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Autor:
Gioilli, B D, Kidane, T Z, Fieten, H, Tellez, M, Dalphin, M, Nguyen, A, Nguyen, K, Linder, M C, Interne geneeskunde GD, dCSCA AVR, dCSCA RMSC-1, CS_Genetics
Publikováno v:
Metallomics, 14(3), 1. Royal Society of Chemistry
Studies with Wilson disease model mice that accumulate excessive copper, due to a dysfunctional ATP7B “copper pump” resulting in decreased biliary excretion, showed that the compensatory increase in urinary copper loss was due to a small copper c