Zobrazeno 1 - 10
of 14
pro vyhledávání: '"Gitte R. Hinrichs"'
Autor:
Gitte R. Hinrichs, Sandrine Baltzer, Tamara Pallien, Per Svenningsen, Emil B. Dalgaard, Jens Michael Hertz, Claus Bistrup, Boye L. Jensen, Enno Klussmann
Publikováno v:
Kidney International Reports. 7:2289-2294
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::0442242474e3e7ba64f74b7f11fb7aed
https://doi.org/10.1016/j.ekir.2022.07.001
https://doi.org/10.1016/j.ekir.2022.07.001
Autor:
Gitte R. Hinrichs, Line Aas Mortensen
Publikováno v:
American journal of kidney diseases : the official journal of the National Kidney Foundation. 81(1)
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::56bf9ca9afb4c45f0f8b95c51d59ce20
https://pubmed.ncbi.nlm.nih.gov/35868958
https://pubmed.ncbi.nlm.nih.gov/35868958
Autor:
Gitte R, Hinrichs, Sandrine, Baltzer, Tamara, Pallien, Per, Svenningsen, Emil B, Dalgaard, Jens Michael, Hertz, Claus, Bistrup, Boye L, Jensen, Enno, Klussmann
Publikováno v:
Kidney international reports. 7(10)
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::f6712b227c382bd30d9e3635281625a2
https://pubmed.ncbi.nlm.nih.gov/36217530
https://pubmed.ncbi.nlm.nih.gov/36217530
Publikováno v:
Mortensen, L A, Bistrup, C, Jensen, B L & Hinrichs, G R 2020, ' A mini-review of pharmacological strategies used to ameliorate polyuria associated with X-linked nephrogenic diabetes insipidus ', American journal of physiology. Renal physiology, vol. 319, no. 5, pp. F746-F753 . https://doi.org/10.1152/ajprenal.00339.2020
Nephrogenic diabetes insipidus (NDI) is characterized by renal resistance to the antidiuretic hormone arginine vasopressin (AVP), which leads to polyuria, plasma hyperosmolarity, polydipsia, and impaired quality of living. Inherited forms are caused
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6130f2fd85cbb3cbb987f28a21af8ff0
https://pubmed.ncbi.nlm.nih.gov/32924547
https://pubmed.ncbi.nlm.nih.gov/32924547
Publikováno v:
Kidney International Reports
Hinrichs, G R, Mortensen, L A, Bistrup, C, Dieperink, H H & Jensen, B L 2020, ' Treatment of Nephrogenic Diabetes Insipidus Patients With cGMP : Stimulating Drugs Does Not Mitigate Polyuria or Increase Urinary Concentrating Ability ', Kidney International Reports, vol. 5, no. 8, pp. 1319-1325 . https://doi.org/10.1016/j.ekir.2020.05.015
Hinrichs, G R, Mortensen, L A, Bistrup, C, Dieperink, H H & Jensen, B L 2020, ' Treatment of Nephrogenic Diabetes Insipidus Patients With cGMP : Stimulating Drugs Does Not Mitigate Polyuria or Increase Urinary Concentrating Ability ', Kidney International Reports, vol. 5, no. 8, pp. 1319-1325 . https://doi.org/10.1016/j.ekir.2020.05.015
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1f7b0b242b418ac963419e0e7f96071c
https://pubmed.ncbi.nlm.nih.gov/32775834
https://pubmed.ncbi.nlm.nih.gov/32775834
Autor:
Rikke Nielsen, Géraldine Mollet, Ida K. Lund, Henrik Birn, Gitte R. Hinrichs, Boye L. Jensen, Per Svenningsen, Claus Bistrup, Ulla G. Friis, Corinne Antignac, Kathrin Weyer
Publikováno v:
Hinrichs, G R, Weyer, K, Friis, U G, Svenningsen, P, Lund, I K, Nielsen, R, Mollet, G, Antignac, C, Bistrup, C, Jensen, B L & Birn, H 2020, ' Sodium retention by uPA-plasmin-ENaC in nephrotic syndrome-Authors reply ', Acta Physiologica
