Zobrazeno 1 - 10 of 12 pro vyhledávání: '"Germán E. González"'
1
Akademický článek
Unraveling the role of galectin-3 in cardiac pathology and physiology
Autor: Ignacio M. Seropian, Pablo Cassaglia, Verónica Miksztowicz, Germán E. González
Publikováno v: Frontiers in Physiology, Vol 14 (2023)
Galectin-3 (Gal-3) is a carbohydrate-binding protein with multiple functions. Gal-3 regulates cell growth, proliferation, and apoptosis by orchestrating cell-cell and cell-matrix interactions. It is implicated in the development and progression of ca
Externí odkaz: https://doaj.org/article/38d409491499468f803f0d1838b87dcc
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2
Akademický článek
Cardiac Natriuretic Peptide Profiles in Chronic Hypertension by Single or Sequentially Combined Renovascular and DOCA-Salt Treatments
Autor: Carolina S. Cerrudo, Susana Cavallero, Martín Rodríguez Fermepín, Germán E. González, Martín Donato, Nicolás M. Kouyoumdzian, Ricardo J. Gelpi, Cecilia M. Hertig, Marcelo R. Choi, Belisario E. Fernández
Publikováno v: Frontiers in Physiology, Vol 12 (2021)
The involvement of natriuretic peptides was studied during the hypertrophic remodeling transition mediated by sequential exposure to chronic hemodynamic overload. We induced hypertension in rats by pressure (renovascular) or volume overload (DOCA-sal
Externí odkaz: https://doaj.org/article/b608038eb7bd45ab98cf15356f8baae6
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3
Akademický článek
Galectin-1 as an Emerging Mediator of Cardiovascular Inflammation: Mechanisms and Therapeutic Opportunities
Autor: Ignacio M. Seropian, Germán E. González, Sebastián M. Maller, Daniel H. Berrocal, Antonio Abbate, Gabriel A. Rabinovich
Publikováno v: Mediators of Inflammation, Vol 2018 (2018)
Galectin-1 (Gal-1), an evolutionarily conserved β-galactoside-binding lectin, controls immune cell homeostasis and tempers acute and chronic inflammation by blunting proinflammatory cytokine synthesis, engaging T-cell apoptotic programs, promoting e
Externí odkaz: https://doaj.org/article/4135328507c6443db4d01c9200ebd0d0
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4
Genetic Deletion of Galectin-3 Alters the Temporal Evolution of Macrophage Infiltration and Healing Affecting the Cardiac Remodeling and Function after Myocardial Infarction in Mice
Autor: Marisa M. Fernández, Florencia Fontana Estevez, María Clara Llamosas, Ágata Carolina Cevey, Celina Morales, Eliana Cicale, Verónica Volberg, Germán E. González, Gabriela Berg, Nora Goren, Verónica Miksztowicz, Vanessa Touceda, Sofía Noli Truant, Celeste Betazza, Federico Nicolás Penas, Luciana Wilensky, Pablo Cassaglia, Nadia L. Martínez Naya
Publikováno v: The American Journal of Pathology. 2020, 190(9)
Repositorio Institucional (UCA)
Pontificia Universidad Católica Argentina
instacron:UCA
CONICET Digital (CONICET)
Consejo Nacional de Investigaciones Científicas y Técnicas
instacron:CONICET
We studied the role of galectin-3 (Gal-3) in the expression of alternative activation markers (M2) on macrophage, cytokines, and fibrosis through the temporal evolution of healing, ventricular remodeling, and function after myocardial infarction (MI)
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d7eb9f48ccae9c6e1d254a1f220d7a58
https://doi.org/10.1016/j.ajpath.2020.05.010
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5
Nebivolol is more effective than atenolol for blood pressure variability attenuation and target organ damage prevention in L-NAME hypertensive rats
Autor: Julieta S, Del Mauro, Paula D, Prince, Yanina, Santander Plantamura, Miguel A, Allo, Luciano, Parola, Nahuel, Fernandez Machulsky, Marcela A, Morettón, Eliana P, Bin, Germán E, González, Facundo M, Bertera, Andrea, Carranza, Gabriela, Berg, Carlos A, Taira, Martín, Donato, Diego A, Chiappetta, Ariel H, Polizio, Christian, Höcht
Publikováno v: Hypertension research : official journal of the Japanese Society of Hypertension. 44(7)
β-Adrenergic blockers are no longer recommended as first-line therapy due to the reduced cardioprotection of traditional β-blockers compared with other antihypertensive drugs. It is unknown whether third-generation β-blockers share the limitations
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::547ebe904ae613a59720efe19c55ad59
https://pubmed.ncbi.nlm.nih.gov/33612826
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7
Abstract 101: Cardiac Deleterious Role of Galectin-3 in Chronic Angiotensin-II Induced Hypertension
Autor: Germán E González, Nour-Eddine Rhaleb, Xiao- P Yang, Oscar A Carretero
Publikováno v: Hypertension. 62
We previously described that chronic infusion with Angiotensin II (Ang II) increases cardiac Galectin-3 (Gal-3) expression, a carbohydrate-binding lectin present on macrophages. Also, Gal-3 was proposed to be a powerful predictor for mortality in pat
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::246e7930c97c75bb4627f5cf6fa60cd2
https://doi.org/10.1161/hyp.62.suppl_1.a101
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8
Abstract 446: Deletion of Il-6 Prevents Development of Cardiac Damage and Dysfunction in Chronic Ang Ii-salt-induced Hypertension
Autor: Germán E González, Nour-Eddine Rhaleb, Pablo Nakagawa, Yun-He Liu, Oscar A Carretero
Publikováno v: Hypertension. 60
IL-6 knockout (KO) mice were reported to spontaneously develop cardiac dysfunction and fibrosis. These KO mice also develop less hypertension when fed high salt and infused with angiotensin II (Ang II). We tested the hypothesis that in IL-6-KO mice t
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::4048147bb36b2f61a239192cdd265cb2
https://doi.org/10.1161/hyp.60.suppl_1.a446
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9
Ludwig: the bioengineer
Autor: Max E, Valentinuzzi, Klaus, Beneke, Germán E, González
Publikováno v: IEEE pulse. 3(4)
On the basis of the strict exclusion of the vis vitalis, the demand was raised by Carl Ludwig, Helmholtz, Du Bois-Reymond, and Brucke for a physiology which was causal-analytical and physically and chemically experimental. If, out of these four inves
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::9884c9cd74ac9c925bb74a16f7864d24
https://pubmed.ncbi.nlm.nih.gov/22835382
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10
Effects of low-calcium reperfusion and adenosine on diastolic behavior during the transitory systolic overshoot of the stunned myocardium in the rabbit
Autor: Celina Morales, M. Donato, Manuel Rodríguez, Ricardo J. Gelpi, Germán E. González, Verónica D'Annunzio, Jimena Palleiro
Publikováno v: Canadian journal of physiology and pharmacology. 84(2)
The aims of the present study were to determine whether the transitory systolic overshoot (TSO) that occurs in the early reperfusion (R) of the stunned myocardium is accompanied by diastolic alterations, and to determine whether the R with low Ca2+ K
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1fba37d9ef902de1a0c4524f8eabe2f2
https://pubmed.ncbi.nlm.nih.gov/16900953
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