Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Gloria Ursu"'
Publikováno v:
Neurobiology of Disease, Vol 8, Iss 1, Pp 59-68 (2001)
Deregulation of intracellular calcium homeostasis is widely considered as one of the underlying pathophysiological mechanisms of hypoxic–ischemic brain injury. Whether this alteration can result in cerebral calcification was investigated in basal g
Externí odkaz:
https://doaj.org/article/aa801656f8324cd99408f30f78542193
Publikováno v:
Neurobiology of Disease, Vol 8, Iss 1, Pp 59-68 (2001)
Deregulation of intracellular calcium homeostasis is widely considered as one of the underlying pathophysiological mechanisms of hypoxic-ischemic brain injury. Whether this alteration can result in cerebral calcification was investigated in basal gan
Publikováno v:
Hippocampus. 9:527-533
This study investigated the modulatory actions of adenosine and gamma-aminobutyric acid (GABA) on several aspects of N-methyl-D-aspartate (NMDA)-induced neurotoxicity, including neuronal loss, atrophy, necrosis, and calcium accumulation in the hippoc
Autor:
Valérie Petegnief, Manuel J. Rodriguez, Marco Pugliese, Nicole Mahy, Gloria Ursu, Fabián Bernal
Publikováno v:
Digital.CSIC. Repositorio Institucional del CSIC
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Human cerebral calcification has been related to deregulation of intracellular calcium homeostasis. In rat basal ganglia, nimodipine and TMB-8, two commonly used calcium antagonists, worsen the chronic AMPA-induced lesion, whereas only nimodipine pot
Autor:
Yolanda Malpesa, Lydia Kerkerian-Le Goff, Lluïsa de Yebra, Nicole Mahy, Marco Pugliese, Gloria Ursu, Jean-Charles Liévens
Publikováno v:
Neurochemistry international. 49(7)
The inflammatory central nervous system response that involves activated microglia and reactive astrocytes may both heal and harm neurons, as inflammatory mediators lead to neuroprotection or excitation at one dose but to injury at a different concen
Publikováno v:
Neurochemistry international. 44(4)
Acute injection of alpha-amino-3-hydroxy-5-methyl-4-isoxazole propionate (AMPA) into the rat globus pallidus leads to calcium precipitation, neuronal death and gliosis. In order to determine whether L-type calcium channels and/or release of Ca(2+) fr