwell, Raimos Olomi, Hugh Reyburn, Eleanor M Riley, Christopher J Drakeley, Taane G Clark, MalariaGEN Consortium
Publikováno v:
PLoS Genetics, Vol 11, Iss 2, p e1004960 (2015)
2015, ' African glucose-6-phosphate dehydrogenase alleles associated with protection from severe malaria in heterozygous females in Tanzania ', PLoS Genetics, vol. 11, no. 2, pp. e1004960 . https://doi.org/10.1371/journal.pgen.1004960
PLoS Genetics
2015, ' African glucose-6-phosphate dehydrogenase alleles associated with protection from severe malaria in heterozygous females in Tanzania ', PLoS Genetics, vol. 11, no. 2, pp. e1004960 . https://doi.org/10.1371/journal.pgen.1004960
PLoS Genetics
X-linked Glucose-6-phosphate dehydrogenase (G6PD) A- deficiency is prevalent in sub-Saharan Africa populations, and has been associated with protection from severe malaria. Whether females and/or males are protected by G6PD deficiency is uncertain, d
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9c4fbc701d2bbb918b61e9b80a83a07e
http://europepmc.org/articles/PMC4335500?pdf=render
http://europepmc.org/articles/PMC4335500?pdf=render
Autor:
Henri Wajcman, Abderraouf Ghanem, Kamran Moradkhani, Imen Moumni, Salem Abbes, Claude Préhu, Raouf Hafsia, Ikbel Benmansour
Publikováno v:
Blood Cells, Molecules and Diseases
Blood Cells, Molecules and Diseases, Elsevier, 2012, 50 (2), pp.110-114. ⟨10.1016/j.bcmd.2012.08.005⟩
Blood Cells, Molecules and Diseases, Elsevier, 2012, 50 (2), pp.110-114. ⟨10.1016/j.bcmd.2012.08.005⟩
We screened 423 patients referred to our laboratory after hemolysis triggered by fava beans ingestion, neonatal jaundice or drug hemolysis. Others were asymptomatic but belonged to a family with a history of G6PD deficiency. The determination of enzy
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4c710844fc8cf4eee1ed7d5a2a7dae1b
https://hal-riip.archives-ouvertes.fr/pasteur-00732993/document
https://hal-riip.archives-ouvertes.fr/pasteur-00732993/document
Publikováno v:
GMS German Medical Science; VOL: 6; DOC10 /20080924/
Objective: The mechanism by which glucose-6-phosphate dehydrogenase (G6PD) deficiency causes neonatal hyperbilirubinemia is not completely understood. However, the genetic disorder G6PD deficiency predisposes red blood cells to oxidative stress. The
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=od______1175::1096d0535a2015e578d05cf22e4cf16f
http://www.egms.de/en/journals/gms/2008-6/000055.shtml
http://www.egms.de/en/journals/gms/2008-6/000055.shtml