, vol. 228, no. 4, e13432 . https://doi.org/10.1111/apha.13432
Hinrichs, G R, Weyer, K, Friis, U G, Svenningsen, P, Lund, I K, Nielsen, R, Mollet, G, Antignac, C, Bistrup, C, Jensen, B L & Birn, H 2020, ' Sodium retention by uPA-plasmin-ENaC in nephrotic syndrome : Authors reply ', Acta Physiologica
, vol. 228, no. 4, e13432 . https://doi.org/10.1111/apha.13432
Hinrichs, G R, Weyer, K, Friis, U G, Svenningsen, P, Lund, I K, Nielsen, R, Mollet, G, Antignac, C, Bistrup, C, Jensen, B L & Birn, H 2020, ' Sodium retention by uPA-plasmin-ENaC in nephrotic syndrome : Authors reply ', Acta Physiologica
There is a great interest in exploring the mechanisms of oedema in nephrotic syndrome (NS) as evidenced by 2 parallel studies published in Acta Physiologica (1, 2). This has generated an editorial comment (3) which we respond to by this rebuttal. The
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a33866c5f169d8ef3fe715eb61ac2102
https://doi.org/10.1111/apha.13432
https://doi.org/10.1111/apha.13432
Publikováno v:
Hinrichs, G R, Jensen, B L & Svenningsen, P 2020, ' Mechanisms of sodium retention in nephrotic syndrome ', Current Opinion in Nephrology & Hypertension, vol. 29, no. 2, pp. 207-212 . https://doi.org/10.1097/MNH.0000000000000578
PURPOSE OF REVIEW: Proteinuria in nephrotic syndrome is associated with sodium retention and edema. Recent studies from mice, rats and humans have shown that the sodium retention depends on urinary serine proteases and that it can be mitigated by blo
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d968d26a9f838cb3939c6810a6494fe2
https://pubmed.ncbi.nlm.nih.gov/31789848
https://pubmed.ncbi.nlm.nih.gov/31789848
Autor:
Jane Stubbe, Claus Bistrup, Reza Zamani, René Frydensbjerg Andersen, Gitte R. Hinrichs, Hiba Iraqi, Kristian Bergholt Buhl, Boye L. Jensen, Rikke Ydegaard, Henrik Dimke, Niels Marcussen, Per Svenningsen
Publikováno v:
Ydegaard, R, Svenningsen, P, Bistrup, C, Andersen, R F, Stubbe, J, Buhl, K B, Marcussen, N, Hinrichs, G R, Iraqi, H, Zamani, R, Dimke, H & Jensen, B L 2019, ' Nephrotic syndrome is associated with increased plasma K + concentration, intestinal K + losses, and attenuated urinary K + excretion : a study in rats and humans ', American Journal of Physiology: Renal Physiology, vol. 317, no. 6, pp. F1549-F1562 . https://doi.org/10.1152/ajprenal.00179.2019
Ydegaard, R, Svenningsen, P, Bistrup, C, Andersen, R F, Stubbe, J, Buhl, K B, Marcussen, N, Hinrichs, G R, Iraqi, H, Zamani, R, Dimke, H & Jensen, B L 2019, ' Nephrotic syndrome is associated with increased plasma K + concentration, intestinal K + losses, and attenuated urinary K + excretion : a study in rats and humans ', American journal of physiology. Renal physiology, vol. 317, no. 6, pp. F1549-F1562 . https://doi.org/10.1152/ajprenal.00179.2019
Ydegaard, R, Svenningsen, P, Bistrup, C, Andersen, R F, Stubbe, J, Buhl, K B, Marcussen, N, Hinrichs, G R, Iraqi, H, Zamani, R, Dimke, H & Jensen, B L 2019, ' Nephrotic syndrome is associated with increased plasma K + concentration, intestinal K + losses, and attenuated urinary K + excretion : a study in rats and humans ', American journal of physiology. Renal physiology, vol. 317, no. 6, pp. F1549-F1562 . https://doi.org/10.1152/ajprenal.00179.2019
