Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Glenna C L Bett"'
Publikováno v:
PLoS ONE, Vol 7, Iss 8, p e42295 (2012)
The fast component of the cardiac transient outward current, I(Ktof), is blocked by a number of drugs. The major molecular bases of I(Ktof) are Kv4.2/Kv4.3 voltage-gated potassium channels. Drugs with similar potencies but different blocking mechanis
Externí odkaz:
https://doaj.org/article/a980f126468b4a07897126871965e24b
Autor:
Bo Lin, Yang Li, Lu Han, Aaron D. Kaplan, Ying Ao, Spandan Kalra, Glenna C. L. Bett, Randall L. Rasmusson, Chris Denning, Lei Yang
Publikováno v:
Disease Models & Mechanisms, Vol 8, Iss 5, Pp 457-466 (2015)
Duchenne muscular dystrophy (DMD) is caused by mutations in the dystrophin gene (DMD), and is characterized by progressive weakness in skeletal and cardiac muscles. Currently, dilated cardiomyopathy due to cardiac muscle loss is one of the major caus
Externí odkaz:
https://doaj.org/article/6a0966a7de3e45bcaed9a21d27eb7ddc
Autor:
Glenna C L, Bett, Michael J, Morales, Derek L, Beahm, Michael E, Duffey, Randall L, Rasmusson
Publikováno v:
The Journal of physiology. 576(Pt 3)
KCNQ1 (Kv7.1 or KvLQT1) encodes the alpha-subunit of a voltage-gated potassium channel found in tissues including heart, brain, epithelia and smooth muscle. Tissue-specific characteristics of KCNQ1 current are diverse, due to modification by ancillar
Autor:
Kenneth R. Kahn, Glenna C. L. Bett
Publikováno v:
Obstetrics & Gynecology. 114:1147
Publikováno v:
Cell Biochemistry & Biophysics; Dec2003, Vol. 39 Issue 3, p183-194, 12p
Autor:
Rita Papp, Glenna C. L. Bett, Agnieszka Lis, Randall L. Rasmusson, István Baczkó, András Varró, Guy Salama
Publikováno v:
Biology of Sex Differences, Vol 8, Iss 1, Pp 1-11 (2017)
Abstract Background Women have a higher risk of lethal arrhythmias than men in long QT syndrome type 2 (LQTS2), but the mechanisms remain uncertain due to the limited availability of healthy control human tissue. We have previously reported that in f
Externí odkaz:
https://doaj.org/article/432274b0d9a74f0d9f9b2de5881f8db1