The present study tested the hypotheses that nephrotic syndrome (NS) leads to renal K+ loss because of augmented epithelial Na+ channel (ENaC) activity followed by downregulation of renal K+ secretory pathways by suppressed aldosterone. The hypothese
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5c062965e488ed4894f8e24eb7cdde4e
https://findresearcher.sdu.dk:8443/ws/files/157638343/ajprenal.00179.2019.pdf
https://findresearcher.sdu.dk:8443/ws/files/157638343/ajprenal.00179.2019.pdf
Autor:
Birgül Kurt, Mette Staehr, Pernille B. Lærkegaard Hansen, Gitte R. Hinrichs, Claus Bistrup, Boye L. Jensen, Christina Stolzenburg Oxlund, Per Svenningsen, Mie R. Hansen, Ilona Schwarzensteiner, Ib Abildgaard Jacobsen, Anja Toft, Anne D Thuesen
Publikováno v:
Stolzenburg Oxlund, C, Kurt, B, Schwarzensteiner, I, Hansen, M R, Stæhr, M, Svenningsen, P, Jacobsen, I A, Hansen, P B, Thuesen, A D, Toft, A, Hinrichs, G R, Bistrup, C & Jensen, B L 2017, ' Albuminuria is associated with an increased prostasin in urine while aldosterone has no direct effect on urine and kidney tissue abundance of prostasin ', Pfluegers Archiv, vol. 469, no. 5-6, pp. 655–667 . https://doi.org/10.1007/s00424-017-1938-6
The proteinase prostasin is a candidate mediator for aldosterone-driven proteolytic activation of the epithelial sodium channel (ENaC). It was hypothesized that the aldosterone-mineralocorticoid receptor (MR) pathway stimulates prostasin abundance in
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6235fd863d3e55514ccd4f982e378325
https://doi.org/10.1007/s00424-017-1938-6
https://doi.org/10.1007/s00424-017-1938-6
Autor:
Gitte R. Hinrichs, Boye L. Jensen, Rikke Nielsen, Géraldine Mollet, Henrik Birn, Ulla G. Friis, Corinne Antignac, Kathrin Weyer, Per Svenningsen, Ida K. Lund, Claus Bistrup
Publikováno v:
Hinrichs, G R, Weyer, K, Friis, U G, Svenningsen, P, Lund, I K, Nielsen, R, Mollet, G, Antignac, C, Bistrup, C, Jensen, B L & Birn, H 2019, ' Urokinase-type plasminogen activator contributes to amiloride-sensitive sodium retention in nephrotic range glomerular proteinuria in mice ', Acta Physiologica, vol. 227, no. 4, e13362 . https://doi.org/10.1111/apha.13362
Hinrichs, G R, Weyer, K, Friis, U G, Svenningsen, P, Lund, I K, Nielsen, R, Mollet, G, Antignac, C, Bistrup, C, Jensen, B L & Birn, H 2019, ' Urokinase-type plasminogen activator contributes to amiloride-sensitive sodium retention in nephrotic range glomerular proteinuria in mice ', Acta Physiologica (Online), vol. 227, no. 4, e13362 . https://doi.org/10.1111/apha.13362
Hinrichs, G R, Weyer, K, Friis, U G, Svenningsen, P, Lund, I K, Nielsen, R, Mollet, G, Antignac, C, Bistrup, C, Jensen, B L & Birn, H 2019, ' Urokinase-type plasminogen activator contributes to amiloride-sensitive sodium retention in nephrotic range glomerular proteinuria in mice ', Acta Physiologica (Online), vol. 227, no. 4, e13362 . https://doi.org/10.1111/apha.13362
AIM: Activation of sodium reabsorption by urinary proteases has been implicated in sodium retention associated with nephrotic syndrome. The study was designed to test the hypothesis that nephrotic proteinuria in mice after conditional deletion of pod
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1505f3e8eb4b67f1cf8a563835045474
https://doi.org/10.1111/apha.13362
https://doi.org/10.1111/apha.13362